Type-III two Higgs doublet model plus a pseudoscalar confronted with h→μτh\rightarrow\mu\tau, muon g−2g-2 and dark matter

In this work, we introduce an extra singlet pseudoscalar into the Type-III two Higgs doublet model (2HDM) which is supposed to solve a series of problems in the modern particle-cosmology. With existence of a light pseudoscalar, the $h\rightarrow\mu\tau$ excess measured at CMS and as well as the $(g-2)_{\mu}$ anomaly could be simultaneously explained within certain parameter spaces that can also tolerate the data on the flavor-violating processes $\tau\rightarrow\mu\gamma$ and Higgs decay gained at LHC. Within the same parameter spaces, the DM relic abundance is well accounted. Moreover, the recently observed Galactic Center gamma ray excess(GCE) is proposed to realize through dark matter(DM) pair annihilations, and in this work, the scenario of the annihilation being mediated by the pseudoscalar is also addressed.Comment: 14 pages, 8 figures, version to appear in NP

Co(salen) catalysed oxidation of synthetic lignin-like polymer: Co(salen) effects

In this paper, Co(salen) [salen = N, N’-bis(salicylidene)ethylenediamine] complex was studied as oxygen activators for the catalytic oxidation of a lignin model polymer using water as the solvent, with molecular oxygen and hydrogen peroxide as the oxidants. The effect of Co(salen) on oxidation was tested by spectroscopic methods (FTIR, 13C-NMR and GC-MS). The reactions catalysed by Co(salen) included Cα-alcohol oxidation, Cα-Cβ side chain cleavage, demethoxylation, aromatic ring cleavage, and β-O-4 cleavage. In addition to the mechanistic information obtained, the effect of Co(salen) suggests that Co(salen) can be important for the catalytic oxidation, as they affect the oxidation of lignin model polymer. The reaction performed in the presence of Co(salen) was more efficient than without it. The formation of aldehyde in the catalytic oxidation, as shown by GC-MS, could be identified as the mechanism of oxidative cleavage of the β-O-4 bonds

Nutrition, Histone Epigenetic Marks, and Disease

The dietary intake of essential nutrients and bioactive food compounds is a process that occurs on a daily basis for the entire life span. Therefore, your diet has a great potential to cause changes in the epigenome. Known histone modifications include acetylation, methylation, biotinylation, poly(ADP-ribosylation), ubiquitination, and sumoylation. Some of these modifications depend directly on dietary nutrients. For other modifications, bioactive dietary compounds may alter the activities of enzymes that establish or remove histone marks, thereby altering the epigenome. This chapter provides an overview of diet-dependent epigenomic marks in histones and their links with human health