The mean hemoglobin level among men and women according to CD4 count.

<p>The numbers of men with CD4 counts of <50, 50–199, 200–349, and ≥350 cells/mm³ were 604, 474, 272, and 126, respectively. The numbers of women with CD4 counts of <50, 50–199, 200–349, and ≥350 cells/mm³ were 192, 137, 98, and 45, respectively. One-way ANOVA was used to compare hemoglobin levels among patients with different CD4 count. The mean hemoglobin level showed an increasing trend with increasing CD4 count (<i>P</i><0.001 overall, men, and women).</p

Characteristics of non-attendees (N = 314,302) and attendees (266,706) and missing data.

<p>Characteristics of non-attendees (N = 314,302) and attendees (266,706) and missing data.</p

Crude odds ratios and adjusted odds ratios with 95% confidence intervals for non-participation in screening program, according to categories of variables.

<p>The analyses were run on complete samples.</p

Prevalence of anemia, mild anemia, moderate anemia, and severe anemia among patients with different CD4 count.

<p>The numbers of patients with CD4 counts of <50, 50–199, 200–349, and ≥350 cells/mm³ were 796, 611, 370, and 171, respectively. A chi-square test was applied to compare differences in prevalence of anemia, mild anemia, moderate anemia, and severe anemia among patients with different CD4 count. The prevalence of anemia, of mild anemia, of moderate anemia, of severe anemia increased with decreasing CD4 count (<i>P</i><0.001, <i>P</i><0.001, <i>P</i><0.001, <i>P</i> = 0.001).</p

Flowchart of enrolment in the study.

<p>Flowchart of enrolment in the study.</p

Basic characteristics of 1948 newly diagnosed HIV/AIDS patients in China.

<p>Basic characteristics of 1948 newly diagnosed HIV/AIDS patients in China.</p

Prevalence of anemia, mild anemia, moderate anemia, and severe anemia among patients with different age.

<p>The numbers of patients who were 18–39, 40–59, and ≥60 years of age were 1082, 708, and 158, respectively. A chi-square test was applied to compare differences in prevalence of anemia, mild anemia, moderate anemia, and severe anemia among patients with different age. The prevalence of anemia increased with increasing age (<i>P</i> = 0.026). The prevalence of mild anemia increased with increasing age (<i>P</i><0.001). The prevalences of moderate anemia and of severe anemia did not differ significantly according to age (<i>P</i> = 0.098, <i>P</i> = 0.154).</p

SOD1 was a direct target of miR-206.

<p>(A) Putative miR-206-binding sequences in the 3’UTR of SOD1 mRNA, identified by RNAhybrid 2.2. (B) The predicted miR-206-SOD1 binding structure, identified by RNAhybrid 2.2. (C) Schematic diagram of constructed vectors. The 3’UTR region of SOD1 was cloned downstream of the luciferase reporter gene (pGL3-promoter vector). (D) The sequence of the luciferase vector in pGL3-SOD1-UTR-WT and pGL3-SOD1-UTR-Del. (E) Primary canine myocardial cells were transfected with <i>Firefly</i> luciferase expression vectors with pGL3-SOD1-UTR. Luciferase activity was assayed 24 h after transfection. The <i>Firefly</i> luciferase activity of each sample was normalized to <i>Renilla</i> luciferase activity. The normalized luciferase activity of the cells transfected with the control lentivirus was set as 100% relative luciferase activity. The column graphs show the means of at least three independent experiments performed in duplicate. (F) Tissues of the LSFPs were collected in each group. SOD1, and internal standard GAPDH protein levels were detected as above. Western blot bands were normalized to GAPDH. **<i>P</i> < 0.01.</p

Cause-specific hazard ratio of cervical cancer from age 61 to age 80 comparing women screened and unscreened at age 61–65, by screening history at age 51–60, based on Cox regression model.

<p>Cause-specific hazard ratio of cervical cancer from age 61 to age 80 comparing women screened and unscreened at age 61–65, by screening history at age 51–60, based on Cox regression model.</p

Characterization of atrial fibrillation (AF) and AERP in the canine model.

<p>AF was induced by A-TP for 4 weeks. During each week of A-TP, a surface electrocardiogram (ECG) was recorded to determine the presence of AF by turning off the pacemaker. The atrial effective refractory period (AERP) was measured with S1-S2 programmed electrical stimulation, with S2 at coupling intervals starting at 150 ms and progressively shortened by 10 ms decrements and with a 2 × diastolic threshold. The longest S1-S2 coupling interval that failed to result in a propagated atrial response was taken as the local AERP. (A) The level of miR-206 in the lenti-control, lenti-miR-206, and lenti-anti-miR-206 groups two weeks after infection. (B) The AERP in control, A-TP, A-TP plus lenti-miR-206 and A-TP plus lenti-anti-miR-206 groups obtained in posterior wall of left atrium. (C) ECG of the sinus rhythm obtained. (D) ECG of AF obtained. * <i>P</i> < 0.05 and ** <i>P</i> < 0.01.</p