4 research outputs found
Comparison of Guided Bone Regeneration using a Bovine Collagen Membrane vs a Calcium Sulfate Barrier
- Author
- Aichelmann-Reidy ME Heath CD, Reynolds MA.
- Al-Arrayed F Adam S, Moran J, Dowell P.
- Andreana S Cornelini R, Edsberg LE, Natiella JR.
- Anson D.
- Anson D.
- Bell WH.
- Benque E Zahedi S, Brocard D, Oscaby F, Justumus P, Brunel G.
- Buser DA Dahlen C, Schenk RK,
- Buser DA Dula K, Belser U, Hirt HP, Berthold H.
- Buser DA Dula K, Belser U, Hirt HP, Berthold H.
- Chen CC Wang HL, Smith F, Glickman GN, Shyr Y, O'Neal RB.
- Chung KM Salkin LM, Stein MD, Freedman AL.
- Couri CJ Maze GI, Hinkson DW, Collins III BH, Dawson DV.
- De Leonardis D Pecora GE.
- Evans GH Yukna RA, Cambre KM, Gardiner DL.
- Friedmann A Strietzel FP, Maretzki B, Bernimoulin JP.
- Friedmann A Strietzel FP, Maretzki B, Pitaru S, Bernimoulin JP.
- Gao C Gao J, You X, et al.
- Gottlow J.
- Guarnieri R Pecora G, Fini M, et al.
- Harris RJ.
- Iasella JM Greenwell H, Miller RL, et al.
- Kim C-K Chai J-K, Cho K-S, et al.
- Kirkland G Greenwell H, Drisko C, Wittwer JW, Yancey J, Rebitski G.
- Mamidwar SS Ricci JL, Alezander H.
- Maragos P Bissada NF, Wang R, Cole BP.
- Mattson JS McLey LL, Jabro MH.
- Melcher AH.
- Orsini M Orsini G, Benlloch D, et al.
- Payne JM Cobb CM, Rapley JW, Killoy WJ, Spencer P.
- Pecora G Andreana S, Margarone JE, Covani U, Sottosanti JS.
- Peltier LF.
- Quteish D Dolby AE.
- Setya AB Bissada NF.
- Shaffer CD App GR.
- Simon BI Von Hagen S, Deasy MJ, Faldu M, Resnansky D.
- Sottosanti JS.
- Strocchi R Orsini G, Iezzi G, et al.
- Vance GS Greenwell H, Miller RL, Hill M, Johnston H, Scheetz JP.
- Wang HL Boyapati L.
- Wang HL MacNei RL.
- Wang HL O'Neal RB, MacNeil LM.
- Wang HL O'Neal RB, Thomas CL, Shyr Y, MacNeil RL.
- Zitzmann NU Naef R, Scharer P.
- Publication venue
- 'Jaypee Brothers Medical Publishing'
- Publication date
- Field of study
Instrumentation for Spectroscopy and other Applications
- Author
- A Cornu
- A Gabriel
- A Guinier
- A Harmsen
- A Malherbe
- AH Compton
- AL Robinson
- AN Zaidel
- AP Lukirskii
- AP Lukirskii
- AP Lukirskii
- AP Lukirskii
- AV Vinogradov
- B Buras
- B Buras
- B Feuerbacher
- B Niemann
- B Niemann
- B Saloman
- BF Sonntag
- BJ Leigh
- BJ Leigh
- BK Tanner
- BL Bracewell
- BL Henke
- BL Henke
- BM Kincaid
- BP Feuerbacher
- BR Sandel
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- C Gähwiller
- C Kunz
- C Kunz
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- C Vogel
- CE Winkler
- CJ Borkowski
- CJ Sparks
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- D Reinke
- D Rudolph
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- DE Eastman
- DE Sayers
- DE Sayers
- DJ Michels
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- DW Berreman
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- E Spiller
- E Spiller
- E Spiller
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- EE Koch
- EE Koch
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- EM Rowe
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- EM Rowe
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- FC Brown
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- G Schmahl
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- S Aoki
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/1979
- Field of study
Publications
- Author
- -Hollande A., Valentin, J.
- 1802a.Wiese L.
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/1973
- Field of study
Evolocumab and clinical outcomes in patients with cardiovascular disease
- Author
- Abelson M
- Aboufakher R
- Abrahamsen Te
- Accini Mendoza Jl
- Acheatel R
- Ackermann D
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- Zilahi Z
- Zrazhevskiy K
- Ă–stergren J
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
BACKGROUND Evolocumab is a monoclonal antibody that inhibits proprotein convertase subtilisin-kexin type 9 (PCSK9) and lowers low-density lipoprotein (LDL) cholesterol levels by approximately 60%. Whether it prevents cardiovascular events is uncertain. METHODS We conducted a randomized, double-blind, placebo-controlled trial involving 27,564 patients with atherosclerotic cardiovascular disease and LDL cholesterol levels of 70 mg per deciliter (1.8 mmol per liter) or higher who were receiving statin therapy. Patients were randomly assigned to receive evolocumab (either 140 mg every 2 weeks or 420 mg monthly) or matching placebo as subcutaneous injections. The primary efficacy end point was the composite of cardiovascular death, myocardial infarction, stroke, hospitalization for unstable angina, or coronary revascularization. The key secondary efficacy end point was the composite of cardiovascular death, myocardial infarction, or stroke. The median duration of follow-up was 2.2 years. RESULTS At 48 weeks, the least-squares mean percentage reduction in LDL cholesterol levels with evolocumab, as compared with placebo, was 59%, from a median baseline value of 92 mg per deciliter (2.4 mmol per liter) to 30 mg per deciliter (0.78 mmol per liter) (P<0.001). Relative to placebo, evolocumab treatment significantly reduced the risk of the primary end point (1344 patients [9.8%] vs. 1563 patients [11.3%]; hazard ratio, 0.85; 95% confidence interval [CI], 0.79 to 0.92; P<0.001) and the key secondary end point (816 [5.9%] vs. 1013 [7.4%]; hazard ratio, 0.80; 95% CI, 0.73 to 0.88; P<0.001). The results were consistent across key subgroups, including the subgroup of patients in the lowest quartile for baseline LDL cholesterol levels (median, 74 mg per deciliter [1.9 mmol per liter]). There was no significant difference between the study groups with regard to adverse events (including new-onset diabetes and neurocognitive events), with the exception of injection-site reactions, which were more common with evolocumab (2.1% vs. 1.6%). CONCLUSIONS In our trial, inhibition of PCSK9 with evolocumab on a background of statin therapy lowered LDL cholesterol levels to a median of 30 mg per deciliter (0.78 mmol per liter) and reduced the risk of cardiovascular events. These findings show that patients with atherosclerotic cardiovascular disease benefit from lowering of LDL cholesterol levels below current targets