22,257 research outputs found
Merger as Intermittent Accretion
The Self-Similar Secondary Infall Model (SSIM) is modified to simulate a
merger event. The model encompass spherical versions of tidal stripping and
dynamical friction that agrees with the Syer & White merger paradigm's
behaviour. The SSIM shows robustness in absorbing even comparable mass
perturbations and returning to its original state. It suggests the approach to
be invertible and allows to consider accretion as smooth mass inflow merging
and mergers as intermittent mass inflow accretion.Comment: letter accepted by A&A 29/09/08, 4 pages, colour figure
X-ray Constraints on Cluster Magnetic Fields
Lower limits to the intracluster magnetic field are derived from modeling
broad band X-ray spectra of 6 galaxy clusters with radio halos (A401, A754,
A1367, Coma (A1656), A2256, and A2319). The 0.7 - 10 keV spectra for all
clusters were obtained with the Advanced Satellite for Cosmology and
Astrophysics (ASCA) Gas Imaging Spectrometer (GIS). For A401, A754, A1367, and
A1656, data from the HEAO1-A2 High Energy Detector (HED) (2-60 keV) was
included in a joint fit to further constrain the allowed non-thermal component.
Upperlimits to the non-thermal X-ray emission are a factor of approximately 10
improvement over previous values. Lower limits to the average magnetic field
derived from these upperlimits and the radio spectral parameters range from
0.08 - 0.30 microGauss. A seed intracluster field amplified by turbulence from
galaxy motion through the intracluster medium should have an average value in
the range of 0.1-0.2 (Goldman & Rephaeli, 1991; De Young, 1992), thus the lack
of any detections makes it unlikely that field amplification is due to galaxy
wakes. In addition, all of these clusters have an asymmetric surface brightness
distribution while several also have X-ray spectroscopic evidence of a merger.
We suggest that cluster mergers rather than galaxy wakes are the source of the
magnetic field amplification.Comment: 24 pages, 6 figures, to appear in PASJ 5
Isolated and non-isolated dark matter halos and the NFW profile
We compare the conclusions reached using the coarse-graining technique
employed by Henriksen (2004) for a one degree of freedom (per particle)
collisionless system, to those presented in a paper by Binney (2004) based on
an exact one degree of freedom model. We find agreement in detail but in
addition we show that the isolated 1D system is self-similar and therefore
unrelaxed. Fine graining of this system recovers much less prominent wave-like
structure than in a spherically symmetric isotropic 3D system. The rate of
central flattening is also reduced in the 1D system. We take this to be
evidence that relaxation of collisionless systems proceeds ultimately by way of
short wavelength Landau damping. N-body systems, both real and simulated, can
be trapped in an incompletely relaxed state because of a break in the cascade
of energy to small scales. This may be due to the rapid dissipation of the
small scale oscillations in an isolated system, to the existence of conserved
quantities such as angular momentum, or to the failure in simulations to
resolve sub-Jeans length scales. Such a partially relaxed state appears to be
the NFW state, and is to be expected especially in young systems. The NFW core
is shown to be isolated. In non-isolated systems continuing coarse-grained
relaxation should be towards a density core in solid body rotation.Comment: 14 pages, MNRAS, submitted 21 June 200
Estimated central blood volume in cirrhosis
The estimated central blood volume (i.e., blood volume in the heart cavities, lungs and central arterial tree) was determined by multiplying cardiac output by circulatory mean transit time in 19 patients with cirrhosis and compared with sympathetic nervous activity and circulating level of atrial natriuretic factor. Arterial norepinephrine level, an index of overall sympathetic nervous activity (3.08 nmol/L in patients vs. 1.36 nmol/L in controls; p < 0.01) was negatively correlated (r = -0.54, p < 0.01) with estimated central blood volume (mean = 23 ml/kg in patients vs. 27 ml/kg in controls; p < 0.05). Similarly, renal venous norepinephrine level (an index of renal sympathetic tone; 4.26 nmol/L in patients vs. 1.78 nmol/L in controls; p < 0.01) was inversely correlated with estimated central blood volume (r = -0.53, n = 18, p < 0.02). No significant correlation could be established between arterial atrial natriuretic factor level (8.9 pmol/L in patients vs. 9.6 pmol/L in controls; not significant) and estimated central blood volume. Hemodynamic values were subsequently modified with oral propranolol (80 mg). During -adrenergic blockade, the mean estimated central blood volume was not altered significantly, except in six patients who exhibited decreases in mean arterial blood pressure (85 to 69 mm Hg; n = 6) and decreases in mean estimated central blood volume (23.2 to 20.6 ml/kg; n = 6, p < 0.05). Slight increases were observed in mean right atrial pressure (2.2 to 3.7 mm Hg; n = 14, p < 0.05); this change was positively correlated with the change in estimated central blood volume (r = 0.44, n = 14, p = 0.06). In conclusion, reduced estimated central blood volume probably unloads volume receptors and baroreceptors, thus provoking enhanced overall and renal sympathetic nervous activity and thereby contributing to increased water and salt retention in cirrhosis. During -adrenergic blockade estimated central blood volume changes correlated with alterations in preload and afterload. These findings indicate that central circulatory and arterial underfilling is a key element of the hemodynamic derangement observed in cirrhosis
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