1,240 research outputs found
Promoting Metacognition and Motivation of Exceptional Children
Metacognition fosters independent learning by providing personal insight into one's own thinking. Such awareness can lead to flexible and confident problem solving as well as feelings of self-efficacy and pride. This is especially important for students who encounter difficulty in school because they do not understand how to appraise and manage their own resources for learning. Too often, students develop debilitating expectations and behavior that undermine learning in school and inhibit transfer of effective learning strategies. We describe four general kinds of instruction that help students learn to think: metacognitive explanation, scaffolded instruction, cognitive coaching, and cooperative learning. Teachers can adapt and combine these methods to teach students how to think as they read, write, and compute in classrooms.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/69089/2/10.1177_074193259001100604.pd
Properties of continuous Fourier extension of the discrete cosine transform and its multidimensional generalization
A versatile method is described for the practical computation of the discrete
Fourier transforms (DFT) of a continuous function given by its values
at the points of a uniform grid generated by conjugacy classes
of elements of finite adjoint order in the fundamental region of
compact semisimple Lie groups. The present implementation of the method is for
the groups SU(2), when is reduced to a one-dimensional segment, and for
in multidimensional cases. This simplest case
turns out to result in a transform known as discrete cosine transform (DCT),
which is often considered to be simply a specific type of the standard DFT.
Here we show that the DCT is very different from the standard DFT when the
properties of the continuous extensions of these two discrete transforms from
the discrete grid points to all points are
considered. (A) Unlike the continuous extension of the DFT, the continuous
extension of (the inverse) DCT, called CEDCT, closely approximates
between the grid points . (B) For increasing , the derivative of CEDCT
converges to the derivative of . And (C), for CEDCT the principle of
locality is valid. Finally, we use the continuous extension of 2-dimensional
DCT to illustrate its potential for interpolation, as well as for the data
compression of 2D images.Comment: submitted to JMP on April 3, 2003; still waiting for the referee's
Repor
An adaptive prefix-assignment technique for symmetry reduction
This paper presents a technique for symmetry reduction that adaptively
assigns a prefix of variables in a system of constraints so that the generated
prefix-assignments are pairwise nonisomorphic under the action of the symmetry
group of the system. The technique is based on McKay's canonical extension
framework [J.~Algorithms 26 (1998), no.~2, 306--324]. Among key features of the
technique are (i) adaptability---the prefix sequence can be user-prescribed and
truncated for compatibility with the group of symmetries; (ii)
parallelizability---prefix-assignments can be processed in parallel
independently of each other; (iii) versatility---the method is applicable
whenever the group of symmetries can be concisely represented as the
automorphism group of a vertex-colored graph; and (iv) implementability---the
method can be implemented relying on a canonical labeling map for
vertex-colored graphs as the only nontrivial subroutine. To demonstrate the
practical applicability of our technique, we have prepared an experimental
open-source implementation of the technique and carry out a set of experiments
that demonstrate ability to reduce symmetry on hard instances. Furthermore, we
demonstrate that the implementation effectively parallelizes to compute
clusters with multiple nodes via a message-passing interface.Comment: Updated manuscript submitted for revie
Ergodic properties of a generic non-integrable quantum many-body system in thermodynamic limit
We study a generic but simple non-integrable quantum {\em many-body} system
of {\em locally} interacting particles, namely a kicked model of spinless
fermions on 1-dim lattice (equivalent to a kicked Heisenberg XX-Z chain of 1/2
spins). Statistical properties of dynamics (quantum ergodicity and quantum
mixing) and the nature of quantum transport in {\em thermodynamic limit} are
considered as the kick parameters (which control the degree of
non-integrability) are varied. We find and demonstrate {\em ballistic}
transport and non-ergodic, non-mixing dynamics (implying infinite conductivity
at all temperatures) in the {\em integrable} regime of zero or very small kick
parameters, and more generally and important, also in {\em non-integrable}
regime of {\em intermediate} values of kicked parameters, whereas only for
sufficiently large kick parameters we recover quantum ergodicity and mixing
implying normal (diffusive) transport. We propose an order parameter (charge
stiffness ) which controls the phase transition from non-mixing/non-ergodic
dynamics (ordered phase, ) to mixing/ergodic dynamics (disordered phase,
D=0) in the thermodynamic limit. Furthermore, we find {\em exponential decay of
time-correlation function} in the regime of mixing dynamics.
