The Carnegie-Irvine Galaxy Survey. III. The Three-Component Structure of Nearby Elliptical Galaxies

Motivated by recent developments in our understanding of the formation and evolution of massive galaxies, we explore the detailed photometric structure of a representative sample of 94 bright, nearby elliptical galaxies, using high-quality optical images from the Carnegie-Irvine Galaxy Survey. The sample spans a range of environments and stellar masses, from M* = 10^{10.2} to 10^{12.0} solar mass. We exploit the unique capabilities of two-dimensional image decomposition to explore the possibility that local elliptical galaxies may contain photometrically distinct substructure that can shed light on their evolutionary history. Compared with the traditional one-dimensional approach, these two-dimensional models are capable of consistently recovering the surface brightness distribution and the systematic radial variation of geometric information at the same time. Contrary to conventional perception, we find that the global light distribution of the majority (>75%) of elliptical galaxies is not well described by a single Sersic function. Instead, we propose that local elliptical galaxies generically contain three subcomponents: a compact (R_e < 1 kpc) inner component with luminosity fraction f ~ 0.1-0.15; an intermediate-scale (R_e ~ 2.5 kpc) middle component with f ~ 0.2-0.25; and a dominant (f = 0.6), extended (R_e ~ 10 kpc) outer envelope. All subcomponents have average Sersic indices n ~ 1-2, significantly lower than the values typically obtained from single-component fits. The individual subcomponents follow well-defined photometric scaling relations and the stellar mass-size relation. We discuss the physical nature of the substructures and their implications for the formation of massive elliptical galaxies.Comment: To appear in The Astrophysical Journal; 36 pages, 2 tables, 38 figures; For the full resolution version, see: http://users.obs.carnegiescience.edu/shuang/PaperIII.pdf ; For the atlas of all selected models, see http://users.obs.carnegiescience.edu/shuang/AppendixE.pd

The Carnegie-Irvine Galaxy Survey. IV. A Method to Determine the Average Mass Ratio of Mergers That Built Massive Elliptical Galaxies

Many recent observations and numerical simulations suggest that nearby massive, early-type galaxies were formed through a "two-phase" process. In the proposed second phase, the extended stellar envelope was accumulated through many dry mergers. However, details of the past merger history of present-day ellipticals, such as the typical merger mass ratio, are difficult to constrain observationally. Within the context and assumptions of the two-phase formation scenario, we propose a straightforward method, using photometric data alone, to estimate the average mass ratio of mergers that contributed to the build-up of massive elliptical galaxies. We study a sample of nearby massive elliptical galaxies selected from the Carnegie-Irvine Galaxy Survey, using two-dimensional analysis to decompose their light distribution into an inner, denser component plus an extended, outer envelope, each having a different optical color. The combination of these two substructures accurately recovers the negative color gradient exhibited by the galaxy as whole. The color difference between the two components ( ~ 0.10 mag; ~ 0.14 mag), based on the slope of the M_stellar-color relation for nearby early-type galaxies, can be translated into an estimate of the average mass ratio of the mergers. The rough estimate, 1:5 to 1:10, is consistent with the expectation of the two-phase formation scenario, suggesting that minor mergers were largely responsible for building up to the outer stellar envelope of present-day massive ellipticals. With the help of accurate photometry, large sample size, and more choices of colors promised by ongoing and future surveys, the approach proposed here can reveal more insights into the growth of massive galaxies during the last few Gyr.Comment: Accepted by ApJ; 20 pages, 11 figures, 1 table; The high resolution figures and the full table can be downloaded from here: https://github.com/dr-guangtou/cgs_colorgra

Determination of a set of constitutive equations for an al-li alloy at SPF conditions

© 2015 The Authors.Uniaxial tensile tests of aluminium-lithium alloy AA1420wereconducted at superplastic forming conditions. The mechanical properties of this Al-Li alloy were then modelled by a set of physicallybased constitutive equations. The constitutive equations describe the isotropic work hardening,recovery and damage by dislocation density changes and grain size evolution. Based on a recent upgraded optimisation technique, the material constants for these constitutive equations were determined

Arsenic trioxide-induced cytotoxicity in small cell lung cancer via altered redox homeostasis and mitochondrial integrity.

