46 research outputs found

    Maize silage for dairy cows: mitigation of methane emissions can be offset bij and use change

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    Increasing the digestibility of cattle rations by feeding grains and whole plant silages from maize have been identified as effective options to mitigate greenhouse gas emissions. The effect of ploughing grassland for maize crops have not been taken into account yet. A intensive dairy farm is used as an example to demonstrate the trade offs by this type of land use change when more maize silage is fed to dairy cows. The model DAIRY WISE has been used to calculate the mitigation by the changed ration, the Introductory Carbon Balance Model to calculate the changes in soil organic carbon and nitrogen caused by ploughing grassland for maize crops. The losses of soil carbon and the loss of sequestration potential are much larger than the annual mitigation by feeding more maize. The ecosystem carbon payback time defines the years of mitigation that are needed before the emissions due to land use change are compensated. For ploughing grassland on sandy soils, the carbon payback time is 60 years. A higher global warming potential for methane can reduce the carbon payback time with 30%. Ploughing clay soils with a higher equilibrium level of soil organic matter increases the payback time by maximally 70%. The payback times occur only in the case of permanent maize cropping, grass maize rotations cause annual losses of nitrous oxide that are larger than the mitigation by feeding more maize

    Depression and anxiety in people with inflammatory bowel disease.

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    STUDY OBJECTIVE: To determine whether depression or anxiety co-occurs with ulcerative colitis (UC) or Crohn's disease (CD) more often than expected by chance, and, if so, whether the mental disorders generally precede or follow the inflammatory bowel diseases (IBD). DESIGN: Nested case-control studies using a database of linked hospital record abstracts. SETTING: Southern England. MAIN RESULTS: Both depression and anxiety preceded UC significantly more often than would be predicted from the control population's experience. The associations were strongest when the mental conditions were diagnosed shortly before UC, although the association between depression and UC was also significant when depression preceded UC by five or more years. Neither depression nor anxiety occurred before CD more often than expected by chance. However, depression and anxiety were significantly more common after CD; the associations were strongest in the year after the initial record of CD. UC was followed by anxiety, but not by depression, more often than expected by chance and, again, the association was strongest within one year of diagnosis with UC. CONCLUSIONS: The concentration of risk of depression or anxiety one year or less before diagnosis with UC suggests that the two psychiatric disorders might be a consequence of early symptoms of the as yet undiagnosed gastrointestinal condition. The data are also, however, compatible with the hypothesis that the psychiatric disorders could be aetiological factors in some patients with UC. Most of the excess anxiety or depression diagnosed subsequent to diagnosis of IBD occurs during the year after IBD is diagnosed and the probable explanation is that the mental disorders are sequelae of IBD

    Schizophrenia and cancer: an epidemiological study.

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    BACKGROUND: For decades there has been interest in the possibility that people with schizophrenia might have some protection against cancer, and that, if this were so, it might hold clues about aetiological mechanisms in schizophrenia. AIMS: To study cancer incidence in schizophrenia. METHOD: Cohort analysis of linked hospital and death records was used to compare cancer rates in people with schizophrenia with a reference cohort. RESULTS: We did not find a reduced risk for cancer overall (rate ratio 0.99,95% CI 0.90-1.08) or for most individual cancers. There was, however, a significantly low rate ratio for skin cancer (0.56,95% CI 0.36-0.83). CONCLUSIONS: We found no evidence that schizophrenia confers protection against cancer in general. Low rates of cancer are consistent with the hypothesis that sun exposure may influence the development of schizophrenia, although other explanations are also possible
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