Child support enforcement for teenage fathers: Problems and prospects

The NLSY data indicate that about 7.3 percent of teenage males become fathers and that very few of these fathers live with their children. Father absence and the concurrent increase in female-headed households are closely associated with the impoverishment of children. Most absent teen fathers never come into contact with the child support enforcement program, and the extent to which they financially support their children informally is not well understood. While the income of absent teen fathers is low in the teen years, it increases over time, as does the potential for collecting child support. Nevertheless, men who were absent teen fathers earn less in early adulthood than men who deferred parenting until age twenty or later and teen fathers who lived with their children. Early establishment of paternity and greater standardization in the treatment of adolescent fathers by the child support enforcement program are recommended. Further, the substantial and persistent income deficit experienced by adolescent fathers who live apart from their children raises an interesting dilemma. While children may benefit financially and psychosocially from living with two parents, the lower income of men who were absent teenage fathers may make them poor marital prospects. This raises doubts about the recent recommendations of some scholars that we should bring back the shotgun wedding.

The role of mucosal immunity in the pathogenesis of necrotizing enterocolitis

Necrotizing enterocolitis (NEC) is the most devastating gastrointestinal disease of prematurity. Although the precise cause is not well understood, the main risk factors thought to contribute to NEC include prematurity, formula feeding, and bacterial colonization. Recent evidence suggests that NEC develops as a consequence of intestinal hyper-responsiveness to microbial ligands upon bacterial colonization in the preterm infant, initiating a cascade of aberrant signaling events, and a robust pro-inflammatory mucosal immune response. We now have a greater understanding of important mechanisms of disease pathogenesis, such as the role of cytokines, immunoglobulins, and immune cells in NEC. In this review, we will provide an overview of the mucosal immunity of the intestine and the relationship between components of the mucosal immune system involved in the pathogenesis of NEC, while highlighting recent advances in the field that have promise as potential therapeutic targets. First, we will describe the cellular components of the intestinal epithelium and mucosal immune system and their relationship to NEC. We will then discuss the relationship between the gut microbiota and cell signaling that underpins disease pathogenesis. We will conclude our discussion by highlighting notable therapeutic advancements in NEC that target the intestinal mucosal immunity

Sensitivity of a climatologically-driven sea ice model to the ocean heat flux

Ocean heat flux sensitivity was studied on a numerical model of sea ice covering the Weddell Sea region of the southern ocean. The model is driven by mean monthly climatological atmospheric variables. For each model run, the ocean heat flux is uniform in both space and time. Ocean heat fluxes below 20 W m to the minus 2 power do not provide sufficient energy to allow the ice to melt to its summertime thicknesses and concentrations by the end of the 14 month simulation, whereas ocean heat fluxes of 30 W m to the minus 2 power and above result in too much ice melt, producing the almost total disappearance of ice in the Weddell Sea by the end of the 14 months. These results are dependent on the atmospheric forcing fields