Risk Behavior Correlates of HIV Infection.

<p>*Not estimated due to zero cells</p><p>Risk Behavior Correlates of HIV Infection.</p

Reported sexual behavior and substance use stratified by length of time on the street.

<p>*Does not sum to total due to missing responses from “time on the street”,</p><p>**of those reporting vaginal sex,</p><p>^† of those reporting insertive anal sex,</p><p>^‡ of those reporting receptive anal sex,</p><p>^{<b>^</b>} of those reporting both insertive and receptive anal sex,</p><p>^{<b>^^</b>} of those reporting vaginal sex and insertive and receptive anal sex.</p><p>Reported sexual behavior and substance use stratified by length of time on the street.</p

Socio-demographic characteristics of the sample population stratified by length of time on the street.

<p>*Does not sum to total due to missing responses from “time on the street”.</p><p>**Participants could choose more than one option.</p><p>Socio-demographic characteristics of the sample population stratified by length of time on the street.</p

HIV Prevalence among Male SCY in Kisumu, Kenya.

<p>HIV Prevalence among Male SCY in Kisumu, Kenya.</p

Socio-demographic Correlates of HIV Infection.

<p>* Not estimated due to zero cell.</p><p>Socio-demographic Correlates of HIV Infection.</p

Correlation of Vulnerability to Coercion and of Marginalization with HIV Infection and HIV Risk Behaviors.

<p>Does not add up to N = 296 because participants could choose more than one answer</p><p>*Not estimated due to zero cell.</p><p>^† Fisher’s Exact Test</p><p>Correlation of Vulnerability to Coercion and of Marginalization with HIV Infection and HIV Risk Behaviors.</p

Proposed evolutionary model for ACME acquisition within CC2 clusters and sub-clusters (CC2-I, CC2-II6, CC2-II5, CC2-II85, CC2-II89).

<p>Each dot represents a strain with specific characteristics with respect to ST and content of the different mobile genetic elements, namely, SCC<i>mec</i> (represented by types I-VI), SCC non-<i>mec</i> (represented by the allotype of <i>ccr</i>) and ACME types (represented by Roman numbers followed by Arabic numbers). The occurrence of genetic events involving a single MLST locus variation and/or SCC and ACME acquisition/deletion are indicated by arrows and the elements involved in the event are shown next to the arrow. Blue and white dots represent strains found within the collection studied, and black dots represent hypothetical <i>S. epidermidis</i> strains. ACME I.02 acquisition is represented in blue.</p

Mobilization of ACME in <i>S. epidermidis</i> 1457 by CcrAB.

<p>(A) Pulsed-field gel electrophoresis after <i>Sma</i>I restriction of chromosomal DNA, for the parental strain <i>S. epidermidis</i> 1457 (lane 1) and its ACME excision mutant (lane 2). M = Lambda ladder, and (B) hybridization of <i>Sma</i>I restriction patterns of strain 1457 (lane 1) and its ACME excision mutant (lane 2) with a DNA probe for ACME (<i>arcCB</i>).</p

MOESM2 of Attenuation of Plasmodium falciparum in vitro drug resistance phenotype following culture adaptation compared to fresh clinical isolates in Cambodia

Additional file 2. IC50 obtained from 12 Plasmodium falciparum fresh isolates were higher than when culture-adapted from cryopreserved samples for both the HRP-2 and SYBR Green I assays

MOESM4 of Attenuation of Plasmodium falciparum in vitro drug resistance phenotype following culture adaptation compared to fresh clinical isolates in Cambodia

Additional file 4. Pfmdr1 copy number and alleles found in original and culture-adapted samples of 12 Plasmodium falciparum isolates from Cambodia