(Methanol-κO)(methano­lato-κO)oxido[N-(2-oxidobenzyl­idene)phenyl­alaninato-κ3 O,N,O′]vanadium(V)

In the title complex, [V(C16H13NO3)(CH3O)O(CH3OH)], the VV atom is six-coordinated by a tridentate ligand derived from the condensation of salicyl­aldehyde and l-phenyl­alanine, a vanadyl O atom, a methano­late O atom and a methanol O atom, forming a distorted octa­hedral coordination geometry. In the crystal, inter­molecular O—H⋯O and C—H⋯O hydrogen bonds result in a two-dimensional structure parallel to (001)

The Impact of Green Supply Chain Management (GSCM) on Company’s Operational Performance-Based on the Mediation Effect of Operation Capabilities

Practitioners and researchers are increasingly paying great attention to green supply chain management (GSCM). However, no agreement has been reached on whether GSCM can directly improve company’s operational performance. From the perspective of resource-based view, this paper divides GSCM into internal environment management (IEM) and supplier environment management (SEM), and studies the mechanism of operation capabilities in the relationship between GSCM and company’s operational performance. Our findings suggest that (1) IEM partially improves company’s operational performance through operation capabilities. (2) SEM has positive impact on company’s operational performance through operation capabilities. The conclusion reveals the role of operation capabilities in the relationship between GSCM and company’s operational performance, opening up the black box of the relationship to some extent, which provides guidance for manufacturing companies

Di-μ-oxido-bis­[(4-formyl-2-methoxy­phenolato-κO 1)oxido(1,10-phenan­throline-κ2 N,N′)vanadium(V)]

The title complex, [V2(C8H7O3)2O4(C12H8N2)2], is a centrosymmetric dimer formed by two VV complex units bridged by two μ2-oxido groups. The VV atom is six-coordinated by three oxide O atoms, one O atom from a vanillinate ligand and two N atoms from a 1,10-phenanthroline ligand in a significantly distorted octa­hedral geometry. In the crystal structure, weak inter­molecular C—H⋯O hydrogen bonds connect the mol­ecules into a three-dimensional network

(Methoxo-κO)oxidobis(quinolin-8-olato-κ2 N,O)vanadium(V)

In the title complex, [V(C9H6NO)2(CH3O)O], the central VV atom is coordinated by the O atoms from the oxido and methoxo ligands and the N and O atoms of two bis-chelating quinolin-8-olate ligands, forming a distorted octa­hedral environment. In the crystal structure, weak inter­molecular C—H⋯O hydrogen bonds connect mol­ecules into centrosymmetric dimers which are, in turn, linked by weak C—H⋯π inter­actions into chains along the b axis

Tetra­kis(μ3-2-{[1,1-bis­(hydroxy­meth­yl)-2-oxidoeth­yl]imino­meth­yl}-6-methoxy­phenol­ato)tetra­nickel(II) tetra­hydrate

The title complex, [Ni4(C12H15NO4)4]·4H2O, has crystal­lographic fourfold inversion symmetry, with each NiII ion coordinated in a slightly distorted square-pyramidal coordination environment and forming an Ni4O4 cubane-like core. In the crystal structure, inter­molecular O—H⋯O hydrogen bonds connect complex and water mol­ecules to form a three-dimensional network. The O atom of one of the unique hydroxy­methyl groups is disordered over two sites, with the ratio of occupancies being approximately 0.79:0.21

(2,2′-Bipyridine-κ2 N,N′)[N-(2-oxido-1-naphthyl­idene)threoninato-κ3 O 1,N,O 2]copper(II)

In the title complex, [Cu(C15H13NO4)(C10H8N2)], the Schiff base ligand is derived from the condensation of 2-hydr­oxy-1-naphthaldehyde and l-threonine. The CuII atom is five-coordinated by one N atom and two O atoms from the Schiff base ligand and by two N atoms from a 2,2′-bipyridine ligand in a distorted square-pyramidal geometry. In the crystal structure, the combination of inter­molecular O—H⋯O and C—H⋯O hydrogen bonds leads to a two-dimensional network

Cigarette Smoke-Induced Left Ventricular Remodelling Is Associated With Activation of Mitogen-Activated Protein Kinases

Aim: To determine the effects of cigarette smoke (CS) exposure on the expression/activation of mitogen-activated protein kinases (MAPKs) (extracellular signal-regulated kinase [ERK1/2], p38-kinase [p38] and c-Jun NH2-terminal protein kinase [JNK]), norepinephrine (NE) levels and myocardial structure and function. Methods: Rats were randomised to two groups: CS-exposed (n=12) or room air (CON) (n=10). After 5 weeks, the animals underwent echocardiography with pulse-wave Doppler flow measurements. Hearts were removed for microscopy and Western blot analysis. Results: CS exposure was associated with significant increases in NE urinary levels and larger ventricular dimensions (mm) (CON=left ventricular end diastolic dimension [LVEDD] 7.99±0.10, LVend systolic dimension [LVESD] 4.55±0.20, CS=LVEDD 8.3±0.10, LVESD 5.3±0.09, p=0.026, p=0.003). There was also evidence of systolic dysfunction in the CS-exposed group compared to the CON group (fractional shortening %, CON=43±2, CS=36±.09, p=0.010). In CS-exposed hearts, significant increases in phosphorylated p38/total p38 (0.975±0.05) and phosphorylated ERK1/2/totalERK1/2 (1.919±0.050) were found compared to CON hearts (0.464±0.008, 0.459±0.050, respectively). No significant differences were found in JNK levels between the groups. Conclusions: Increased NE levels and MAPK activation are associated with CS-related left ventricular remodelling. Published by Elsevier B.V. on behalf of European Society of Cardiology