The progenitors of Type Ia supernovae with long delay times

The nature of the progenitors of Type Ia supernovae (SNe Ia) is still unclear. In this paper, by considering the effect of the instability of accretion disk on the evolution of white dwarf (WD) binaries, we performed binary evolution calculations for about 2400 close WD binaries, in which a carbon--oxygen WD accretes material from a main-sequence star or a slightly evolved subgiant star (WD + MS channel), or a red-giant star (WD + RG channel) to increase its mass to the Chandrasekhar (Ch) mass limit. According to these calculations, we mapped out the initial parameters for SNe Ia in the orbital period--secondary mass ($\log P^{\rm i}-M^{\rm i}_2$) plane for various WD masses for these two channels, respectively. We confirm that WDs in the WD + MS channel with a mass as low as $0.61 M_\odot$ can accrete efficiently and reach the Ch limit, while the lowest WD mass for the WD + RG channel is $1.0 \rm M_\odot$. We have implemented these results in a binary population synthesis study to obtain the SN Ia birthrates and the evolution of SN Ia birthrates with time for both a constant star formation rate and a single starburst. We find that the Galactic SN Ia birthrate from the WD + MS channel is $\sim$$1.8\times 10^{-3} {\rm yr}^{-1}$ according to our standard model, which is higher than previous results. However, similar to previous studies, the birthrate from the WD + RG channel is still low ($\sim$$3\times 10^{-5} {\rm yr}^{-1}$). We also find that about one third of SNe Ia from the WD + MS channel and all SNe Ia from the WD + RG channel can contribute to the old populations (\ga1 Gyr) of SN Ia progenitors.Comment: 11 pages, 9 figures, 1 table, accepted for publication in MNRA

Tissue factor in tumor microenvironment: a systematic review

The aberrant hemostasis is a common manifestation of cancer, and venous thromboembolism (VTE) is the second leading cause of cancer patients’ mortality. Tissue factor (TF), comprising of a 47-kDa transmembrane protein that presents in subendothelial tissues and leukocytes and a soluble isoform, have distinct roles in the initiation of extrinsic coagulation cascade and thrombosis. Laboratory and clinical evidence showed the deviant expression of TF in several cancer systems and its tumor-promoting effects. TF contributes to myeloid cell recruitment in tumor stroma, thereby remodeling of tumor microenvironment. Additionally, the number of TF-positive-microparticles (TF+MP) from tumor origins correlates with the VTE rates in cancer patients. In this review, we summarize our current understanding of the TF regulation and roles in tumor progression and clinical complications

Deterministic versus probabilistic quantum information masking

We investigate quantum information masking for arbitrary dimensional quantum states. We show that mutually orthogonal quantum states can always be served for deterministic masking of quantum information. We further construct a probabilistic masking machine for linearly independent states. It is shown that a set of d dimensional states, $\{ |a_1 \rangle_A, |t a_2 \rangle_A, \dots, |a_n \rangle_A \}$, $n \leq d$, can be probabilistically masked by a general unitary-reduction operation if they are linearly independent. The maximal successful probability of probabilistic masking is analyzed and derived for the case of two initial states.Comment: 5 pages, 1 figure