728 research outputs found

    Triangle singularity in the J/ψ→K+K−f0(980)(a0(980))J/\psi \rightarrow K^+ K^- f_0(980)(a_0(980)) decays

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    We study the J/ψ→K+K−f0(980)(a0(980))J/\psi \rightarrow K^+ K^- f_0(980)(a_0(980)) reaction and find that the mechanism to produce this decay develops a triangle singularity around Minv(K−f0/K−a0)≈1515M_{\rm inv}(K^- f_0/K^- a_0) \approx 1515~MeV. The differential width dΓ/dMinv(K−f0/K−a0)d\Gamma / dM_{\rm inv}(K^- f_0/K^- a_0) shows a rapid growth around the invariant mass being 1515~MeV as a consequence of the triangle singularity of this mechanism, which is directly tied to the nature of the f0(980)f_0(980) and a0(980)a_0(980) as dynamically generated resonances from the interaction of pseudoscalar mesons. The branching ratios obtained for the J/ψ→K+K−f0(980)(a0(980))J/\psi \rightarrow K^+ K^- f_0(980)(a_0(980)) decays are of the order of 10−510^{-5}, accessible in present facilities, and we argue that their observation should provide relevant information concerning the nature of the low-lying scalar mesons.Comment: 12 pages, 8 figures, published in EPJ

    Revisiting the Ω(2012)\Omega(2012) as a hadronic molecule and its strong decays

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    Recently, the Belle collaboration measured the ratios of the branching fractions of the newly observed Ω(2012)\Omega(2012) excited state. They did not observe significant signals for the Ω(2012)→KˉΞ∗(1530)→KˉπΞ\Omega(2012) \to \bar{K} \Xi^*(1530) \to \bar{K} \pi \Xi decay, and reported an upper limit for the ratio of the three body decay to the two body decay mode of Ω(2012)→KˉΞ\Omega(2012) \to \bar{K} \Xi. In this work, we revisit the newly observed Ω(2012)\Omega(2012) from the molecular perspective where this resonance appears to be a dynamically generated state with spin-parity 3/2−3/2^- from the coupled channels interactions of the KˉΞ∗(1530)\bar{K} \Xi^*(1530) and ηΩ\eta \Omega in ss-wave and KˉΞ\bar{K} \Xi in dd-wave. With the model parameters for the dd-wave interaction, we show that the ratio of these decay fractions reported recently by the Belle collaboration can be easily accommodated.Comment: Published version. Published in Eur.\ Phys.\ J.\ C {\bf 80}, 361 (2020

    Hadronic molecular states with the quark contents bcsˉqˉbc\bar{s}\bar{q}, bcˉsqˉb\bar{c}s\bar{q}, and bcˉsˉqb\bar{c}\bar{s}q

