Deep Level Transient Spectroscopy in Quantum Dot Characterization

Deep level transient spectroscopy (DLTS) for investigating electronic properties of self-assembled InAs/GaAs quantum dots (QDs) is described in an approach, where experimental and theoretical DLTS data are compared in a temperature-voltage representation. From such comparative studies, the main mechanisms of electron escape from QD-related levels in tunneling and more complex thermal processes are discovered. Measurement conditions for proper characterization of the levels by identifying thermal and tunneling processes are discussed in terms of the complexity resulting from the features of self-assembled QDs and multiple paths for electron escape

Dependence Logic with Generalized Quantifiers: Axiomatizations

We prove two completeness results, one for the extension of dependence logic by a monotone generalized quantifier Q with weak interpretation, weak in the meaning that the interpretation of Q varies with the structures. The second result considers the extension of dependence logic where Q is interpreted as "there exists uncountable many." Both of the axiomatizations are shown to be sound and complete for FO(Q) consequences.Comment: 17 page

Nitrogen removal in marine environments: recent findings and future research challenges

Respiratory reduction of nitrate (denitrification) is recognized as the most important process converting biologically available (fixed) nitrogen to N2. In current N cycle models, a major proportion of global marine denitrification (50–70%) is assumed to take place on the sea floor, particularly in organic rich continental margin sediments. Recent observations indicate that present conceptual views of denitrification and pathways of nitrate reduction and N2 formation are incomplete. Alternative N cycle pathways, particularly in sediments, include anaerobic ammonium oxidation to nitrite, nitrate and N2 by Mn-oxides, and anaerobic ammonium oxidation coupled to nitrite reduction and subsequent N2 mobilization. The discovery of new links and feedback mechanisms between the redox cycles of, e.g., C, N, S, Mn and Fe casts doubt on the present general understanding of the global N cycle. Recent models of the oceanic N budget indicate that total inputs are significantly smaller than estimated fixed N removal. The occurrence of alternative N reaction pathways further exacerbates the apparent imbalance as they introduce additional routes of N removal. In this contribution, we give a brief historical background of the conceptual understanding of N cycling in marine ecosystems, emphasizing pathways of aerobic and anaerobic N mineralization in marine sediments, and the implications of recently recognized metabolic pathways for N removal in marine environments

Considering Polymorphism in Change-Based Test Suite Reduction

With the increasing popularity of continuous integration, algorithms for selecting the minimal test-suite to cover a given set of changes are in order. This paper reports on how polymorphism can handle false negatives in a previous algorithm which uses method-level changes in the base-code to deduce which tests need to be rerun. We compare the approach with and without polymorphism on two distinct cases ---PMD and CruiseControl--- and discovered an interesting trade-off: incorporating polymorphism results in more relevant tests to be included in the test suite (hence improves accuracy), however comes at the cost of a larger test suite (hence increases the time to run the minimal test-suite).Comment: The final publication is available at link.springer.co

Homogenization of random degenerated nonlinear monotone operators

This paper deals with homogenization of random nonlinear monotone operators in divergence form. We assume that the structure conditions (strict monotonicity and continuity conditions) degenerate and are given in terms of a weight function. Under proper integrability assumptions on the weight function we construct the effective operator and prove the homogenization result

Mass Fractionation and Energy Distribution of Sputtered Monatomic Positive Ions

Secondary ion yields in sputtering depend significantly on the mass of the emitted species. Ionization as observed by secondary ion mass spectrometry is characterized by isotope fractionation; the yield of an isotope ion of mass Mi being proportional to M-, where a varies with the emitted species, its kinetic energy Ek, and the matrix. By means of SIMS, isotope ratios have been measured for ions at energies up to ca 120 eV in different metallic matrices. For singly charged positive monatomic ions, a has been found to range between O and ca 4. While a may drop steeply at low or moderate Ek, at higher energies the gradient decreases and usually becomes positive. To some extent the trends of a are complementary to those of the energy dependence of elemental ion yields. In the present work, the main tendencies are surveyed for pure element matrices as well as for several elements sputtered from a given metallic matrix. It is attempted to correlate a with the energy distributions of ionic yields. Isotope effects appear inherent in all three basic mechanisms of ion emission, i.e., in sputter yield, ionization, and charge survival

Survival of Clostridium perfringens During Simulated Transport and Stability of Some Plasmid-borne Toxin Genes under Aerobic Conditions

Clostridium perfringens is a pathogen of great concern in veterinary medicine, because it causes enteric diseases and different types of toxaemias in domesticated animals. It is important that bacteria in tissue samples, which have been collected in the field, survive and for the classification of C. perfringens into the correct toxin group, it is crucial that plasmid-borne genes are not lost during transportation or in the diagnostic laboratory. The objectives of this study were to investigate the survival of C. perfringens in a simulated transport of field samples and to determine the stability of the plasmid-borne toxin genes cpb1 and etx after storage at room temperature and at 4°C. Stability of the plasmid-borne genes cpb1 and etx of C. perfringens CCUG 2035, and cpb2 from C. perfringens CIP 106526, JF 2255 and 6 field isolates in aerobic atmosphere was also studied. Survival of C. perfringens was similar in all experiments. The cpb1 and etx genes were detected in all isolates from samples stored either at room temperature or at 4°C for 24–44 h. Repeated aerobic treatment of C. perfringens CCUG 2035 and CIP 106526 did not result in the loss of the plasmid-borne genes cpb1, cpb2 or etx. Plasmid-borne genes in C. perfringens were found to be more stable than generally reported. Therefore, C. perfringens toxinotyping by PCR can be performed reliably, as the risk of plasmid loss seems to be a minor problem

High synovial expression of the inhibitory FcγRIIb in rheumatoid arthritis

Activating Fc gamma receptors (FcγRs) have been identified as having important roles in the inflammatory joint reaction in rheumatoid arthritis (RA) and murine models of arthritis. However, the role of the inhibitory FcγRIIb in the regulation of the synovial inflammation in RA is less known. Here we have investigated synovial tissue from RA patients using a novel monoclonal antibody (GB3) specific for the FcγRIIb isoform. FcγRIIb was abundantly expressed in synovia of RA patients, in sharp contrast to the absence or weak staining of FcγRIIb in synovial biopsies from healthy volunteers. In addition, the expression of FcγRI, FcγRII and FcγRIII was analyzed in synovia obtained from early and late stages of RA. Compared with healthy synovia, which expressed FcγRII, FcγRIII but not FcγRI, all activating FcγRs were expressed and significantly up-regulated in RA, regardless of disease duration. Macrophages were one of the major cell types in the RA synovium expressing FcγRIIb and the activating FcγRs. Anti-inflammatory treatment with glucocorticoids reduced FcγR expression in arthritic joints, particularly that of FcγRI. This study demonstrates for the first time that RA patients do not fail to up-regulate FcγRIIb upon synovial inflammation, but suggests that the balance between expression of the inhibitory FcγRIIb and activating FcγRs may be in favour of the latter throughout the disease course. Anti-inflammatory drugs that target activating FcγRs may represent valuable therapeutics in this disease