LFE as a development tool for next generation earthquake professionals

In January 2017 the Earthquake Engineering Research Institute in partnership with the National Research Center for Integrated Disaster Risk Management (CIGIDEN) led a five-day travel study program in Chile in which students and young professionals engaged in learning from earthquakes activities. The 16 participants attended lectures and field trips and completed two resilience projects to contribute to the body of knowledge about recovery since the 2010 Maule earthquake while also becoming familiar with reconnaissance tools and techniques. The program was created to provide learning-from-earthquakes opportunities for younger members outside the limited postevent reconnaissance teams; and to engage younger members in EERI activities and train them for future reconnaissance, which might include long-term resilience and recovery components. The success of the program can be attributed to the strong partnership with CIGIDEN, experienced mentors who accompanied the group, senior academics and practitioners who lectured and led tours, as well as a strong interdisciplinary team of participants who worked extremely hard interviewing locals and compiling the data for their resilience project

Precision surveying using very long baseline interferometry

Radio interferometry measurements were used to measure the vector baselines between large microwave radio antennas. A 1.24 km baseline in Massachusetts between the 36 meter Haystack Observatory antenna and the 18 meter Westford antenna of Lincoln Laboratory was measured with 5 mm repeatability in 12 separate experiments. Preliminary results from measurements of the 3,928 km baseline between the Haystack antenna and the 40 meter antenna at the Owens Valley Radio Observatory in California are presented

Models of chronic obstructive pulmonary disease

Chronic obstructive pulmonary disease (COPD) is a major global health problem and is predicted to become the third most common cause of death by 2020. Apart from the important preventive steps of smoking cessation, there are no other specific treatments for COPD that are as effective in reversing the condition, and therefore there is a need to understand the pathophysiological mechanisms that could lead to new therapeutic strategies. The development of experimental models will help to dissect these mechanisms at the cellular and molecular level. COPD is a disease characterized by progressive airflow obstruction of the peripheral airways, associated with lung inflammation, emphysema and mucus hypersecretion. Different approaches to mimic COPD have been developed but are limited in comparison to models of allergic asthma. COPD models usually do not mimic the major features of human COPD and are commonly based on the induction of COPD-like lesions in the lungs and airways using noxious inhalants such as tobacco smoke, nitrogen dioxide, or sulfur dioxide. Depending on the duration and intensity of exposure, these noxious stimuli induce signs of chronic inflammation and airway remodelling. Emphysema can be achieved by combining such exposure with instillation of tissue-degrading enzymes. Other approaches are based on genetically-targeted mice which develop COPD-like lesions with emphysema, and such mice provide deep insights into pathophysiological mechanisms. Future approaches should aim to mimic irreversible airflow obstruction, associated with cough and sputum production, with the possibility of inducing exacerbations