30 research outputs found

    Distribution of CI and RI values across all characters.

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    <p>Frequencies are calculated from the number of characters informative for parsimony.</p

    Majority-rule consensus tree of 271,160 trees of 835 steps (CI 0.6 and RI 0.7) showing relationships of musical patrimonies from Gabon.

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    <p>(Blue: patrilineal clade; Pink: matrilineal clade; Brown: geographic clades; Green: ethnonymic clades). Bremer indices are indicated on each node. Bremer index of 0 means that the node collapses in a strict consensus tree.</p

    Description of the taxons.

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    <p>In column 2 is a code for the map <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0151570#pone.0151570.g001" target="_blank">Fig 1</a>; column 3 name of the ethnic group and its localisation; column 3: province; column 4: side of the Ogooue river.</p

    Distribution of RI values across character types.

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    <p>Frequencies are calculated from the number of characters informative for parsimony of the corresponding category.</p

    Geographical distribution of musical patrimonies with descent rule system identification of the ethnic group.

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    <p>Blue corresponding to patrilineal groups, pink to matrilineal groups and green to ambivalent groups). Codes (e.g. M9) refer to a patrimony described in <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0151570#pone.0151570.t001" target="_blank">Table 1</a>. Map created by Sylvie Le Bomin from fieldwork data. (Source adapted from Institut national de cartographie du Gabon).</p

    Distribution of CI values across character categories.

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    <p>Frequencies are calculated from the number of characters informative for parsimony of the corresponding category.</p

    Comparison of nasal bone shape in B6, 66H and in Chr1, Chr13, and Chr18 congenic mice, by LDA based on 15 principal components axes on a combination of Procrustes superimposition and elliptic Fourier descriptors (30 harmonics).

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    <p>The first two canonical axes are represented, totaling 80.67% of total variance. Chr1 congenics are close to 66H, while Chr13 congenics partially overlap with B6. The shape of Chr18 congenics is intermediate between that of the two parental strains.</p

    Plots of the first and second canonical axes from LDA with elliptic Fourier descriptor on nasal bone shape to assess the mode of inheritance of the three chromosomal regions.

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    <p>A: The first and second canonical axes for 66H, B6, Chr1 (Chr1 congenics), and Chr1H (heterozygotes for Chr1) are represented, totaling 95.5% of the total variation and showed the same phenotype between 66H and Chr1S whereas Chr1H exhibited a shape difference with B6 and Chr1S. Therefore Chr 1 QTL is semi-dominant. B: the first and second canonical axes of 66H, B6, Chr13 (Chr13 congenics), Chr13H (heterozygotes for Chr13) accounted for 92.4% of the total shape variation displayed no specific inheritance pattern. Neither heterozygotes nor homozygotes for Chr13 are different from B6 C: the first and second canonical axes of 66H, B6, Chr18 (Chr18 congenics), Chr18H (heterozygotes for Chr18) represented 95.4% of the total variance exhibited a similar intermediate shape between 66H and B6 for Chr18H and Chr18S. Therefore Chr18 QTL is dominantly inherited. For the explanation of the shape changes, see <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0037721#pone-0037721-g003" target="_blank">Figure 3</a>. Shape changes were not amplified.</p

    Evaluation of epistatic interactions between the three congenic fragments. Each graph represents B6, 66H, two congenic strains and the corresponding bicongenic strain.

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    <p>A: The first and second canonical axes for 66H, B6, Chr1, Chr13, and Chr1+13 are represented, totaling 89.4% of the total variation and showed the same phenotype between Chr13 and Chr1+13 indicating that Chr13 segment decreases the effect of Chr1 QTL. B: The first and second canonical axes for 66H, B6, Chr13, Chr18 and Crh13+18 displayed 82.44% of the total variance and exhibited no differences in nasal bone shape between Chr 18 and Chr13+18. Therefore Chr13 QTL has no effect when combined with Chr18. C: The first and second canonical axes for 66H, B6, Chr1, Chr18 and Chr1+18 represented 81.7% of the nasal bone shape variation. Chr1+Chr18 bicongenic mice show a phenotype similar to that of the Chr1 congenic and 66H. For the explanation of the shape changes, see <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0037721#pone-0037721-g003" target="_blank">Figure 3</a>. No amplification of the nasal bone shape was effected.</p

    Comparison of nasal bone shape in B6, 66H and their F1 hybrids by linear discriminant analysis (LDA) based on 15 principal components axes on a combination of Procrustes superimposition and elliptic Fourier descriptors (30 harmonics).

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    <p>The first and second canonical axes were represented. The number of mice in each group is given in parentheses. Shapes drawn outside the graph describe nasal bone shape variations associated with low values (dashed line) or high values (solid line) along the axes. B6 and 66H fall into two well separated groups. F1 hybrids are distinct from either parent. Shape drawn outside the scatterplot, calculated with a multivariate regression, describes nasal bone shape variation along the canonical axes with low values (in dashed lines) and high values (in solid lines). No amplification and the nasal bone shape changes was effected.</p
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