No experiments: federal privatisation politics in West Germany 1949-1989

Privatisation has been a key policy in the late 20th century in many countries. In West Germany, the federal government sold most of its corporate industrial shareholdings to private investors between 1949 and 1989. Unlike many other countries, West Germany did not nationalise entire industries after the Second World War. Instead, the portfolio of public enterprises and participations was mainly an inheritance from the Third Reich. The aim of the thesis is to explore the causes of privatisation and the driving and delaying forces in the privatisation process between 1949 and 1989 based on qualitative historical documents. After the sale of participations stemming from the war economy in the early 1950s, the conservative federal government of CDU and CSU and later the conservative-liberal government of CDU, CSU and FDP under the Federal Chancellors Konrad Adenauer (CDU) and Ludwig Erhard (CDU) pursued a larger scale privatisation programme by issuing people's shares between 1959 and 1965. The programme featured social elements and aimed at the property formation of employees and a wide dispersion of shares in the society. In the 1970s, public enterprises expanded under a social-liberal government of SPD and FDP, until a conservative-liberal government of CDU, CSU and FDP under Federal Chancellor Kohl (CDU) sold most of the remaining federal participations in industrial enterprises between 1984 and 1989. The total volume of privatisation as measured by revenues remained modest compared to other West European countries and strong political resistance within the government parties CDU and CSU manifested in the process. Findings indicate a high continuity of thought and policy patterns from the 1950s until the end of the 1980s while the main reasons for privatisation shifted slightly. In the 1950s and 1960s, privatisation was primarily motivated by fiscal reasons - access to equity capital proved to be limited for the growing federal enterprises. Privatisation in the 1980s was caused by re-interpretations of the economic situation due to globally changing conditions and increased international competition. Hence, it can be interpreted as a lagged response to market crisis in the 1970s. Ideological shifts of paradigm did not drive privatisation. Rather, advocates of ordoliberalism focused on other economic reforms in the 1950s and liberal ideas in the 1980s co-developed with privatisation politics. For many decades, public enterprises were not viewed as ineffcient per se as long as they were operating in competitive markets. This perception only began to change slowly in the 1980s

Immunolocalisation of P2Y receptors in the rat eye

Nucleotides present an important role in ocular physiology which has been demonstrated by recent works that indicate their involvement in many ocular processes. P2Y are important among P2 receptors since they can control tear production, corneal wound healing, aqueous humour dynamics and retinal physiology. Commercial antibodies have allowed us to investigate the distribution of P2Y receptors in the cornea, anterior and posterior chamber of the eye and retina. The P2Y1 receptor was present mainly in cornea, ciliary processes, and trabecular meshwork. The P2Y2 receptors were present in cornea, ciliary processes and retinal pigmented epithelium. P2Y4 was present in cornea, ciliary processes, photoreceptors, outer plexiform layer and ganglion cell layer. The P2Y6 presented almost an identical distribution as the P2Y4 receptor. The P2Y11 was also detectable in the retinal pigmented epithelium. The detailed distribution of the receptors clearly supports the recent findings indicating the relevant role of nucleotides in the ocular function

Mechanisms Involved in Nicotinic Acetylcholine Receptor-Induced Neurotransmitter Release from Sympathetic Nerve Terminals in the Mouse Vas Deferens

