1,381 research outputs found
Elastic amplitudes studied with the LHC measurements at 7 and 8 TeV
Recent measurements of the differential cross sections in the forward region
of pp elastic scattering at 7 and 8 TeV show precise form of the
dependence. We propose a detailed analysis of these measurements including the
structures of the real and imaginary parts of the scattering amplitude. A good
description is achieved, confirming in all experiments the existence of a zero
in the real part in the forward region close to the origin, in agreement with
the prediction of a theorem by A. Martin, with important role in the observed
form of . Universal value for the position of this zero and
regularity in other features of the amplitudes are found, leading to
quantitative predictions for the forward elastic scattering at 13 TeV.Comment: 22 pages, 17 figures and 4 table
Photon and Pomeron -- induced production of Dijets in , and collisions
In this paper we present a detailed comparison of the dijet production by
photon -- photon, photon -- pomeron and pomeron -- pomeron interactions in
, and collisions at the LHC energy. The transverse
momentum, pseudo -- rapidity and angular dependencies of the cross sections are
calculated at LHC energy using the Forward Physics Monte Carlo (FPMC), which
allows to obtain realistic predictions for the dijet production with two
leading intact hadrons. We obtain that \gamma \pom channel is dominant at
forward rapidities in collisions and in the full kinematical range in the
nuclear collisions of heavy nuclei. Our results indicate that the analysis of
dijet production at the LHC can be useful to test the Resolved Pomeron model as
well as to constrain the magnitude of the absorption effects.Comment: 11 pages, 6 figures, 1 table. Improved and enlarged version published
in European Physical Journal
New properties of the Lerch's transcendent
A new representation of the Lerch''s transcendent F(z, s, a), valid for positive integer s=n=1, 2, … and for z and a belonging to certain regions of the complex plane, is presented. It allows to write an equation relating F(z, n, a) and F(1/z, n, 1-a), which in turn provides an expansion of F(z, n, a) as a power series of 1/z, convergent for |z|>1
nfi-1 affects behavior and life-span in C. elegans but is not essential for DNA replication or survival
BACKGROUND: The Nuclear Factor I (one) (NFI) family of transcription/replication factors plays essential roles in mammalian gene expression and development and in adenovirus DNA replication. Because of its role in viral DNA replication NFI has long been suspected to function in host DNA synthesis. Determining the requirement for NFI proteins in mammalian DNA replication is complicated by the presence of 4 NFI genes in mice and humans. Loss of individual NFI genes in mice cause defects in brain, lung and tooth development, but the presence of 4 homologous NFI genes raises the issue of redundant roles for NFI genes in DNA replication. No NFI genes are present in bacteria, fungi or plants. However single NFI genes are present in several simple animals including Drosophila and C. elegans, making it possible to test for a requirement for NFI in multicellular eukaryotic DNA replication and development. Here we assess the functions of the single nfi-1 gene in C. elegans. RESULTS: C. elegans NFI protein (CeNFI) binds specifically to the same NFI-binding site recognized by vertebrate NFIs. nfi-1 encodes alternatively-spliced, maternally-inherited transcripts that are expressed at the single cell stage, during embryogenesis, and in adult muscles, neurons and gut cells. Worms lacking nfi-1 survive but have defects in movement, pharyngeal pumping and egg-laying and have a reduced life-span. Expression of the muscle gene Ce titin is decreased in nfi-1 mutant worms. CONCLUSION: NFI gene function is not needed for survival in C. elegans and thus NFI is likely not essential for DNA replication in multi-cellular eukaryotes. The multiple defects in motility, egg-laying, pharyngeal pumping, and reduced lifespan indicate that NFI is important for these processes. Reduction in Ce titin expression could affect muscle function in multiple tissues. The phenotype of nfi-1 null worms indicates that NFI functions in multiple developmental and behavioral systems in C. elegans, likely regulating genes that function in motility, egg-laying, pharyngeal pumping and lifespan maintenance
In Situ Synchrotron Radiation Study of TiH2-6Al-4V and Ti-6Al-4V: Accelerated Alloying and Phase Transformation, and Formation of an Oxygen-Enriched Ti4Fe2O Phase in TiH2-6Al-4V
In situ heating, synchrotron radiation X-ray diffraction has been used to study the alloying and phase transformation behavior of TiH2-6Al-4V and Ti-6Al-4V alloys. Accelerated alloying and phase transformation were observed in the powder compact of the TiH2-6Al-4V alloy subjected to a high heating rate. In addition, an oxygen-stabilized Ti4Fe2O phase, which is present as sub-micron or nanoscaled particles, has been identified in the TiH2-6Al-4V alloy. The implications of these experimental findings have been discussed in terms of alloying, improved densification and oxygen scavenging in titanium and titanium alloys
Hepatitis C virus attenuates mitochondrial lipid β-oxidation by downregulating mitochondrial trifunctional-protein expression
The course of hepatitis C virus (HCV) infection and disease progression involves alterations in lipid metabolism, leading to symptoms such as hypocholesterolemia and steatosis. Steatosis can be induced by multiple mechanisms, including increases in lipid biosynthesis and uptake, impaired lipoprotein secretion, and/or attenuation of lipid β-oxidation. However, little is known about the effects of HCV on lipid β-oxidation. A previous proteomics study revealed that HCV interacted with both the α- and β-subunits of the mitochondrial trifunctional protein (MTP), an enzyme complex which catalyzes the last 3 steps of mitochondrial lipid β-oxidation for cellular energy production. Here we show that in HCV-infected Huh7.5 cells, lipid β-oxidation was significantly attenuated. Consistently with this, MTP protein and mRNA levels were suppressed by HCV infection. A loss-offunction study showed that MTP depletion rendered cells less responsive to alpha interferon (IFN-α) treatment by impairing IFN-stimulated gene expression. These aspects of host-virus interaction explain how HCV alters host energy homeostasis and how it may also contribute to the establishment of persistent infection in the liver
Nonet Symmetry and Two-Body Decays of Charmed Mesons
The decay of charmed mesons into pseudoscalar (P) and vector (V) mesons is
studied in the context of nonet symmetry. We have found that it is badly broken
in the PP channels and in the P sector of the PV channels as expected from the
non-ideal mixing of the \eta and the \eta'. In the VV channels, it is also
found that nonet symmetry does not describe the data well. We have found that
this discrepancy cannot be attributed entirely to SU(3) breaking at the usual
level of 20--30%. At least one, or both, of nonet and SU(3) symmetry must be
very badly broken. The possibility of resolving the problem in the future is
also discussed.Comment: 9 pages, UTAPHY-HEP-
SU(3)_flavor analysis of two-body weak decays of charmed baryons
We study two-body weak decays of charmed baryons \Lambda_c and \Xi_c into an
octet or decuplet baryon and a pseudoscalar meson employing the SU(3) flavor
symmetry. Using certain measured Cabibbo-favored modes, we fix the reduced
amplitudes and predict the branching ratios of various decays of charmed
baryons in the Cabibbo-enhanced, -suppressed and -doubly suppressed modes.Comment: 25 pages, No figure, Phys. Rev. D (to appear
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