1,198 research outputs found
Air Force joint coordination meeting at IBM on January 13, 1955
In attendance: C. A. Rice, F. Moore, P. K. Philhower, C. F. Lynch, C. Watt, J. F. Martell, M. R. Zale, R. J. Whalen, Major Zaney, G. Solomon, J. Fraser, F. Paul, D. M. Gallagher, G. Cullen, Major D. H. Vlcek, Lt. F. Harding, Mr. Widmaier, R. V. Baylis.Minutes of the Air Force joint coordination meeting discussing XD-1 and XD-2 progress; the production engineering aspect of work on the duplex machine; and manufacturing activities
Selected Characteristics of North Dakota Farm Families Engaged in Sustainable Agricultural Practices
Environmental Economics and Policy, Farm Management, Resource /Energy Economics and Policy,
Extracting the Distribution Amplitudes of the rho meson from the Color Glass Condensate
We extract the leading twist-2 and subleading twist-3 Distribution Amplitudes
(DAs) of the rho meson using the HERA data on diffractive rho photoproduction.
We do so using several Colour Glass Condensate (CGC) inspired and a Regge
inspired dipole models. We find that our extracted twist-2 DA is not much model
dependent and is consistent with QCD Sum Rules and lattice predictions. The
extracted twist-3 DA is more model dependent but is still consistent with the
Sum Rules prediction.Comment: 21 pages, 10 figures, 3 tables. Section 6 revised, figures 8 and 9
and table 3 updated. Conclusions essentially unchange
BAFF and MyD88 signals promote a lupuslike disease independent of T cells
Systemic lupus erythernatosus (SLE) is a systemic autoimmune disease characterized by the production of autoantibodies. However, the underlying cause of disease appears to relate to defects in T cell tolerance or T cell help to 13 cells. Transgenic (Tg) mice over-expressing the cytokine 13 cell-activating factor of the tumor necrosis factor family (BAFF) develop an autoimmune disorder similar to SLE and show impaired B cell tolerance and altered T cell differentiation. We generated BAFF Tg mice that were completely deficient in T cells, and, surprisingly, these mice developed an SLE-like disease indistinguishable from that of BAFF Tg mice. Autoimmunity in BAFF Tg mice did, however, require 13 cell-intrinsic signals through the Toll-like receptor (TLR)-associated signaling adaptor MyD88, which controlled the production of proinflammatory autoantibody isotypes. TLR7/9 activation strongly up-regulated expression of transmembrane activator and calcium modulator and cyclophilin ligand interactor (TACI), which is a receptor for BAFF involved in 13 cell responses to T cell-independent antigens. Moreover, BAFF enhanced TLR7/9 expression on 13 cells and TLR-mediated production of autoantibodies. Therefore, autoirnmunity in BAFF Tg mice results from altered 13 cell tolerance, but requires TLR signaling and is independent of T cell help. It is possible that SLE patients with elevated levels of BAFF show a similar basis for disease
Prompt photon and associated heavy quark production at hadron colliders with kt-factorization
In the framework of the kt-factorization approach, the production of prompt
photons in association with a heavy (charm or beauty) quarks at high energies
is studied. The consideration is based on the O(\alpha \alpha_s^2) off-shell
amplitudes of gluon-gluon fusion and quark-(anti)quark interaction
subprocesses. The unintegrated parton densities in a proton are determined
using the Kimber-Martin-Ryskin prescription. The analysis covers the total and
differential cross sections and extends to specific angular correlations
between the produced prompt photons and muons originating from the semileptonic
decays of associated heavy quarks. Theoretical uncertainties of our evaluations
are studied and comparison with the results of standard NLO pQCD calculations
is performed. Our numerical predictions are compared with the recent
experimental data taken by the D0 and CDF collaborations at the Tevatron.
Finally, we extend our results to LHC energies.Comment: 14 pages, 10 figure
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