12 research outputs found
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
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- The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group
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- ZZ Chong
- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Carbon-bridged oligo(p-phenylenevinylene)s for photostable and broadly tunable, solution-processable thin film organic lasers
- Author
- A Camposeo
- BK Yap
- C Grivas
- C Karnutsch
- C Karnutsch
- C Vannahme
- E Heydari
- E Ishow
- E Laquai
- EM Calzado
- EM Calzado
- EM Calzado
- ER Martins
- EY Choi
- G Tsiminis
- H Langhals
- J Casado
- J Clark
- J Herrnsdorf
- K Sugiyasu
- LJ Guo
- MA Díaz-García
- MA Noginov
- MG Ramírez
- MG Ramírez
- MG Ramírez
- MG Ramírez
- MG Ramírez
- MM Mroz
- P Mayorga Burrezo
- R Sastre
- R Xia
- R Xia
- S Chénais
- S Klinkhammer
- V Navarro-Fuster
- V Navarro-Fuster
- X Zhu
- X Zhu
- Y Wang
- Y Yang
- Y Yang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Solution-processed nanographene distributed feedback lasers
- Author
- A Camposeo
- A Yang
- AJC Kuehne
- C Grivas
- D Wang
- D Yadav
- E Heydari
- EM Calzado
- EM Calzado
- EM Calzado
- F Bonaccorso
- G Qian
- G Tsiminis
- GM Paternò
- GM Paternò
- H Kogelnik
- IDW Samuel
- J Wu
- JA Myer
- JA Quintana
- JRC Smirnov
- K Nakada
- KP Loh
- L Cerdán
- L Chen
- M Anni
- M Karl
- M Kolmer
- M Lu
- M Morales-Vidal
- M Morales-Vidal
- M Morales-Vidal
- MD McGehee
- MG Ramirez
- MG Ramírez
- MG Ramírez
- MG Ramírez
- MG Stanford
- N Tsutsumi
- P Das
- Q Chen
- R Englman
- R Kazarinov
- R Muñoz-Mármol
- R Swanepoel
- S Klinkhammer
- S Schauer
- T Enoki
- T Ohta
- T Zhai
- V Navarro-Fuster
- V Navarro-Fuster
- X Wang
- X-Y Wang
- Y Gu
- Y Ni
- Y Wang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Polymer Lasers and Optical Amplifiers
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
A novel small molecule, HK-156, inhibits lipopolysaccharide-induced activation of NF-κB signaling and improves survival in mouse models of sepsis
- Author
- A Barochia
- A Rossi
- AF Suffredini
- B Beutler
- B Liu
- B Liu
- CG Leon
- CH Leung
- CH Leung
- CS Robbins
- DS Wheeler
- E Abraham
- EM Palsson-McDermott
- F Yang
- FH Sarkar
- H Wang
- HL Pahl
- J Dong
- Jian-ping Fang
- Jie Li
- Kan Ding
- KJ Tracey
- L Ling
- LA O'Neill
- M Delhase
- M Guha
- M Guha
- M Landstrom
- MA Calzado
- MS Hayden
- MS Hayden
- P Bamborough
- RC Bone
- RP Wang
- RS Munford
- S Gerondakis
- S Sato
- S Vallabhapurapu
- SF Liu
- SM Miggin
- T Huxford
- T Kawai
- T Lawrence
- T Secher
- T Suzuki
- TD Gilmore
- V Anest
- W Huang
- Wen-feng Liao
- Y Lee
- Y Yamamoto
- Y Yamamoto
- YA Ivanenkov
- Yang Liu
- YC Lu
- You-hong Hu
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Crystalline Organic Materials Toward Laser Application
- Author
- A Bilge
- B Yang
- BBY Hsu
- BBY Hsu
- C-H Zhao
- CJ Chiang
- CL Wang
- CW Tang
- EB Namdas
- EB Namdas
- EM Calzado
- F He
- F Hide
- F Hide
- FP Schäfer
- H Uoyama
- I Sokolik
- J Cornil
- J Cornil
- J Zaumseil
- JE Anthony
- JE Anthony
- JH Andrews
- JL Brédas
- JS Swensen
- JS Wilson
- K Kobayashi
- L Yao
- M Kasha
- M Muccini
- M Ullah
- MA Baldo
- MC Gwinner
- N Tessler
- PFR Atkins
- Q Zhang
- Q Zhang
- R Marcus
- S Chénais
- S Sinha
- SK Kim
- SW Yin
- T Förster
- T Siegrist
- TH Maiman
- V Podzorov
- VC Sundar
- VC Sundar
- WJ Feast
- WJ Li
- WJ Li
- Y Cao
- Y Hong
- Y Qian
- YP Li
- Z Shuai
- ZQ Xie
- ZQ Xie
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Disentangling the influence of environmental and anthropogenic factors on the distribution of endemic vascular plants in Sardinia
- Author
- A Balmford
- A Grill
- A Guisan
- A Mazziotta
- AD Caperta
- AS Kallimanis
- AS Wulff
- B Céspedes
- C Fløjgaard
- C Körner
- CDL Orme
- D Smiraglia
- ED Concepción
- EM Cañadas
- Eva Maria Cañadas
- F Conti
- F Médail
- FJ Pérez-García
- G Bacchetta
- G Bacchetta
- G Bacchetta
- G Bonanno
- G Fenu
- G Fenu
- G Kier
