Association between ocular dominance and average macular ganglion cell-inner plexiform layer thickness.

<p>Association between ocular dominance and average macular ganglion cell-inner plexiform layer thickness.</p

Univariate linear regression analysis of demographics and clinical variables: effect on average macular ganglion cell-inner plexiform layer thickness.

<p>Univariate linear regression analysis of demographics and clinical variables: effect on average macular ganglion cell-inner plexiform layer thickness.</p

Representative case showing the characteristic of ganglion cell-inner plexiform layer (GCIPL) associated with ocular dominance.

<p>Images from a 36-year-old man with ocular dominance in his right eye. The average GCIPL thickness (84 μm) of the dominant eye was greater than that (78 μm) in the non-dominant eye. The average inferior GCIPL thickness of the dominant eye (84 μm) was also greater than that (78 μm) in the non-dominant eye.</p

A 40-year-old female with primary open-angle glaucoma.

<p>This patient had recurrent disc hemorrhages (DHs) at the superotemporal and inferotemporal locations at the borders of localized retinal nerve fiber layer defects (A, black arrows). Disc filling defect is present on fluorescein angiography at the inferotemporal location (white dotted area), where a previous DH had occurred (B and C).</p

Fluorescein angiography of normal control eye (A) and glaucoma eyes with disc hemorrhage (DH) (B and C).

<p>The angiography of normal control eye was from a patient who underwent this examination due to unknown blurred vision (this case is not included in the analysis of the present study, however, is added in this figure to help comparison with glaucoma eyes). Arteriovenous transit time is the time between the entrance of the opaque substance into the retinal artery (arrowhead in A-1, B-1, C-1) and entrance into the vein (arrowhead in A-2, B-2, C-2) from a distance of 2 optic discs. Glaucomatous eye with DH occurring at the border of localized retinal nerve fiber layer (RNFL) defect (B’ and B”; localized RNFL defect, arrowhead in B’) shows prolonged arteriovenous transit time (time interval between B-1, when fluorescein dye is introduced into the artery, and B-2, when fluorescein dye enters the vein from a distance of 2 optic discs) (delayed by 3 seconds). Glaucomatous eye with DH occurring at the 6 o’clock position not related to a localized RNFL defect (C’ and C”) shows prolonged arm-retina time, which is the time from the administration of fluorescein dye to the antecubital vein until it becomes visible in the retinal arteries (C-1, 23 seconds).</p

Inter-ocular comparisons of clinical characteristics according to ocular dominance.

<p>Inter-ocular comparisons of clinical characteristics according to ocular dominance.</p

Inter-ocular comparisons of the macular ganglion cell-inner plexiform layer (GCIPL) according to ocular dominance.

<p>Inter-ocular comparisons of the macular ganglion cell-inner plexiform layer (GCIPL) according to ocular dominance.</p

A 61-year-old female with normal-tension glaucoma.

<p>This patient had a disc hemorrhage (DH) at the 9 o’clock position at the cup base (white arrow). Fluorescein angiography revealed a thrombus, shown as a filling defect inside the vessel, where DH had occurred.</p

Subject Characteristics.

<p>Data are presented as mean ± standard deviation.</p><p>Subject Characteristics.</p

A 54-year-old female with normal-tension glaucoma.

<p>Disc hemorrhage not related to a localized retinal nerve fiber layer defect shows no specific findings suggesting hemodynamic changes, such as vessel filling defect or delayed filling, disc leak, and disc filling defects.</p