3,047 research outputs found
The Liouville Theorem for a Quasi-Linear Elliptic Partial Differential Equation
The classical Liouville Theorem of analytic function theory can be stated in either of two equivalent forms: The Liouville Theorem states: If f(w) is analytic and bounded throughout the finite w-plane, then f(w) is constant. If z(x, y) is a real valued function of the real variables x and y which is a solution of zxx + zyy = 0 and is bounded either above or below throughout the finite plane, then z(x, y) is a constant. Here we are concerned with the question of whether or not the second formulation of the above theorem is valid for solutions of more general elliptic partial differential equations
The Liouville Theorem for a Quasi-Linear Elliptic Partial Differential Equation
The classical Liouville Theorem of analytic function theory can be stated in either of two equivalent forms: The Liouville Theorem states: If f(w) is analytic and bounded throughout the finite w-plane, then f(w) is constant. If z(x, y) is a real valued function of the real variables x and y which is a solution of zxx + zyy = 0 and is bounded either above or below throughout the finite plane, then z(x, y) is a constant. Here we are concerned with the question of whether or not the second formulation of the above theorem is valid for solutions of more general elliptic partial differential equations
Final State Interactions and CP Violation in
Using chiral perturbation theory we calculate the imaginary parts of the form factors that arise from and
rescattering. We discuss their influence on CP violating variables in .Comment: ; 12 pages, 2 figures, TeX format; uses epsf.tex, tables.tex, and
phyzzx.te
The CP-violating asymmetry in \eta\to\pi^+ \pi^- e^+e^-
We study the CP-violating asymmetry {\cal A}_{\rm CP}, which arises, in
\eta\to\pi^+\pi^- e^+e^-, from the angular correlation of the e^+ e^- and
\pi^+\pi^- planes due to the interference between the magnetic and electric
decay amplitudes. With the phenomenologically determined magnetic amplitude and
branching ratio as input, the asymmetry, induced by the electric bremsstrahlung
amplitude through the CP-violating decay \eta\to\pi^+\pi^-, and by an
unconventional tensor type operator, has been estimated respectively. The upper
bound of {\cal A}_{\rm CP} from the former is about 10^{-3}, and the asymmetry
from the latter might be up to O(10^{-2}). One can therefore expect that this
CP asymmetry would be an interesting CP-violating observable for the future
precise measurements in the \eta factories.Comment: LaTeX, 6 pages. One reference corrected, and some new references
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Bright Electroplating of Copper in the Acid Bath
Plating of various objects with mirror-like surfaces of chromium, nickel, and other metals has expanded considerably during the past decade, and now ranks as an important enterprise, particularly with respect to the automotive industry
Exponentially Increasing Incidences of Cutaneous Malignant Melanoma in Europe Correlate with Low Personal Annual UV Doses and Suggests 2 Major Risk Factors
For several decades the incidence of cutaneous malignant melanoma (CMM) steadily increased in fair-skinned, indoor-working people around the world. Scientists think poor tanning ability resulting in sunburns initiate CMM, but they do not understand why the incidence continues to increase despite the increased use of sunscreens and formulations offering more protection. This paradox, along with lower incidences of CMM in outdoor workers, although they have significantly higher annual UV doses than indoor workers have, perplexes scientists. We found a temporal exponential increase in the CMM incidence indicating second-order reaction kinetics revealing the existence of 2 major risk factors. From epidemiology studies, we know one major risk factor for getting CMM is poor tanning ability and we now propose the other major risk factor may be the Human Papilloma Virus (HPV) because clinicians find β HPVs in over half the biopsies. Moreover, we uncovered yet another paradox; the increasing CMM incidences significantly correlate with decreasing personal annual UV dose, a proxy for low vitamin D3 levels. We also discovered the incidence of CMM significantly increased with decreasing personal annual UV dose from 1960, when it was almost insignificant, to 2000. UV and other DNA-damaging agents can activate viruses, and UV-induced cytokines can hide HPV from immune surveillance, which may explain why CMM also occurs in anatomical locations where the sun does not shine. Thus, we propose the 2 major risk factors for getting CMM are intermittent UV exposures that result in low cutaneous levels of vitamin D3 and possibly viral infection
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