265 research outputs found

    The Correspondence between Convergence Peaks from Weak Lensing and Massive Dark Matter Haloes

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    The convergence peaks, constructed from galaxy shape measurement in weak lensing, is a powerful probe of cosmology as the peaks can be connected with the underlined dark matter haloes. However the capability of convergence peak statistic is affected by the noise in galaxy shape measurement, signal to noise ratio as well as the contribution from the projected mass distribution from the large-scale structures along the line of sight (LOS). In this paper we use the ray-tracing simulation on a curved sky to investigate the correspondence between the convergence peak and the dark matter haloes at the LOS. We find that, in case of no noise and for source galaxies at zs=1z_{\rm s}=1, more than 65%65\% peaks with SNR≥3\text{SNR} \geq 3 (signal to noise ratio) are related to more than one massive haloes with mass larger than 1013M⊙10^{13} {\rm M}_{\odot}. Those massive haloes contribute 87.2%87.2\% to high peaks (SNR≥5\text{SNR} \geq 5) with the remaining contributions are from the large-scale structures. On the other hand, the peaks distribution is skewed by the noise in galaxy shape measurement, especially for lower SNR peaks. In the noisy field where the shape noise is modelled as a Gaussian distribution, about 60%60\% high peaks (SNR≥5\text{SNR} \geq 5) are true peaks and the fraction decreases to 20%20\% for lower peaks (3≤SNR<5 3 \leq \text{SNR} < 5). Furthermore, we find that high peaks (SNR≥5\text{SNR} \geq 5) are dominated by very massive haloes larger than 1014M⊙10^{14} {\rm M}_{\odot}.Comment: 13 pages, 11 figures, 4 tables, accepted for publication in MNRAS. Our mock galaxy catalog is available upon request by email to the author ([email protected]

    Blue straggler evolution caught in the act in the Large Magellanic Cloud globular cluster Hodge 11

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    High-resolution {\sl Hubble Space Telescope} imaging observations show that the radial distribution of the field-decontaminated sample of 162 'blue straggler' stars (BSs) in the 11.7−0.1+0.211.7^{+0.2}_{-0.1} Gyr-old Large Magellanic Cloud cluster Hodge 11 exhibits a clear bimodality. In combination with their distinct loci in color--magnitude space, this offers new evidence in support of theoretical expectations that suggest different BS formation channels as a function of stellar density. In the cluster's color--magnitude diagram, the BSs in the inner 15"" (roughly corresponding to the cluster's core radius) are located more closely to the theoretical sequence resulting from stellar collisions, while those in the periphery (at radii between 85"" and 100"") are preferentially found in the region expected to contain objects formed through binary mass transfer or coalescence. In addition, the objects' distribution in color--magntiude space provides us with the rare opportunity in an extragalactic environment to quantify the evolution of the cluster's collisionally induced BS population and the likely period that has elapsed since their formation epoch, which we estimate to have occurred ∼\sim4--5 Gyr ago.Comment: 13 pages, 4 figure, accepted by Astrophysical Journal Letter

    S-antigen specific T helper type 1 response is present in Behcet’s disease

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    PURPOSE: To investigate the frequency and phenotypic and functional characteristics of S-antigen (S-Ag) specific T cells in patients with Behcet's disease (BD). METHODS: Blood was taken from 23 active BD patients, 12 inactive BD patients, and 14 healthy controls. The clinical features of the patients were summarized. T cell response against 40 mixed S-Ag peptides was identified by interferon gamma (IFN-gamma) enzyme-linked immunospot assay (ELISPOT). CD69 and CD45RO were used to characterize the phenotype of S-Ag specific T cells. The functional property of S-Ag specific T cells was investigated by measuring the production of cytokines. RESULTS: Response to the mixed S-Ag peptides was found in 56.5% and 25% of active and inactive BD patients, respectively. The responsiveness to S-Ag peptides was unrelated to the clinical features of the patients. About 65.8% of IFN-gamma(+) CD4(+) T cells in active BD patients expressed CD69 and CD45RO concomitantly. S-Ag peptides significantly induced a production of IFN-gamma and tumor necrosis factor (TNF)-alpha but not interleukin (IL)-2, IL-4, and IL-17 by peripheral blood mononuclear cells (PBMCs) in active BD patients with a response to S-Ag. CONCLUSIONS: S-Ag specific T cells are present in certain active BD patients, and most of them are activated memory CD4(+) T cells. These T cells may be involved in the pathogenesis of BD via producing Th1-dominant cytokine
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