Arsenic trioxide (ATO) has demonstrated anticancer activity in different malignancies, especially acute promyelocytic leukemia, with a wide array of putative mechanisms. In this study, we aimed to elucidate the activity and mechanisms of ATO in small cell lung cancer (SCLC). A panel of SCLC cell lines (H841, DMS79, H526, H69 and H187) was employed to demonstrate the activity of ATO. Cell viability, apoptosis and mitochondrial membrane depolarization were assessed. Western blotting was performed to determine the alteration of pro-apoptotic and anti-apoptotic mediators. Reactive oxygen species (ROS) (hydrogen peroxide and superoxide) and intracellular glutathione (GSH) were measured. Antioxidants, N-acetyl-L-cysteine (NAC) and butylated hydroxyanisole (BHA), were applied to restore GSH content and reduce production of ROS. All SCLC cell lines were relatively sensitive to ATO with IC50 values below 10 µM. ATO induced cell death mainly through apoptosis in H841 cells in a dose-dependent manner. Hydrogen peroxide was the major ROS in SCLC cells induced by ATO. Along with GSH depletion and Bcl-2 downregulation, mitochondrial membrane permeabilization was enhanced, followed by release of AIF and SMAC from mitochondria to initiate different cell death pathways. NAC reversed cell death and molecular changes induced by ATO via restoring GSH and reducing ROS content. BHA inhibited hydrogen peroxide production completely and partially restored GSH content accounting for partial reversal of cell inhibition and mitochondrial dysfunction. Nonetheless, ATO reduced both reduced and oxidized form of thioredoxin 1 (Trx1) with no effect on Trx1 redox potential. ATO led to cell death in SCLC mainly through mitochondrial dysfunction, resulting from altered cellular redox homeostasis, namely, hydrogen peroxide generation, GSH depletion and Trx1 downregulation.published_or_final_versio

Numerical simulation of transient force and eddy current loss in a 720-MVA power transformer

Author name used in this publication: S. L. HoAuthor name used in this publication: H. C. WongVersion of RecordPublishe

A 3-D study of eddy current field and temperature rises in a compact bus duct system

Author name used in this publication: S. L. HoAuthor name used in this publication: H. C. WongAuthor name used in this publication: K. W. E. Cheng2005-2006 > Academic research: refereed > Publication in refereed journalVersion of RecordPublishe

Calculations of transient eddy current field and dynamic short circuit forces in a large power transformer

Author name used in this publication: S. L. HoAuthor name used in this publication: S. Y. YangAuthor name used in this publication: H. C. WongRefereed conference paper2005-2006 > Academic research: refereed > Refereed conference paperVersion of RecordPublishe

Calculation of eddy current field in the ascending flange for the bushings and tank wall of a large power transformer

Author name used in this publication: S. L. HoAuthor name used in this publication: K. W. E. Cheng2007-2008 > Academic research: refereed > Publication in refereed journalVersion of RecordPublishe

Accretion-modified Stars in Accretion Disks of Active Galactic Nuclei: the Low-luminosity Cases and an Application to Sgr A\!^{*}

In this paper, we investigate the astrophysical processes of stellar-mass black holes (sMBHs) embedded in advection-dominated accretion flows (ADAFs) of supermassive black holes (SMBHs) in low-luminosity active galactic nuclei (AGNs). The sMBH is undergoing Bondi accretion at a rate lower than the SMBH. Outflows from the sMBH-ADAF dynamically interact with their surroundings and form a cavity inside the SMBH-ADAF, thereby quenching the accretion onto the SMBH. Rejuvenation of the Bondi accretion is rapidly done by turbulence. These processes give rise to quasi-periodic episodes of sMBH activities and create flickerings from relativistic jets developed by the Blandford-Znajek mechanism if the sMBH is maximally rotating. Accumulating successive sMBH-outflows trigger viscous instability of the SMBH-ADAF, leading to a flare following a series of flickerings. Recently, the similarity of near-infrared flare's orbits has been found by GRAVITY/VLTI astrometric observations of Sgr A\!^{*}: their loci during the last 4-years consist of a ring in agreement with the well-determined SMBH mass. We apply the present model to Sgr A\!^{*}, which shows quasi-periodic flickerings. A SMBHH of $\sim 40 M_{\odot}$ is preferred orbiting around the central SMBH of Sgr A\!^{*} from fitting radio to X-ray continuum. Such an extreme mass ratio inspiraling (EMRI) provides an excellent laboratory for LISA, Taiji and Tianqin detection of mHz gravitational waves with strains of $\sim 10^{-17}$, as well as their polarization.Comment: 16 pages, 3 figures, accepted by ApJ

Prevention of colitis-associated colon cancer using a vaccine to target abnormal expression of the MUC1 tumor antigen

Association between chronic inflammation and cancer development is exemplified by inflammatory bowel disease (IBD) where patients with chronic uncontrolled colitis have a significantly increased risk of developing colitis-associated colorectal cancer (CACC). CACC appears to progresses through the inflammation-dysplasia-carcinoma sequence. This highlights the need to identify targets and interventions that reduce inflammation and prevent development of dysplasia in the context of IBD. Using the dextran sulfate sodium (DSS) mouse model of chronic colitis and CACC, we show that an important target of intervention in human disease would be the epithelial cell molecule MUC1 that is aberrantly expressed on inflamed colonocytes and promotes inflammation and progression to CACC. We show that a MUC1 vaccine can ameliorate chronic colitis and prevent development of dysplasia in the colon and thus extend survival in human MUC1 transgenic mice. This study supports the potential of prophylactic vaccines to target antigens that become aberrantly expressed in chronic inflammation (e.g., IBD) and continue to be expressed on the associated cancers (e.g., colon cancer), to prevent and/or treat both diseases