Effect of Impurities on the Superheating field of Type II superconductors

We consider the effect of nonmagnetic and magnetic impurities on the superheating field $H_s$ in a type-II superconductor. We solved the Eilenberger equations, which take into account the nonlinear pairbreaking of Meissner screening currents, and calculated $H_s(T)$ for arbitrary temperatures and impurity concentrations in a single-band s-wave superconductor with a large Ginzburg-Landau parameter. At low temperatures nonmagnetic impurities suppress a weak maximum in $H_s(T)$ which has been predicted for the clean limit, resulting instead in a maximum of $H_s$ as a function of impurity concentration in a moderately clean limit. It is shown that nonmagnetic impurities weakly affect $H_s$ even in the dirty limit, while magnetic impurities suppress both $H_s$ and the critical temperature $T_c$. The density of quasiparticles states $N(\epsilon)$ is strongly affected by an interplay of impurity scattering and current pairbreaking. We show that a clean superconductor at $H=H_s$ is in a gapless state, but a quasiparticle gap $\epsilon_g$ in $N(\epsilon)$ at $H=H_s$ appears as the concentration of nonmagnetic impurities increases. As the nonmagnetic scattering rate $\alpha$ increases above $\alpha_c=0.36$, the quasiparticle gap $\epsilon_g(\alpha)$ at $H=H_s$ increases, approaching $\epsilon_g\approx 0.32\Delta_0$ in the dirty limit $\alpha\gg 1$, where $\Delta_0$ is the superconducting gap parameter at zero field. The effects of impurities on $H_s$ can be essential for the nonlinear surface resistance and superconductivity breakdown by strong RF fields.Comment: 12 pages, 13 figure

Glucocorticoid Receptor and Adipocyte Biology.

Glucocorticoids are steroid hormones that play a key role in metabolic adaptations during stress, such as fasting and starvation, in order to maintain plasma glucose levels. Excess and chronic glucocorticoid exposure, however, causes metabolic syndrome including insulin resistance, dyslipidemia, and hyperglycemia. Studies in animal models of metabolic disorders frequently demonstrate that suppressing glucocorticoid signaling improves insulin sensitivity and metabolic profiles. Glucocorticoids convey their signals through an intracellular glucocorticoid receptor (GR), which is a transcriptional regulator. The adipocyte is one cell type that contributes to whole body metabolic homeostasis under the influence of GR. Glucocorticoids' functions on adipose tissues are complex. Depending on various physiological or pathophysiological states as well as distinct fat depots, glucocorticoids can either increase or decrease lipid storage in adipose tissues. In rodents, glucocorticoids have been shown to reduce the thermogenic activity of brown adipocytes. However, in human acute glucocorticoid exposure, glucocorticoids act to promote thermogenesis. In this article, we will review the recent studies on the mechanisms underlying the complex metabolic functions of GR in adipocytes. These include studies of the metabolic outcomes of adipocyte specific GR knockout mice and identification of novel GR primary target genes that mediate glucocorticoid action in adipocytes

The leafage of a chordal graph

The leafage l(G) of a chordal graph G is the minimum number of leaves of a tree in which G has an intersection representation by subtrees. We obtain upper and lower bounds on l(G) and compute it on special classes. The maximum of l(G) on n-vertex graphs is n - lg n - (1/2) lg lg n + O(1). The proper leafage l*(G) is the minimum number of leaves when no subtree may contain another; we obtain upper and lower bounds on l*(G). Leafage equals proper leafage on claw-free chordal graphs. We use asteroidal sets and structural properties of chordal graphs.Comment: 19 pages, 3 figure

