Pion condensation in a dense neutrino gas

We argue that using an equilibrated gas of neutrinos it is possible to probe the phase diagram of QCD for finite isospin and small baryon chemical potentials. We discuss this region of the phase diagram in detail and demonstrate that for large enough neutrino densities a Bose-Einstein condensate of positively charged pions arises. Moreover, we show that for nonzero neutrino density the degeneracy in the lifetimes and masses of the charged pions is lifted.Comment: 10 pages, 7 figures. Modifications to Section II, IIIc, and I

Quark and pion condensation in a chromomagnetic background field

The general features of quark and pion condensation in dense quark matter with flavor asymmetry have been considered at finite temperature in the presence of a chromomagnetic background field modelling the gluon condensate. In particular, pion condensation in the case of a constant abelian chromomagnetic field and zero temperature has been studied both analytically and numerically. Under the influence of the chromomagnetic background field the effective potential of the system is found to have a global minimum for a finite pion condensate even for small values of the effective quark coupling constant. In the strong field limit, an effective dimensional reduction has been found to take place.Comment: 17 pages, 6 figure

Cloning and characterization of MN1, a gene from chromosome 22q11, which is disrupted by a balanced translocation in a meningioma

We have isolated a gene, called MN1, which resides on chromosome 22 and which was found to be disrupted by a balanced translocation (4;22) in meningioma 32. The MN1 gene spans about 70 kb and consists of at least two large exons of approximately 4.7 kb and 2.8 kb. The MN1 cDNA codes for a protein of 1319 amino acids when the first methionine in the open reading frame is used. The MN1 cDNA contains two CAG repeats, one of which codes for a string of 28 glutamines. The t(4;22) disrupts the 5'-exon within the open reading frame, In meningioma 32 no expression of the MN1 mRNA is observed. These results suggest that inactivation of the MN1 gene in this tumour may contribute to its pathogenesis.</p

Cloning and characterization of MN1, a gene from chromosome 22q11, which is disrupted by a balanced translocation in a meningioma

We have isolated a gene, called MN1, which resides on chromosome 22 and which was found to be disrupted by a balanced translocation (4;22) in meningioma 32. The MN1 gene spans about 70 kb and consists of at least two large exons of approximately 4.7 kb and 2.8 kb. The MN1 cDNA codes for a protein of 1319 amino acids when the first methionine in the open reading frame is used. The MN1 cDNA contains two CAG repeats, one of which codes for a string of 28 glutamines. The t(4;22) disrupts the 5'-exon within the open reading frame, In meningioma 32 no expression of the MN1 mRNA is observed. These results suggest that inactivation of the MN1 gene in this tumour may contribute to its pathogenesis.</p