Bis(μ-4-hydroxy­benzoato-κ2 O:O′)bis­[triaqua­bis(4-hydroxy­benzoato)-κO;κ2 O,O′-terbium(III)] deca­hydrate

The title dinuclear compound, [Tb2(C7H5O3)6(H2O)6]·10H2O, lies on a center of inversion and the two TbIII atoms are bridged by two 4-hydroxy­benzoate anions; each metal atom is further coordinated by one monodentate anion and chelated by the third anion. The eight-coordinate geometry approximates a square anti­prism. Hydrogen bonds of the O—H⋯O type connect the uncoordinated water mol­ecules to the dinuclear species, forming a three-dimensional network

Penta­aqua­bis[4-(2-hydroxy­benzyl­idene­amino)benzene­sulfonato]lead(II)

In the structure of the title compound, [Pb(C13H10NO4S)2(H2O)5], two S—O bonds and one C—N bond have lengths of 1.421 (9), 1.425 (8) and 1.268 (11) Å, respectively, which suggests they are double bonds. Mol­ecules form a two-dimensional layered structure via O—H⋯O and O—H⋯N inter­actions. The Pb atom adopts distorted cubo-octahedral coordination

Hexaaqua­nickel(II) bis­[4-(2-hydroxy­benzyl­ideneamino)benzene­sulfonate]

In the title compound, [Ni(H2O)6](C(C13H10NO4S)2, the nickel(II) atom, lying on a center of symmetry, is six-coordinated by six aqua O-atom donors. The dihedral angle between the two benzene rings is 33.1 (3)°. The crystal structure is stabilized by aqua–anion O—H⋯O hydrogen bonds. Intra­molecular O—H⋯N and C—H⋯O hydrogen-bonding inter­actions occur in the anion

2,2′-(1,1′-Azinodiethyl­idyne)diphenol

In the title mol­ecule, C16H16N2O2, the C—N bond lengths are 1.295 (5) and 1.300 (5) Å, which suggests that they are double bonds. The structure is stabilized by intra­molecular O—H⋯N and C—H⋯N, and inter­molecular C—H⋯O hydrogen-bond inter­actions

Hexaaqua­cadmium(II) bis­[4-(2-hydroxy­benzyl­ideneamino)benzene­sulfonate] dihydrate

In the title compound, [Cd(H2O)6](C13H10NO4S)2·2H2O, the Cd atom (site symmetry ) adopts a regular octa­hedral coordination and the anion is stabilized by an intra­molecular O—H⋯N hydrogen bond. O—H⋯O hydrogen bonds involving the coordinated and uncoordinated water mol­ecules lead to a three-dimensional network

N′-[1-(2-Hydroxy­phen­yl)ethyl­idene]benzene­sulfonohydrazide

In the title compound, C14H14N2O3S, the conformation is stabilized by an intra­moleclar O—H⋯N hydrogen bond and the dihedral angle between the aromatic ring planes is 79.55 (18)°. In the crystal structure, inter­molecular N—H⋯O hydrogen bonds lead to [100] chains of mol­ecules

Circular Polarization in two Active Repeating Fast Radio Bursts

Fast radio bursts (FRBs) are bright millisecond radio bursts at cosmological distances. Only a small fraction of FRBs apparently repeat. Polarization, a fundamental property of electromagnetic signals, often carries critical information about the radiation processes, the environment, and the intervening medium of FRBs. Here we report circular polarization detections of two active repeating FRBs, namely FRBs 20121102A and 20190520B, with the Five-hundred-meter Aperture Spherical radio Telescope. We detect circular polarization in both active repeating FRBs, which increases the number of repeating FRBs with circular polarization to three. In one of the bursts of FRB 20121102A, we detect 64% degree of circular polarization. The observed circular polarization is unlikely induced by multipath propagation. Our observations favor circular polarization induced by Faraday conversion or radiation mechanism intrinsic to the FRB source. The conditions to generate circular polarization have to be rare in either case.Comment: 9pages, 3 figures, and 1 table, published in Science Bulleti

S-allylcysteine Improves Blood Flow Recovery and Prevents Ischemic Injury by Augmenting Neovasculogenesis.

Studies suggest that a low level of circulating human endothelial progenitor cells (EPCs) is a risk factor for ischemic injury and coronary artery disease (CAD). Consumption of S-allylcysteine (SAC) is known to prevent CAD. However, the protective effects of SAC on the ischemic injury are not yet clear. In this study, we examined whether SAC could improve blood flow recovery in ischemic tissues through EPC-mediated neovasculogenesis. The results demonstrate that SAC significantly enhances the neovasculogenesis of EPCs in vitro. The molecular mechanisms for SAC enhancement of neovasculogenesis include the activation of Akt/endothelial nitric oxide synthase signaling cascades. SAC increased the expression of c-kit, β-catenin, cyclin D1, and Cyclin-dependent kinase 4 (CDK4) proteins in EPCs. Daily intake of SAC at dosages of 0.2 and 2 mg/kg body weight significantly enhanced c-kit protein levels in vivo. We conclude that dietary consumption of SAC improves blood flow recovery and prevents ischemic injury by inducing neovasculogenesis in experimental models