HOW TO SELL FRESH PRODUCE TO SUPERMARKET CHAINS

Marketing,

DRAWING THE PROFILE OF EFFICIENT FOOD INDUSTRIES-VERTICAL INTEGRATION, ECONOMIES OF SCALE, AND LOCATION ADVANTAGES IN THE DISTRIBUTION OF PRODUCTS: A CASE STUDY FROM THE GREEK FOOD INDUSTRY

A stochastic frontier production function that incorporates a model for technical inefficiency effects is used to investigate the industrial production of Greek food industries. Panel data comes from 29 Greek firms in 1988 through 1992. Parameters considered in the model for inefficiency effects include the degree of vertical integration, capital intensity, location, and time. A translog stochastic frontier function is estimated simultaneously with those variables in the model for inefficiency effects. The results indicate that technical efficiency among the firms ranges from 42 percent to 99 percent. More efficient firms are those with a higher degree of vertical integration that are located in rural areas and have sufficient investment in human capital to exploit the economies of scale obtained through investment in fixed capital. Most firms improve their performance over time, reducing the efficiency gap.Agribusiness,

Report of the Committee on Continuing Education, April 7, 1971

https://egrove.olemiss.edu/aicpa_assoc/2015/thumbnail.jp

Academic preparation for professional accounting careers

https://egrove.olemiss.edu/aicpa_comm/1072/thumbnail.jp

Report of the Committee on Education and Experience Requirements for CPAs

https://egrove.olemiss.edu/aicpa_comm/1084/thumbnail.jp

Fine map of the Gct1 spontaneous ovarian granulosa cell tumor locus

The spontaneous development of juvenile-onset, ovarian granulosa cell (GC) tumors in the SWR/Bm (SWR) inbred mouse strain is a model for juvenile-type GC tumors that appear in infants and young girls. GC tumor susceptibility is supported by multiple Granulosa cell tumor (Gct) loci, but the Gct1 locus on Chr 4 derived from SWR strain background is fundamental for GC tumor development and uniquely responsive to the androgenic precursor dehydroepiandrosterone (DHEA). To resolve the location of Gct1 independently from other susceptibility loci, Gct1 was isolated in a congenic strain that replaces the distal segment of Chr 4 in SWR mice with a 47 × 10(6)-bp genomic segment from the Castaneus/Ei (CAST) strain. SWR females homozygous for the CAST donor segment were confirmed to be resistant to DHEA- and testosterone-induced GC tumorigenesis, indicating successful exchange of CAST alleles (Gct1(CA)) for SWR alleles (Gct1(SW)) at this tumor susceptibility locus. A series of nested, overlapping, congenic sublines was created to fine-map Gct1 based on GC tumor susceptibility under the influence of pubertal DHEA treatment. Twelve informative lines have resolved the Gct1 locus to a 1.31 × 10(6)-bp interval on mouse Chr 4, a region orthologous to human Chr 1p36.22. ELECTRONIC SUPPLEMENTARY MATERIAL: The online version of this article (doi:10.1007/s00335-012-9439-6) contains supplementary material, which is available to authorized users