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    We study the hadronic molecular states with the quark content bcsˉqˉbc\bar{s}\bar{q} by investigating the interactions of the BˉsD\bar{B}_s D, BˉDs\bar{B} D_s, Bˉs∗D\bar{B}_s^* D, Bˉ∗Ds\bar{B}^* D_s, BˉsD∗\bar{B}_s D^*, BˉDs∗\bar{B} D_s^*, Bˉs∗D∗\bar{B}_s^* D^*, and Bˉ∗Ds∗\bar{B}^* D_s^* systems. By solving the Bethe-Salpeter equation within the extended local hidden gauge formalism, we find altogether six poles qualifying as possible hadronic molecular states: one pole of JP=0+J^P=0^+ below the BˉsD\bar{B}_s D-BˉDs\bar{B}D_s threshold, one pole of JP=1+J^P=1^+ below the Bˉs∗D\bar{B}_s^* D-Bˉ∗Ds\bar{B}^* D_s threshold, one pole of JP=1+J^P=1^+ below the BˉsD∗\bar{B}_s D^*-BˉDs∗\bar{B}D_s^* threshold, and three poles of JP=0+/1+/2+J^P=0^+/1^+/2^+ below the Bˉs∗D∗\bar{B}_s^* D^*-Bˉ∗Ds∗\bar{B}^* D_s^* threshold. Their binding energies are calculated to be about 10-20 MeV with the cut-off momentum qmax=600 MeVq_\textrm{max}=600\textrm{ MeV}. Similarly, we study the hadronic molecular states with bscˉqˉbs\bar{c}\bar{q} by investigating the interactions of the BˉDˉs\bar{B}\bar{D}_s, BˉcKˉ\bar{B}_c\bar{K}, Bˉ∗Dˉs\bar{B}^*\bar{D}_s, Bˉc∗Kˉ\bar{B}_c^*\bar{K}, BˉDˉs∗\bar{B}\bar{D}_s^*, BˉcKˉ∗\bar{B}_c\bar{K}^*, Bˉ∗Dˉs∗\bar{B}^*\bar{D}_s^*, Bˉc∗Kˉ∗\bar{B}_c^*\bar{K}^* systems, and the states with bqcˉsˉbq\bar{c}\bar{s} by investigating the interactions of the BˉsDˉ\bar{B}_s\bar{D}, BˉcK\bar{B}_cK, Bˉs∗Dˉ\bar{B}_s^*\bar{D}, Bˉc∗K\bar{B}_c^*K, BˉsDˉ∗\bar{B}_s\bar{D}^*, BˉcK∗\bar{B}_cK^*, Bˉs∗Dˉ∗\bar{B}_s^*\bar{D}^*, Bˉc∗K∗\bar{B}_c^*K^* systems. However, no deeply-bound poles are found in these systems.Comment: 12 pages, 9 figures, revised version to be published in PR

    A case of ‘fat-free’ pleomorphic lipoma occurring in the upper back and axilla simultaneously

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    Pleomorphic lipoma is a rare neoplasm that predominantly occurs in the dermis or subcutis of the posterior neck, upper back, and shoulders. Although pleomorphic lipoma is a benign tumor, it may contain atypical cells. As a variant of spindle cell lipoma, pleomorphic lipoma clinically presents as a slow-growing and well-circumscribed subcutaneous mass. Rarely, some patients have multiple lesions. Histologically, pleomorphic lipoma is composed of mature fat, bland spindle-shaped mesenchymal cells, and coarse ‘rope-like’ collagen bands. In addition, lipoma contains multinucleated floret-like giant cells. Although spindle cell lipoma/pleomorphic lipoma with little fat was seen in the original series described by Enzinger and Harvey, cases with little to no fat remain diagnostically challenging. Herein, we report a case of ‘fat-free’ pleomorphic lipoma occurring in the upper back and axilla simultaneously. Although the lipoma was typically composed of bland spindle-shaped cells, rope-like collagen, scattered floret-like giant cells, and striking stromal myxoid change in the background, mature fat was absent. Immunohistochemical analyses showed positive staining for CD34, vimentin, and Bcl-2, and negative staining for S100, confirming the diagnosis of pleomorphic lipoma

    Impaired ubiquitin–proteasome system activity in the synapses of Huntington's disease mice

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    Huntington's disease (HD) is caused by the expansion of a polyglutamine tract in the N-terminal region of huntingtin (htt) and is characterized by selective neurodegeneration. In addition to forming nuclear aggregates, mutant htt accumulates in neuronal processes as well as synapses and affects synaptic function. However, the mechanism for the synaptic toxicity of mutant htt remains to be investigated. We targeted fluorescent reporters for the ubiquitin–proteasome system (UPS) to presynaptic or postsynaptic terminals of neurons. Using these reporters and biochemical assays of isolated synaptosomes, we found that mutant htt decreases synaptic UPS activity in cultured neurons and in HD mouse brains that express N-terminal or full-length mutant htt. Given that the UPS is a key regulator of synaptic plasticity and function, our findings offer insight into the selective neuronal dysfunction seen in HD and also establish a method to measure synaptic UPS activity in other neurological disease models
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