- G Mansion
- Gianluigi Bacchetta
- Giuseppe Fenu
- J Li
- JD Thompson
- L Anselin
- L Anselin
- L Bosso
- L Carmignani
- Lorenzo Peruzzi
- M Abdelaal
- M Fois
- M Fois
- M Fois
- M Fois
- M Panitsa
- M Panitsa
- M Porceddu
- Mauro Fois
- MB Araújo
- MJ Steinbauer
- MJ Steinbauer
- P Birnbaum
- P Trigas
- P Trigas
- R Fernández-Calzado
- R Lecis
- R Ohlemüller
- R Real
- R Vodă
- RH MacArthur
- RJ Hijmans
- S Canu
- S Harrison
- S Sfenthourakis
- SDH Irl
- TF Stuessy
- U Grömping
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- Field of study
Wnt signaling through T-cell factor phosphorylation
- Author
- A Gregorieff
- A Hammerlein
- A Ruzov
- A Schambony
- AF Schier
- AJ Mikels
- AJ Mikels
- AN Billin
- B Goldstein
- B Riggleman
- BM Gumbiner
- BT MacDonald
- BT Phillips
- C Kanei-Ishii
- C Mosimann
- C Rinaldo
- C van Genderen
- CE Rocheleau
- CE Rocheleau
- CJ Thorpe
- CJ Thorpe
- CY Choi
- CY Logan
- D Calvo
- D Gradl
- D Levanon
- D Onichtchouk
- DL Daniels
- DM Eisenmann
- DW Houston
- E Brunner
- E Doxakis
- E Lee
- EA Kim
- EK Vladar
- EM De Robertis
- F Liu
- F Ye
- F Yi
- FA Atcha
- FM van Roy
- G Chen
- G D'Orazi
- G Roel
- G Wei
- GM Pierantoni
- H Brantjes
- H Clevers
- H Haegel
- H Hikasa
- H Hikasa
- H Hikasa
- H Nguyen
- H Nguyen
- H Shibuya
- H Song
- HC Korswagen
- HC Korswagen
- HJ Standley
- I Dauth
- ID Keenan
- III Kidd AR
- J Behrens
- J Bonner
- J Galceran
- J Heasman
- J Huelsken
- J Li
- J Riese
- J Roose
- J Sierra
- JE Schmidt
- JI Park
- K Isono
- K Itoh
- K Mizumoto
- K Vleminckx
- K Yamaguchi
- KM Cadigan
- KR Siegfried
- KR Siegfried
- L Arce
- L Arce
- L Smit
- M Brannon
- M Brannon
- M Epstein
- M Gasperowicz
- M Nishita
- M Nishita
- M van de Wetering
- MA Calzado
- MC Lo
- MC Ramel
- MD Meneghini
- MF Cole
- MF Maduro
- MF Maduro
- MJ Fan
- N Lam
- N Pilon
- P Bhanot
- P Shetty
- Q Zhang
- R Ladher
- R Lin
- R Lin
- R Najdi
- RA Cavallo
- RI Dorsky
- S Angers
- S Barolo
- S Hoppler
- S Huang
- S Kondo
- S Lin
- S Miravet
- S Wang
- Sergei Y Sokol
- SH Louie
- T Grigoryan
- T Haremaki
- T Ishitani
- T Ishitani
- T Ishitani
- T Kaletta
- T Shimizu
- T Valenta
- TG Hofmann
- TG Hofmann
- TH Shin
- TJ Klein
- V Gawantka
- V Sander
- W Lee
- W Lu
- WJ Nelson
- WL Tam
- X He
- XQ Gan
- Y Imai
- YH Kim
- YM Elkouby
- Publication venue
- Nature Publishing Group
- Publication date
- Field of study
Embryonic signaling pathways often lead to a switch from default repression to transcriptional activation of target genes. A major consequence of Wnt signaling is stabilization of β-catenin, which associates with T-cell factors (TCFs) and 'converts' them from repressors into transcriptional activators. The molecular mechanisms responsible for this conversion remain poorly understood. Several studies have reported on the regulation of TCF by phosphorylation, yet its physiological significance has been unclear: in some cases it appears to promote target gene activation, in others Wnt-dependent transcription is inhibited. This review focuses on recent progress in the understanding of context-dependent post-translational regulation of TCF function by Wnt signaling
Molecular targets for treatment of kidney fibrosis
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- A Krupa
- A Lautrette
- A Leask
- AB Sanz
- AC Chung
- AC Chung
- B Bielesz
- B Wang
- BC Liu
- BM Brenner
- BS Kasinath
- BX Li
- C Nathan
- C Ruster
- C Zoja
- CJ Kelly
- D Shan
- DJ Kelly
- DO Schlondorff
- DR Holmes Jr
- E Sanchez-Lopez
- E Vilayur
- EJ Lewis
- EJ Lewis
- EM Zeisberg
- EP Bottinger
- EP Bottinger
- F Eitner
- F Strutz
- F Strutz
- F Yang
- FN Ziyadeh
- G Wolf
- G Wolf
- GE Striker
- GJ Ko
- GR Grotendorst
- H Shimizu
- H Sugimoto
- H Takayama
- H Yokoi
- H Yokoi
- HJ Anders
- HY Lan
- I Hwang
- J Chen
- J Massague
- J Massague
- J Soma
- J Soma
- JC Bonner
- JF Navarro
- JG Abreu
- JH Li
- JJ Letterio
- JL Wrana
- John C. He
- K Pulkkinen
- K Schroder
- K Sharma
- K Sharma
- K Takakuta
- KA Nath
- L Yaswen