Decoherence and Recoherence in Model Quantum Systems

We discuss the various manifestations of quantum decoherence in the forms of dephasing, entanglement with the environment, and revelation of "which-path" information. As a specific example, we consider an electron interference experiment. The coupling of the coherent electrons to the quantized electromagnetic field illustrates all of these versions of decoherence. This decoherence has two equivalent interpretations, in terms of photon emission or in terms of Aharonov-Bohm phase fluctuations. We consider the case when the coherent electrons are coupled to photons in a squeezed vacuum state. The time-averaged result is increased decoherence. However, if only electrons which are emitted during selected periods are counted, the decoherence can be suppressed below the level for the photon vacuum. This is the phenomenon of recoherence. This effect is closely related to the quantum violations of the weak energy condition, and is restricted by similar inequalities. We give some estimates of the magnitude of the recoherence effect and discuss prospects for observing it in an electron interferometry experiment.Comment: 8 pages, 3 figures, talk presented at the 7th Friedmann Seminar, Joao Pessoa, Brazil, July 200

Polarization conversion spectroscopy of hybrid modes

Enhanced polarization conversion in reflection for the Otto and Kretschmann configurations is introduced as a new method for hybrid-mode spectroscopy. Polarization conversion in reflection appears when hybrid-modes are excited in a guiding structure composed of at least one anisotropic media. In contrast to a dark dip, in this case modes are associated to a peak in the converted reflectance spectrum, increasing the detection sensitivity and avoiding confusion with reflection dips associated with other processes as can be transmission.Comment: 4 pages, 4 figure

Rapid degradation of mutant SLC25A46 by the ubiquitin-proteasome system results in MFN1/2-mediated hyperfusion of mitochondria.

SCL25A46 is a mitochondrial carrier protein that surprisingly localizes to the outer membrane and is distantly related to Ugo1. Here we show that a subset of SLC25A46 interacts with mitochondrial dynamics components and the MICOS complex. Decreased expression of SLC25A46 results in increased stability and oligomerization of MFN1 and MFN2 on mitochondria, promoting mitochondrial hyperfusion. A mutation at L341P causes rapid degradation of SLC25A46, which manifests as a rare disease, pontocerebellar hypoplasia. The E3 ubiquitin ligases MULAN and MARCH5 coordinate ubiquitylation of SLC25A46 L341P, leading to degradation by organized activities of P97 and the proteasome. Whereas outer mitochondrial membrane-associated degradation is typically associated with apoptosis or a specialized type of autophagy termed mitophagy, SLC25A46 degradation operates independently of activation of outer membrane stress pathways. Thus SLC25A46 is a new component in mitochondrial dynamics that serves as a regulator for MFN1/2 oligomerization. Moreover, SLC25A46 is selectively degraded from the outer membrane independently of mitophagy and apoptosis, providing a framework for mechanistic studies in the proteolysis of outer membrane proteins

Experimental Examination of the Effect of Short Ray Trajectories in Two-port Wave-Chaotic Scattering Systems

Predicting the statistics of realistic wave-chaotic scattering systems requires, in addition to random matrix theory, introduction of system-specific information. This paper investigates experimentally one aspect of system-specific behavior, namely the effects of short ray trajectories in wave-chaotic systems open to outside scattering channels. In particular, we consider ray trajectories of limited length that enter a scattering region through a channel (port) and subsequently exit through a channel (port). We show that a suitably averaged value of the impedance can be computed from these trajectories and that this can improve the ability to describe the statistical properties of the scattering systems. We illustrate and test these points through experiments on a realistic two-port microwave scattering billiard.Comment: 14 pages, 9 figure

Parental Co‐Construction of 5‐ to 13‐Year‐Olds\u27 Global Self‐Esteem Through Reminiscing About Past Events

The current study explored parental processes associated with children\u27s global self‐esteem development. Eighty 5‐ to 13‐year‐olds and one of their parents provided qualitative and quantitative data through questionnaires, open‐ended questions, and a laboratory‐based reminiscing task. Parents who included more explanations of emotions when writing about the lowest points in their lives were more likely to discuss explanations of emotions experienced in negative past events with their child, which was associated with child attachment security. Attachment was associated with concurrent self‐esteem, which predicted relative increases in self‐esteem 16 months later, on average. Finally, parent support also predicted residual increases in self‐esteem. Findings extend prior research by including younger ages and uncovering a process by which two theoretically relevant parenting behaviors impact self‐esteem development