The results are obtained consistently within three different numerical and
analytical approaches: (i) time evolution of a finite system and direct
computation of time correlation functions, (ii) full diagonalization of finite
systems and statistical analysis of stationary data, and (iii) algebraic
construction of quantum invariants of motion of an infinite system, in
particular the time averaged observables.Comment: 18 pages in REVTeX with 14 eps figures included, Submitted to
Physical Review
Π£ΠΏΡΠ°Π²Π»Π΅Π½ΠΈΠ΅ ΠΎΠΏΠ΅ΡΠ°ΡΠΈΡΠΌΠΈ Ρ ΠΏΠ»Π°ΡΡΠΈΠΊΠΎΠ²ΡΠΌΠΈ ΠΊΠ°ΡΡΠ°ΠΌΠΈ Π² ΠΊΠΎΠΌΠΌΠ΅ΡΡΠ΅ΡΠΊΠΎΠΌ Π±Π°Π½ΠΊΠ΅
Π Π°Π±ΠΎΡΠ° ΠΏΠΎΡΠ²ΡΡΠ΅Π½Π° ΡΠΏΡΠ°Π²Π»Π΅Π½ΠΈΡ ΠΎΠΏΠ΅ΡΠ°ΡΠΈΡΠΌΠΈ Ρ Π±Π°Π½ΠΊΠΎΠ²ΡΠΊΠΈΠΌΠΈ ΠΊΠ°ΡΡΠ°ΠΌΠΈ Π² ΠΊΠΎΠΌΠΌΠ΅ΡΡΠ΅ΡΠΊΠΎΠΌ Π±Π°Π½ΠΊΠ΅. Π Π°ΡΡΠΌΠ°ΡΡΠΈΠ²Π°ΡΡΡΡ Π²ΠΈΠ΄Ρ Π±Π°Π½ΠΊΠΎΠ²ΡΠΊΠΈΡ
ΠΊΠ°ΡΡ, ΠΊΠ»Π°ΡΡΠΈΡΠΈΠΊΠ°ΡΠΈΡ ΠΎΠΏΠ΅ΡΠ°ΡΠΈΠΉ Ρ Π½ΠΈΠΌΠΈ ΠΈ ΠΈΠ·ΡΡΠ΅Π½ΠΈΠ΅ ΡΠΎΠ²ΡΠ΅ΠΌΠ΅Π½Π½ΡΡ
ΡΠ΅Π½Π΄Π΅Π½ΡΠΈΠΉ ΡΠ°Π·Π²ΠΈΡΠΈΡ ΠΏΠ»Π°ΡΡΠΈΠΊΠΎΠ²ΡΡ
ΠΊΠ°ΡΡ Π² Π ΠΎΡΡΠΈΠΉΡΠΊΠΎΠΉ Π€Π΅Π΄Π΅ΡΠ°ΡΠΈΠΈ. Π ΠΊΠΎΠ½ΡΠ΅ ΡΠ°Π±ΠΎΡΡ Π΄Π°ΡΡΡΡ ΡΠ΅ΠΊΠΎΠΌΠ΅Π½Π΄Π°ΡΠΈΠΈ ΠΏΠΎ ΡΠ°Π·Π²ΠΈΡΠΈΡ ΠΎΠΏΠ΅ΡΠ°ΡΠΈΠΉ Ρ ΠΏΠ»Π°ΡΡΠΈΠΊΠΎΠ²ΡΠΌΠΈ ΠΊΠ°ΡΡΠ°ΠΌΠΈ Π² ΠΊΠΎΠΌΠΌΠ΅ΡΡΠ΅ΡΠΊΠΎΠΌ Π±Π°Π½ΠΊΠ΅.The work is dedicated to the management of card transactions in the commercial bank. Consider the types of bank cards , the classification of transactions with them and study modern trends in the development of plastic cards in the Russian Federation . At the end of the paper provides recommendations on the development of operations with plastic cards in a commercial bank
Inhibition of HMG CoA reductase reveals an unexpected role for cholesterol during PGC migration in the mouse
<p>Abstract</p> <p>Background</p> <p>Primordial germ cells (PGCs) are the embryonic precursors of the sperm and eggs. Environmental or genetic defects that alter PGC development can impair fertility or cause formation of germ cell tumors.</p> <p>Results</p> <p>We demonstrate a novel role for cholesterol during germ cell migration in mice. Cholesterol was measured in living tissue dissected from mouse embryos and was found to accumulate within the developing gonads as germ cells migrate to colonize these structures. Cholesterol synthesis was blocked in culture by inhibiting the activity of HMG CoA reductase (HMGCR) resulting in germ cell survival and migration defects. These defects were rescued by co-addition of isoprenoids and cholesterol, but neither compound alone was sufficient. In contrast, loss of the last or penultimate enzyme in cholesterol biosynthesis did not alter PGC numbers or position in vivo. However embryos that lack these enzymes do not exhibit cholesterol defects at the stage at which PGCs are migrating. This demonstrates that during gestation, the cholesterol required for PGC migration can be supplied maternally.</p> <p>Conclusion</p> <p>In the mouse, cholesterol is required for PGC survival and motility. It may act cell-autonomously by regulating clustering of growth factor receptors within PGCs or non cell-autonomously by controlling release of growth factors required for PGC guidance and survival.</p
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