- LK Jones
- M Abbate
- M Adamczak
- M Bitzer
- M Guha
- M Iwano
- M Kato
- M Kato
- M Murea
- M Ots
- M Rossini
- M Zeisberg
- M Zeisberg
- M Zeisberg
- M Zeisberg
- MA Calzado
- Madhav C. Menon
- ME Cho
- MI Phillips
- MT Gandolfo
- MT Grande
- N Liu
- N Picard
- P Boor
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- P Kantharidis
- P Kavsak
- Peter Y. Chuang
- Q Wang
- Q Wang
- R Gong
- RC Harris
- RM Perkins
- S Hao
- S Mackensen-Haen
- S Miyazawa
- SA Mezzano
- Semedo P Correa-Costa M, Antonio Cenedeze M, Maria Avancini Costa Malheiros D, Antonia dos Reis M, Shimizu MH, Seguro AC, Pacheco-Silva A, Saraiva Camara NO
- SG Adler
- SL Lin
- SL Lin
- SL Lin
- SL Lin
- SN Wang
- SP RamachandraRao
- T Ostendorf
- T Wada
- TJ Tsai
- TS Doi
- TT Tapmeier
- V Esteban
- V Leroy
- W He
- W Kriz
- W Wang
- WA Border
- WA Border
- WA Border
- WB Melenhorst
- WG Jiang
- XM Chen
- Y Isaka
- Y Ito
- Y Jin
- Y Liu
- Y Liu
- Y Liu
- Y Liu
- Y Sirin
- Y Wang
- YY Wan
- Z Liu
- Z Zhang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Clinical and genetic characteristics of late-onset Huntington's disease
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- Aanonsen No
- Aaserud O
- Abbruzzese G
- Acera Gil Ma
- Ackermann O
- Adarmes A
- Agarwal V
- Agosti C
- Akhtar S
- Alaerts N
- Alarcón Md
- Albanese A
- Allain P
- Almagro Ca
- Almeida M
- Alonso-Frech F
- Alusi S
- Anderson M
- Andersson C
- Andrade C
- Andrew A
- Andrews T
- Andrews T
- Andrich J
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- Anjum U
- Anna Bryl Ja
- Annic A
- Antczak J
- Antequera Torres Mm
- Antonova V
- Arbelo Jm
- Arkuszewski M
- Armstrong R
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- Arntsen V
- Arques Pn
- Arran N
- Azulay Jp
- Baake V
- Babiloni B
- Bachmeier M
- Bachoud-Lévi Ac
- Bachoud-Lévi Ac
- Badei F
- Bailey W
- Banaszkiewicz K
- Bandettini di Poggio M
- Bandmann O
- Barbera Ma
- Barker Ra
- Barlati S
- Barral Vm
- Barrero F
- Barrett W
- Barth K
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- Barthélémy R
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- Becanovic K
- Bechtel N
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- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2019
- Field of study
Background: The frequency of late-onset Huntington's disease (>59 years) is assumed to be low and the clinical course milder. However, previous literature on late-onset disease is scarce and inconclusive. Objective: Our aim is to study clinical characteristics of late-onset compared to common-onset HD patients in a large cohort of HD patients from the Registry database. Methods: Participants with late- and common-onset (30\u201350 years)were compared for first clinical symptoms, disease progression, CAG repeat size and family history. Participants with a missing CAG repeat size, a repeat size of 6435 or a UHDRS motor score of 645 were excluded. Results: Of 6007 eligible participants, 687 had late-onset (11.4%) and 3216 (53.5%) common-onset HD. Late-onset (n = 577) had significantly more gait and balance problems as first symptom compared to common-onset (n = 2408) (P <.001). Overall motor and cognitive performance (P <.001) were worse, however only disease motor progression was slower (coefficient, 120.58; SE 0.16; P <.001) compared to the common-onset group. Repeat size was significantly lower in the late-onset (n = 40.8; SD 1.6) compared to common-onset (n = 44.4; SD 2.8) (P <.001). Fewer late-onset patients (n = 451) had a positive family history compared to common-onset (n = 2940) (P <.001). Conclusions: Late-onset patients present more frequently with gait and balance problems as first symptom, and disease progression is not milder compared to common-onset HD patients apart from motor progression. The family history is likely to be negative, which might make diagnosing HD more difficult in this population. However, the balance and gait problems might be helpful in diagnosing HD in elderly patients