Vortex-line liquid phases: Longitudinal superconductivity in the lattice London model

We study the vortex-line lattice and liquid phases of a clean type-II superconductor by means of Monte Carlo simulations of the lattice London model. Motivated by a recent controversy regarding the presence, within this model, of a vortex-liquid regime with longitudinal superconducting coherence over long length scales, we directly compare two different ways to calculate the longitudinal coherence. For an isotropic superconductor, we interpret our results in terms of a temperature regime within the liquid phase in which longitudinal superconducting coherence extends over length scales larger than the system thickness studied. We note that this regime disappears in the moderately anisotropic case due to a proliferation, close to the flux-line lattice melting temperature, of vortex loops between the layers.Comment: 8 pages, Revtex, with eps figures. To appear in Phys. Rev.

Far infrared reflectance of a mixed phase Y-Ba-Cu-O superconductor

The response of polycrystalline Tl–Ba–Ca–Cu–O superconducting thin films on short laser pulses has been investigated for radiation between 10 mgrm and 500 mgrm wavelength. Fast signals with time constants less than 1 ns were observed for wavelengths longer than about 100 mgrm whereas for shorter wavelengths only a bolometric signal could be detected

Immune Boosting Explains Regime-Shifts in Prevaccine-Era Pertussis Dynamics

Understanding the biological mechanisms underlying episodic outbreaks of infectious diseases is one of mathematical epidemiology’s major goals. Historic records are an invaluable source of information in this enterprise. Pertussis (whooping cough) is a re-emerging infection whose intermittent bouts of large multiannual epidemics interspersed between periods of smaller-amplitude cycles remain an enigma. It has been suggested that recent increases in pertussis incidence and shifts in the age-distribution of cases may be due to diminished natural immune boosting. Here we show that a model that incorporates this mechanism can account for a unique set of pre-vaccine-era data from Copenhagen. Under this model, immune boosting induces transient bursts of large amplitude outbreaks. In the face of mass vaccination, the boosting model predicts larger and more frequent outbreaks than do models with permanent or passively-waning immunity. Our results emphasize the importance of understanding the mechanisms responsible for maintaining immune memory fo