709 research outputs found
Fibronectin is required to prevent thyroid cell apoptosis through an integrin-mediated adhesion mechanism.
Prenyltransferase inhibitors induce apoptosis in proliferating thyroid cells through a p53-independent CrmA-sensitive, and caspase-3-like protease-dependent mechanism.
Retinoic acid induces intercellular adhesion molecule-1 hyperexpression in human thyroid carcinoma cell lines.
Thyrotropin and growth promoting immunoglobulin (TGI) of FRTL-5 cells have no growth stimulating activity on human thyroid epithelial cell cultures
Serum withdrawal-induced apoptosis in thyroid cells is caused by loss of fibronectin-integrin interaction.
Calcium/calmodulin-dependent protein kinase II binds to Raf-1 and modulates integrin-stimulated ERK activation.
Integrin-dependent cell growth and survival are mediated by different signals in thyroid cells.
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