27 research outputs found
Proportion of individuals self-reporting as being “definitely a morning person”, average L5 timing, and average of sleep-midpoint across all nights for each genotype group of variants previously reported to be causal for familial advanced sleep phase in the UK Biobank (UKB), Finnish and MESA studies.
Accelerometer-based estimates of sleep timing unavailable in the Finnish studies. Self-reported “morningness” and accelerometer estimates of L5-timing unavailable in MESA.</p
Summary of self-reported sleep duration in UK Biobank (data field 1160) between carriers of variants previously reported to be causal for familial natural short sleep and non-carriers (homozygous reference called by UK Biobank exome-sequencing) who are also non-carriers for any of the other 12 variants described in this article.
Summary of self-reported sleep duration in UK Biobank (data field 1160) between carriers of variants previously reported to be causal for familial natural short sleep and non-carriers (homozygous reference called by UK Biobank exome-sequencing) who are also non-carriers for any of the other 12 variants described in this article.</p
Summary of average accelerometer derived sleep duration (hours) (all nights) in the subset of exome-sequenced unrelated Europeans in UK Biobank, split by carriers and non-carriers for variants previously reported to be causal for familial natural short sleep.
Summary of average accelerometer derived sleep duration (hours) (all nights) in the subset of exome-sequenced unrelated Europeans in UK Biobank, split by carriers and non-carriers for variants previously reported to be causal for familial natural short sleep.</p
Summary statistics of “eveningness” across genotype groups for variants previously reported as causal for delayed sleep phase.
Data on being “more or definitely an evening person” unavailable in the Finnish studies. (DOCX)</p