New physics searches at the ILC positron and electron beam dumps

We study capability of the ILC beam dump experiment to search for new physics, comparing the performance of the electron and positron beam dumps. The dark photon, axion-like particles, and light scalar bosons are considered as new physics scenarios, where all the important production mechanisms are included: electron-positron pair-annihilation, Primakoff process, and bremsstrahlung productions. We find that the ILC beam dump experiment has higher sensitivity than past beam dump experiments, with the positron beam dump having slightly better performance for new physics particles which are produced by the electron-positron pair-annihilation.Comment: 26 pages, 7 figure

Abstraction Model of Probing and DFA Attacks on Block Ciphers

A thread of physical attacks that try to obtain secret information from cryptographic modules has been of academic and practical interest. One of the concerns is determining its efficiency, e.g., the number of attack trials to recover the secret key. However, the accurate estimation of the attack efficiency is generally expensive because of the complexity of the physical attack on a cryptographic algorithm. Based on this background, in this study, we propose a new abstraction model for evaluating the attack efficiency of the probing and DFA attacks. The proposed model includes an abstracted attack target and attacker to determine the amount of leaked information obtained in a single attack trial. We can adapt the model flexibly to various attack scenarios and can get the attack efficiency quickly and precisely. In the probing attack on AES, the difference in the attack efficiency is only approximately 0.3% between the model and experimental values, whereas that of a previous model is approximately 16%. We also apply the probing attack on DES, and the results show that DES has a high resistance to the probing attack. Moreover, the proposed model works accurately also for the DFA attack on AES

Chronological urodynamic evaluation of changing bladder and urethral functions after robot-assisted radical prostatectomy

Objective To examine chronological changes in urethral and bladder functions before, immediately after, and 1 year after robot-assisted radical prostatectomy (RARP), urodynamic studies were prospectively performed. Methods Sixty-three consecutive patients underwent pressure-flow studies, urethral pressure profiles, and abdominal leak point pressure (ALPP) tests 1-2 days before, immediately after, and 1 year after RARP. Results The mean bladder compliance was 28.3 mL/cm H2O before RARP; it worsened to 16.3 mL/cm H2O immediately after RARP and recovered to 27.1 mL/cm H2O at 1 year. The mean detrusor pressure at maximum flow rate was 61.9 cm H2O before RARP; it decreased to 34.3 cm H2O immediately after RARP and remained at 35.6 cm H2O at 1 year. The mean maximum urethral closure pressure was 84.2 cm H2O before RARP; it decreased to 33.4 cm H2O immediately after RARP and recovered to 63.0 cm H2O at 1 year. Intrinsic sphincter deficiency (ISD) evaluated by the ALPP test was observed in 53 patients immediately after RARP, although no patient showed ISD before RARP. ISD remained in 7 patients at 1 year. Both ALPP and maximum urethral closure pressure at 1 year were significant factors for continence in multivariate analysis. Conclusion Urethral sphincter and bladder function worsen immediately after RARP and recover over time. The bladder storage function after RARP returns to almost the same level before RARP, and the voiding function improves compared with the condition before RARP; however, the urethral sphincter function does not return to its preoperative level. Urethral sphincter dysfunction is considered the main factor for urinary incontinence after RARP. © 2015 Elsevier Inc.Embargo Period 12 month

Dose Measurements through the Concrete and Iron Shields under the 100 to 400 MeV Quasi-Monoenergetic Neutron Field (at RCNP, Osaka Univ.)

Shielding benchmark experiments are useful to verify the accuracy of calculation methods for the radiation shielding designs of high-energy accelerator facilities. In the present work, the benchmark experiments were carried out for 244- and 387-MeV quasi-monoenergetic neutron field at RCNP of Osaka University. Neutron dose rates through the test shields, 100-300 cm thick concrete and 40-100 cm thick iron, were measured by four kinds of neutron dose equivalent monitors, three kinds of wide-energy range monitors applied to high-energy neutron fields above 20 MeV and a conventional type rem monitor for neutrons up to 20 MeV, placed behind the test shields. Measured dose rates were compared one another. Measured results with the wide-energy range monitors were in agreement one another for both the concrete and the iron shields. For the conventional type rem monitor, measured results are smaller than those with the wide-energy range monitors for the concrete shields, while that are in agreements for the iron shields. The attenuation lengths were obtained from the measurements. The lengths from all the monitors are in agreement on the whole, though some differences are shown. These results are almost same as those from others measured at several hundred MeV neutron fields

Redundant roles of EGFR ligands in the ERK activation waves during collective cell migration

Epidermal growth factor receptor (EGFR) plays a pivotal role in collective cell migration by mediating cell-to-cell propagation of extracellular signal-regulated kinase (ERK) activation. Here, we aimed to determine which EGFR ligands mediate the ERK activation waves. We found that epidermal growth factor (EGF)–deficient cells exhibited lower basal ERK activity than the cells deficient in heparin-binding EGF (HBEGF), transforming growth factor alpha (TGFα) or epiregulin (EREG), but all cell lines deficient in a single EGFR ligand retained the ERK activation waves. Surprisingly, ERK activation waves were markedly suppressed, albeit incompletely, only when all four EGFR ligands were knocked out. Re-expression of the EGFR ligands revealed that all but HBEGF could restore the ERK activation waves. Aiming at complete elimination of the ERK activation waves, we further attempted to knockout NRG1, a ligand for ErbB3 and ErbB4, and found that NRG1-deficiency induced growth arrest in the absence of all four EGFR ligand genes. Collectively, these results showed that EGFR ligands exhibit remarkable redundancy in the propagation of ERK activation waves during collective cell migration

Green emitting β‐SiAlON:Eu2+ phosphors derived from chemically modified perhydropolysilazanes

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