Volcanism by melt-driven Rayleigh-Taylor instabilities and possible consequences of melting for admittance ratios on Venus

A large number of volcanic features exist on Venus, ranging from tens of thousands of small domes to large shields and coronae. It is difficult to reconcile all these with an explanation involving deep mantle plumes, since a number of separate arguments lead to the conclusion that deep mantle plumes reaching the base of the lithosphere must exceed a certain size. In addition, the fraction of basal heating in Venus' mantle may be significantly lower than in Earth's mantle reducing the number of strong plumes from the core-mantle boundary. In three-dimensional convection simulations with mainly internal heating, weak, distributed upwellings are usually observed. We present an alternative mechanism for such volcanism, originally proposed for the Earth and for Venus, involving Rayleigh-Taylor instabilities driven by melt buoyancy, occurring spontaneously in partially or incipiently molten regions

Unifying metastasis--Integrating intravasation, circulation and end organ colonization

Recent technological advances that have enabled the measurement of circulating tumour cells (CTCs) in patients have spurred interest in the circulatory phase of metastasis. Techniques that do not solely rely on a blood sample allow substantial biological interrogation beyond simply counting CTCs

The Zero Age Main Sequence of WIMP burners

We modify a stellar structure code to estimate the effect upon the main sequence of the accretion of weakly interacting dark matter onto stars and its subsequent annihilation. The effect upon the stars depends upon whether the energy generation rate from dark matter annihilation is large enough to shut off the nuclear burning in the star. Main sequence WIMP burners look much like protostars moving on the Hayashi track, although they are in principle completely stable. We make some brief comments about where such stars could be found, how they might be observed and more detailed simulations which are currently in progress. Finally we comment on whether or not it is possible to link the paradoxically young OB stars found at the galactic centre with WIMP burners.Comment: 4 pages, 3 figs. Matches published versio

JPL Ephemeris Tapes E9510, E9511, and E9512

The first issue of JPL Ephemeris Tapes is described. These tapes carry the positions and velocities of the planets and of the Moon, plus nutations and nutation rates in longitude and obliquity, together with second and fourth modified differences, for the interval December 30, 1949, to January 5, 2000

Ventricular Tachycardia in the Absence of Structural Heart Disease

In up to 10% of patients who present with ventricular tachycardia (VT), obvious structural heart disease is not identified. In such patients, causes of ventricular arrhythmia include right ventricular outflow tract (RVOT) VT, extrasystoles, idiopathic left ventricular tachycardia (ILVT), idiopathic propranolol-sensitive VT (IPVT), catecholaminergic polymorphic VT (CPVT), Brugada syndrome, and long QT syndrome (LQTS). RVOT VT, ILVT, and IPVT are referred to as idiopathic VT and generally do not have a familial basis. RVOT VT and ILVT are monomorphic, whereas IPVT may be monomorphic or polymorphic. The idiopathic VTs are classified by the ventricle of origin, the response to pharmacologic agents, catecholamine dependence, and the specific morphologic features of the arrhythmia. CPVT, Brugada syndrome, and LQTS are inherited ion channelopathies. CPVT may present as bidirectional VT, polymorphic VT, or catecholaminergic ventricular fibrillation. Syncope and sudden death in Brugada syndrome are usually due to polymorphic VT. The characteristic arrhythmia of LQTS is torsades de pointes. Overall, patients with idiopathic VT have a better prognosis than do patients with ventricular arrhythmias and structural heart disease. Initial treatment approach is pharmacologic and radiofrequency ablation is curative in most patients. However, radiofrequency ablation is not useful in the management of inherited ion channelopathies. Prognosis for patients with VT secondary to ion channelopathies is variable. High-risk patients (recurrent syncope and sudden cardiac death survivors) with inherited ion channelopathies benefit from implantable cardioverter-defibrillator placement. This paper reviews the mechanism, clinical presentation, and management of VT in the absence of structural heart disease

Formation of fundamental structures in Bose-Einstein Condensates

The meanfield interaction in a Bose condensate provides a nonlinearity which can allow stable structures to exist in the meanfield wavefunction. We discuss a number of examples where condensates, modelled by the one dimensional Gross Pitaevskii equation, can produce gray solitons and we consider in detail the case of two identical condensates colliding in a harmonic trap. Solitons are shown to form from dark interference fringes when the soliton structure, constrained in a defined manner, has lower energy than the interference fringe and an analytic expression is given for this condition.Comment: 7 pages, 3 figures, requires ioplppt.st

The decay and collisions of dark solitons in superfluid Fermi gases

We study soliton collisions and the decay of solitons into sound in superfluid Fermi gases across the Bose-Einstein condensate to Bardeen-Cooper-Schrieffer (BEC-BCS) crossover by performing numerical simulations of the time-dependent Bogoliubov-de Gennes equations. This decay process occurs when the solitons are accelerated to the bulk pair-breaking speed by an external potential. A similar decay process may occur when solitons are accelerated by an inelastic collision with another soliton. We find that soliton collisions become increasingly inelastic as we move from the BEC to BCS regimes, and the excess energy is converted into sound. We interpret this effect as being due to evolution of Andreev bound states localized within the soliton.Comment: 9 pages, 5 figure