2018 Scholars at Work Conference Program

Program for the 2018 Scholars at Work Conference at Minnesota State University, Mankato on March 30, 2018

2019 Scholars at Work Conference Program

Program for the 2019 Scholars at Work Conference at Minnesota State University, Mankato on March 29, 2019

Some Things are More Important than My Syllabus: Reflections on Disrupting My Teaching

Sometimes I throw my plans for class out the window to respond to some current crisis such as another mass shooting. As a sociologist I am uniquely positioned and feel a deep sense of responsibility to help students process and understand the social world as it is happening. This presentation will offer suggestions for leaning into these disruptions and incorporating time-sensitive events into course material based on my experiences

The Effect of Flexible Classroom Space on Attitudes and Grades in a Team-Based Learning Course

Flexible learning spaces are common features of newly built elementary and secondary schools, but little research has been conducted on flexible learning spaces in higher education. Spaces allowing for social learning have been found to increase student engagement; engaged students receive higher grades and tend to have better attitudes about the class. To better understand the effect classroom space has on student learning and experience, we designed a study to compare a flexible learning space with an inflexible one. Results suggest that classroom flexibility matters, but not in the ways we may expect. We discuss our findings on student attitudes and learning outcomes. We also provide some recommendations for designing effective classrooms to balance financial and practical realities while still supporting a positive classroom experience

NMDA receptor gene variations as modifiers in Huntington disease: A replication study

Several candidate modifier genes which, in addition to the pathogenic CAG repeat expansion, influence the age at onset (AO) in Huntington disease (HD) have already been described. The aim of this study was to replicate association of variations in the N-methyl D-aspartate receptor subtype genes GRIN2A and GRIN2B in the "REGISTRY" cohort from the European Huntington Disease Network (EHDN). The analyses did replicate the association reported between the GRIN2A rs2650427 variation and AO in the entire cohort. Yet, when subjects were stratified by AO subtypes, we found nominally significant evidence for an association of the GRIN2A rs1969060 variation and the GRIN2B rs1806201 variation. These findings further implicate the N-methyl D-aspartate receptor subtype genes as loci containing variation associated with AO in HD

NMDA receptor gene variations as modifiers in Huntington disease: a replication study.

none12siSeveral candidate modifier genes which, in addition to the pathogenic CAG repeat expansion, influence the age at onset (AO) in Huntington disease (HD) have already been described. The aim of this study was to replicate association of variations in the N-methyl D-aspartate receptor subtype genes GRIN2A and GRIN2B in the "REGISTRY" cohort from the European Huntington Disease Network (EHDN). The analyses did replicate the association reported between the GRIN2A rs2650427 variation and AO in the entire cohort. Yet, when subjects were stratified by AO subtypes, we found nominally significant evidence for an association of the GRIN2A rs1969060 variation and the GRIN2B rs1806201 variation. These findings further implicate the N-methyl D-aspartate receptor subtype genes as loci containing variation associated with AO in HD.openC. Saft; J. T. Epplen; S. Wieczorek; G. B. Landwehrmeyer; R. A. C;J. G. de;M. Dose;S. J. Tabrizi;D. Craufurd;R. E. G.;Investigators of the European Huntington's Disease Network [E. Di Maria];L. ArningC., Saft; J. T., Epplen; S., Wieczorek; G. B., Landwehrmeyer; R. A., C; J. G., De; M., Dose; S. J., Tabrizi; D., Craufurd; R. E., G.; DI MARIA, Emilio; L., Arnin

NMDA receptor gene variations as modifiers in Huntington disease: a replication study.

Several candidate modifier genes which, in addition to the pathogenic CAG repeat expansion, influence the age at onset (AO) in Huntington disease (HD) have already been described. The aim of this study was to replicate association of variations in the N-methyl D-aspartate receptor subtype genes GRIN2A and GRIN2B in the “REGISTRY” cohort from the European Huntington Disease Network (EHDN). The analyses did replicate the association reported between the GRIN2A rs2650427 variation and AO in the entire cohort. Yet, when subjects were stratified by AO subtypes, we found nominally significant evidence for an association of the GRIN2A rs1969060 variation and the GRIN2B rs1806201 variation. These findings further implicate the N-methyl D-aspartate receptor subtype genes as loci containing variation associated with AO in HD