5 research outputs found
New insights into the genetic etiology of Alzheimer's disease and related dementias
- Author
- Aaltonen L.
- Aaltonen V.
- Aarsland Dag
- Aavikko M.
- Abalos M.S.
- Abdelnour Carla
- Abner E.
- Abraham R.
- Adams H.
- Adams P.M.
- Adarmes-GĂłmez A.
- Adarmes-GĂłmez A.D.
- Aguilera N.
- Aguilera N.
- Aguirre A.
- Ahmad S.
- Akinyemi R.O.
- Al-Chalabi A.
- AlarcĂłn-MartĂn Emilio
- Albert M.S.
- Albin R.L.
- Alcolea Daniel
- Alegret Montserrat
- Ali M.
- Allen M.
- Allende I.R.
- Alonso M.D.
- Alonso M.D.
- Alvarez I.
- Alvarez L.
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- Amouyel Philippe
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- Yaqub A.
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- 01/01/2022
- Field of study
Get PDFCharacterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele
Family involvement and helping behaviour in teams
- Author
- Allen T.D.
- Anderson S.E.
- Anderson S.E.
- Andrews A.
- Arbuckle J.L.
- Bakker A.B.
- Bakker A.B.
- Baron R.M.
- Bernasco W.
- Brief A.P.
- Browne M.W.
- Burke R.J.
- Carlson D.S.
- Cinamon R.G.
- De Dreu C.K.W.
- Demerouti E.
- Duxbury L.E.
- Dykstra P.A.
- Eagle B.W.
- Eby L.T.
- Edwards J.
- Elliott M.
- Erickson R.J.
- Esther S. Kluwer
- Flap H.
- Friedman S.D.
- Frone M.R.
- Glass J.L.
- Goode W.J.
- Goodman S.A.
- Graves L.M.
- Greenhaus J.
- Greenhaus J.H.
- Grzywacz J.G.
- Hechter M.
- Hill E.J.
- Hobfoll S.E.
- Hobfoll S.E.
- Hodson R.
- Kalmijn M.
- Kluwer E.S.
- Koster F.
- Lavee Y.
- Lee C.
- Lieke L. ten Brummelhuis
- Loerch K.J.
- Loscocco K.A.
- Lundberg U.
- MacKinnon D.P.
- Marks S.R.
- Maslach C.
- McPherson J.M.
- Organ D.W.
- Parasuraman S.
- Pleck J.H.
- Poortman A.R.
- Preacher K.J.
- Ruderman M.N.
- Schaufeli W.B.
- SCP.
- Shrout P.E.
- Stewart G.L.
- Tanja van der Lippe
- ten Brummelhuis L.L.
- ten Brummelhuis L.L.
- ten Brummelhuis L.L.
- Van Daalen G.
- Van Steenbergen E.F.
- Vandenberg R.J.
- Voydanoff P.
- Wayne J.H.
- Publication venue
- Publication date
- 01/01/2010
- Field of study
Get PDFHelping behavior at work has become increasingly important, with organizations making more
and more use of cooperative work practices. The difficulty is that employees are facing growing
demands beyond the workplace. This study investigates the mechanisms by which family
involvement (family structure, family tasks, family support) affects helping behavior in teams.
Based on a sample of 495 team members, the results show that having a supportive partner and
performing care tasks increase helping behavior via enhanced fulfillment and skills. Having
young children is directly and negatively related to helping behavior. The authors also conducted
separate analyses for men and women.
Family Involvement and Helping Behavior in Teams
- Author
- Allen T.D.
- Anderson S.E.
- Anderson S.E.
- Andrews A.
- Arbuckle J.L.
- Bakker A.B.
- Bakker A.B.
- Baron R.M.
- Bernasco W.
- Brief A.P.
- Browne M.W.
- Burke R.J.
- Carlson D.S.
- Cinamon R.G.
- De Dreu C.K.W.
- Demerouti E.
- Duxbury L.E.
- Dykstra P.A.
- Eagle B.W.
- Eby L.T.
- Edwards J.
- Elliott M.
- Erickson R.J.
- Esther S. Kluwer
- Flap H.
- Friedman S.D.
- Frone M.R.
- Glass J.L.
- Goode W.J.
- Goodman S.A.
- Graves L.M.
- Greenhaus J.
- Greenhaus J.H.
- Grzywacz J.G.
- Hechter M.
- Hill E.J.
- Hobfoll S.E.
- Hobfoll S.E.
- Hodson R.
- Kalmijn M.
- Kluwer E.S.
- Koster F.
- Lavee Y.
- Lee C.
- Lieke L. ten Brummelhuis
- Loerch K.J.
- Loscocco K.A.
- Lundberg U.
- MacKinnon D.P.
- Marks S.R.
- Maslach C.
- McPherson J.M.
- Organ D.W.
- Parasuraman S.
- Pleck J.H.
- Poortman A.R.
- Preacher K.J.
- Ruderman M.N.
- Schaufeli W.B.
- SCP.
- Shrout P.E.
- Stewart G.L.
- Tanja van der Lippe
- ten Brummelhuis L.L.
- ten Brummelhuis L.L.
- ten Brummelhuis L.L.
- Van Daalen G.
- Van Steenbergen E.F.
- Vandenberg R.J.
- Voydanoff P.
- Wayne J.H.
- Publication venue
- 'SAGE Publications'
- Publication date
- Field of study
No full textLearning and forgetting in the jet fighter aircraft industry
- Author
- A Alchian
- A Alchian
- A Bongers
- A Young
- AD Searle
- AG Loerch
- AnelĂ Bongers
- C.L Benkard
- CL Benkard
- CR Frischtak
- D Besanko
- D Fudenberg
- D Irwin
- D Levhari
- D Levhari
- E Darr
- E Sheshinski
- FC Lane
- G David
- H Asher
- H Gruber
- H Gruber
- J Dutton
- J Hirshleifer
- JE Yelle
- JR Crawford
- K Hartley
- K Hartley
- K Mishina
- K.J Arrow
- L Argote
- L Rapping
- LE Preston
- LM Cabral
- M Kleiner
- M Knaack
- MA Spence
- MarĂa Carmen DĂaz RoldĂĄn
- MB Zimmerman
- N Stokey
- NK Womer
- NK Womer
- NK Womer
- P Dasgupta
- P Joskow
- P Joskow
- P Thompson
- P Thompson
- R Ziemer
- RA Thornton
- S Levitt
- S Rosen
- S Shafer
- SG Sturmey
- TL Smunt
- TP Wright
- UE Reinhardt
- WB Hirschmann
- WY Lee
- WY Oi
- WZ Hirsch
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- Field of study
No full textNew insights into the genetic etiology of Alzheimerâs disease and related dementias
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
No full textCharacterization of the genetic landscape of Alzheimerâs disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/âproxyâ AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele. © 2022, The Author(s)
