Quantum Noise of Kramers-Kronig Receiver

Abstrac--Kramers-Kronig (KK) receiver, which is equivalent to heterodyne detection with one single photodetector, provides an efficient method to reconstruct the complex-valued optical field by means of intensity detection given a minimum-phase signal. In this paper, quantum noise of the KK receiver is derived analytically and compared with that of the balanced heterodyne detection. We show that the quantum noise of the KK receiver keeps the radical fluctuation of the measured signal the same as that of the balanced heterodyne detection, while compressing the tangential noise to 1/3 times the radical one using the information provided by the Hilbert transform. In consequence, the KK receiver has 3/2 times the signal-to-noise ratio of balanced heterodyne detection while presenting an asymmetric distribution of fluctuations, which is also different from that of the latter. More interestingly, the projected in-phase and quadrature field operators of the retrieved signal after down conversion have a time dependent quantum noise distribution depending on the time-varying phase. This property provides a feasible scheme for controlling the fluctuation distribution according to the requirements of measurement accuracy in the specific direction. Under the condition of strong carrier wave, the fluctuations of the component requiring to be measured more accurately can be compressed to 1 / 6, which is even lower than 1/4 by measuring a coherent state. Finally, we prove the analytic conclusions by simulation results

Knockdown of TNFAIP1 mitigates sevoflurane-induced cognitive dysfunction by activating CREB/Nrf2 pathway

Purpose: To investigate the role of tumor necrosis factor-induced protein 1 (TNFAIP1) and cAMPresponsive element binding protein (CREB)/nuclear factor-erythroid factor 2-related factor 2 (Nrf2) pathway in sevoflurane (SEV)-induced cognitive dysfunction. Methods: A SEV-induced cognitive dysfunction rat model was developed. Bcl-2, Bax, heme oxygenase-1, Nrf2, p-CREB, and CREB protein levels in rat hippocampal tissue were assessed by western blot. Learning and long-term memory were evaluated using Morris water maze test. Glutathione peroxidase, malondialdehyde, and superoxide dismutase levels in hippocampal tissue were measured by enzyme-linked immunosorbent assay (ELISA). The 2,7-dichlorodihydro-fluorescein diacetate fluorescent assay was used to measure reactive oxygen species, while TUNEL staining was used to assess neuronal cell apoptosis. Results: Knockdown of TNFAIP1 attenuated SEV-induced learning and long-term memory dysfunction (p < 0.005), oxidative stress (p < 0.005), apoptosis (p < 0.005), and inhibition of the CREB/Nrf2 signaling pathway. Conclusion: This study demonstrates that knockdown of TNFAIP1 alleviates SEV-induced cognitive dysfunction by reversing inhibition of the CREB/Nrf2 signaling pathway. Keywords: TNFAIP1; Postoperative cognitive dysfunction; Sevoflurane; cAMP-responsive element binding protein (CREB); Nuclear factor-erythroid factor 2-related factor 2 (Nrf2

Influence of Heat Treatment on the Morphologies of Copper Nanoparticles Based Films by a Spin Coating Method

We have investigated the influence of heat treatment on the morphologies of copper nanoparticles based films on glass slides by a spin coating method. The experiments show that heat treatment can modify the sizes and morphologies of copper nanoparticles based films on glass slides. We suggest that through changing the parameters of heat treatment process may be helpful to vary the scattering and absorbing intensity of copper nanoparticles when used in energy harvesting/conversion and optical devices