Prejunctional nicotinic acetylcholine receptors (nAChRs) amplify postganglionic sympathetic neurotransmission, and there are indications that intraterminal Ca2+ stores might be involved. However, the mechanisms by which nAChR activation stimulates neurotransmitter release at such junctions is unknown. Rapid local delivery (picospritzing) of the nAChR agonist epibatidine was combined with intracellular sharp microelectrode recording to monitor spontaneous and field-stimulation-evoked neurotransmitter release from sympathetic nerve terminals in the mouse isolated vas deferens. Locally applied epibatidine (1 µM) produced ‘epibatidine-induced depolarisations’ (EIDs) that were similar in shape to spontaneous excitatory junction potentials (SEJPs) and were abolished by nonselective nAChR antagonists and the purinergic desensitizing agonist α,β-methylene ATP. The amplitude distribution of EIDs was only slightly shifted towards lower amplitudes by the selective α7 nAChR antagonists α-bungarotoxin and methyllcaconitine, the voltage-gated Na+ channel blocker tetrodotoxin or by blocking voltage-gated Ca2+ channels with Cd2+. Lowering the extracellular Ca2+ concentration reduced the frequency of EIDs by 69%, but more surprisingly, the Ca2+-induced Ca2+ release blocker ryanodine greatly decreased the amplitude (by 41%) and the frequency of EIDs by 36%. Ryanodine had no effect on electrically-evoked neurotransmitter release, paired-pulse facilitation, SEJP frequency, SEJP amplitude or SEJP amplitude distribution. These results show that activation of non-α7 nAChRs on sympathetic postganglionic nerve terminals induces high-amplitude junctional potentials that are argued to represent multipacketed neurotransmitter release synchronized by intraterminal Ca2+-induced Ca2+ release, triggered by Ca2+ influx directly through the nAChR. This nAChR-induced neurotransmitter release can be targeted pharmacologically without affecting spontaneous or electrically-evoked neurotransmitter release

P2 receptor-mediated modulation of neurotransmitter release—an update

Presynaptic nerve terminals are equipped with a number of presynaptic auto- and heteroreceptors, including ionotropic P2X and metabotropic P2Y receptors. P2 receptors serve as modulation sites of transmitter release by ATP and other nucleotides released by neuronal activity and pathological signals. A wide variety of P2X and P2Y receptors expressed at pre- and postsynaptic sites as well as in glial cells are involved directly or indirectly in the modulation of neurotransmitter release. Nucleotides are released from synaptic and nonsynaptic sites throughout the nervous system and might reach concentrations high enough to activate these receptors. By providing a fine-tuning mechanism these receptors also offer attractive sites for pharmacotherapy in nervous system diseases. Here we review the rapidly emerging data on the modulation of transmitter release by facilitatory and inhibitory P2 receptors and the receptor subtypes involved in these interactions

Die Beschaffenheitsvereinbarung im System des Kaufgewährleistungsrechts

Durch eine Beschaffenheitsvereinbarung legen die Parteien die Soll-Beschaffenheit des Kaufobjekts rechtsgeschäftlich fest. Weicht die Ist-Beschaffenheit der Sache von dieser Vereinbarung negativ ab, begründet dies einen Sachmangel, der dem Käufer gewährleistungsrechtliche Ansprüche gegen den Verkäufer eröffnet. Die Beschaffenheitsvereinbarung nimmt eine bedeutende Rolle im kaufrechtlichen Sachmangeltatbestand ein und steht regelmäßig im Fokus der höchstrichterlichen Rechtsprechung des BGH. Zugleich bewirkte die Umsetzung der Warenkaufrichtlinie eine Reihe struktureller Änderungen des kaufrechtlichen Sachmangeltatbestandes. Die Arbeit unterzieht die Rechtsprechung des BGH zur Beschaffenheitsvereinbarung einer kritischen Würdigung und beleuchtet zugleich die Einflüsse der Warenkaufrichtlinie auf die Beschaffenheitsvereinbarung. Ihr Ziel ist es, die Beschaffenheitsvereinbarung in ein kohärentes Konzept einzufügen, das Einzelfragen einer sachgerechten Lösung zuführt.»The Quality Agreement in the System of Purchase Warranty Law«: The agreement on quality under the law on the sale of goods occupies a central position within the factual elements of a material defect under the law on the sale of goods. It is regularly the focus of case law by the highest courts and has been influenced by the implementation of the Sale of Goods Directive. The present work acknowledges these influences in detail and integrates the agreement on quality under the law of sale into a coherent concept that provides an appropriate solution to individual questions