The flow chart of the included studies in the meta-analysis.

<p>The flow chart of the included studies in the meta-analysis.</p

False positive reporting probability values for associations between the <i>NQO1</i> 609C>T polymorphism and the risk of GI cancers.

<p>False positive reporting probability values for associations between the <i>NQO1</i> 609C>T polymorphism and the risk of GI cancers.</p

Meta-analysis for the association between the <i>NQO1</i> 609C>T polymorphism and cancer risk.

a<p>Number of studies. The studies investigating multiple types of cancers or multiple ethnicities were separated into groups for the subgroup analysis.</p>b<p>CT/TT <i>vs</i>. CC: Dominant model.</p>c<p>TT <i>vs</i>. CT/CC: Recessive model.</p>d<p>Heterogeneity across studies.</p

Forest plot (Fixed effects model) describing the association of the <i>NQO1</i> 609C>T polymorphism with risk of gastrointestinal (GI) cancers.

<p>The <i>NQO1</i> 609C>T polymorphism was associated with a modestly increased risk of GI cancers in a dominant model (CT/TT <i>vs.</i> CC).</p

Characteristics of studies included in the meta-analysis.

a<p><i>P</i>-value of the chi-square goodness of fit test for Hardy-Weinberg equilibrium (HWE) in controls.</p>b<p>the HWE test can not be conducted because only the total number of genotypes (TT <i>vs</i>. CT/CC) was available, and the HWE test was not mentioned in this study.</p

The genotype frequencies of the <i>NQO1</i> 609C >T polymorphism in controls in different ethnic groups.

a<p>The study by Lafuente et al was excluded when calculating the genotype frequency because the numbers for the CC and TT genotypes were not provided in this study.</p>b<p>Two-side Student's <i>t</i> test within the stratum.</p

Funnel plot analysis to detect publication bias.

<p>Each point represents an individual study for the indicated association.</p

Statistical analysis of matched sequences from the Liaoning cashmere goat.

<p>The clear bars represent the top-hit species distribution of BLAST matches of Liaoning cashmere goat unigenes. Note that nearly 68% of top hits are to <i>Bos </i><i>taurus</i>, whose complete genome has been sequenced. The shaded bars (inset) represented the percent of matched unigenes in the total sequences of the NCBI Nr database of each species in the top 20. Nearly 6% of the <i>Capra </i><i>hircus</i> sequences in NCBI NR database are covered.</p

Length distribution of Liaoning cashmere goat transcriptomic sequences.

<p>(<b>A</b>) total transcriptomic reads, (<b>B</b>) isotigs, (<b>C</b>) singletons.</p

Chromate Interaction with the Chromate Reducing Actinobacterium <i>Intrasporangium chromatireducens</i> Q5-1

<div><p>This study was conducted to determine the microbe-chromate [Cr(VI)] interaction and the effect of quinoid analogue anthraquinone-2-sulfonate (AQS) on aerobic Cr(VI) reduction by <i>Intrasporangium chromatireducens</i> Q5-1. The addition of redox mediator AQS, which might expedite the electron transfer, promoted Cr(VI) bioreduction. Addition of carbon sources, such as maltose, acetate, sucrose and lactose stimulated the AQS-promoted Cr(VI) reduction of strain Q5-1. Induction experiment clarified that the enzyme involves in the Cr(VI) reduction is constitutive. Energy-dispersive spectroscopy (EDS) spectra showed the existence of trace Cr distributed on the cell surface. X-ray photoelectron spectroscopy (XPS) analysis revealed that the Cr(III) complex was bound to the cell surface (0.87%, atomic percent). The spectra shifts detected by Fourier transform infrared (FTIR) spectroscopy indicated that Cr(III) was bound to the carbonyl and amide groups. In addition, Cr(VI) reduction by different cell fractions showed that Cr(VI) reduction was occurred extracellularly rather than intracellularly. The results disclosed that Cr(VI) detoxification of strain Q5-1 was mainly associated with extracellular Cr(VI) reduction process in combination with trace Cr(III) adsorption on the cell surface. A schematic figure depicting the interactions between strain Q5-1 and Cr(VI) was presented. This study enhanced the understanding of the microbe-Cr(VI) interaction mechanism and revealed the AQS-promoted aerobic Cr(VI) reduction of strain Q5-1. Such strain and quinoid analogue-mediated bacterial Cr(VI) reduction may facilitate the bioremediation for Cr(VI)-polluted environment.</p></div