Feminism and Couple Finance: Power as a Mediator Between Financial Processes and Relationship Outcomes

Feminism is rarely used as a theoretical framework for couple finance research. The purposes of the present paper are (a) to discuss couple finance research in the context of feminism to encourage more frequent and explicit use of feminism in couple finance research, (b) to present a gender and couple finances model, and (c) to test this model with longitudinal dyadic data. Using actor-partner interdependence models (APIM) and data from 327U.S. mixed-gender couples, relational power was explored as a potential mediator between four couple financial processes (earners of money, access to money, management of money, and conflict about money) and two relationship outcomes (relationship quality and relationship stability). Results suggest that couple financial processes are associated with relationship outcomes and with joint management as well as low conflict being key longitudinally. Additionally, although power may not play a mediating role, it appears to be connected to couple financial processes and relationship outcomes concurrently. Gender differences as well as both actor and partner effects are explored. This research has implications for researchers, clinicians, and educators. For example, clinicians may want to encourage their clients to use joint bank accounts, manage their money jointly, and minimize financial conflict. Gender, and therefore power, are inseparably tied to couple finances. When both spouses are involved in financial processes, partners tend to be more empowered, and relationship quality and stability tend to be higher.12 month embargo; published online: 26 November 2018This item from the UA Faculty Publications collection is made available by the University of Arizona with support from the University of Arizona Libraries. If you have questions, please contact us at [email protected]

The Budget and the Bedroom: Associations between Financial Management Behaviors, Perceptions of Economic Pressure, and Sexual Satisfaction

Although money and sex are both salient to romantic relationships, previous literature has rarely examined the association between the two. In the current study, we evaluate associations between financial management behaviors, perceptions of economic pressure, and sexual satisfaction. We used nationally representative opposite-sex newlywed dyadic data (N = 1,447 couples) and an actor-partner interdependent structural equation model to test these associations. Results indicate that as financial therapists aid opposite-sex newlywed clients in their financial management, they may also be lessening perceptions of economic pressure. For wives, this lessening of perceptions of economic pressure may benefit husbands’ and wives’ sexual satisfaction. Financial therapists may consider using these findings to encourage opposite-sex newlywed clients to practice healthy financial management behaviors and communicate about finances and sex. In short, our findings suggest that for opposite-sex newlywed couples, the budget may have something to do with the bedroom

Diazepam actions in the VTA enhance social dominance and mitochondrial function in the nucleus accumbens by activation of dopamine D1 receptors.

Benzodiazepines can ameliorate social disturbances and increase social competition, particularly in high-anxious individuals. However, the neural circuits and mechanisms underlying benzodiazepines' effects in social competition are not understood. Converging evidence points to the mesolimbic system as a potential site of action for at least some benzodiazepine-mediated effects. Furthermore, mitochondrial function in the nucleus accumbens (NAc) has been causally implicated in the link between anxiety and social competitiveness. Here, we show that diazepam facilitates social dominance, ameliorating both the competitive disadvantage and low NAc mitochondrial function displayed by high-anxious rats, and identify the ventral tegmental area (VTA) as a key site of action for direct diazepam effects. We also show that intra-VTA diazepam infusion increases accumbal dopamine and DOPAC, as well as activity of dopamine D1- but not D2-containing cells. In addition, intra-NAc infusion of a D1-, but not D2, receptor agonist facilitates social dominance and mitochondrial respiration. Conversely, intra-VTA diazepam actions on social dominance and NAc mitochondrial respiration are blocked by pharmacological NAc micro-infusion of a mitochondrial complex I inhibitor or an antagonist of D1 receptors. Our data support the view that diazepam disinhibits VTA dopaminergic neurons, leading to the release of dopamine into the NAc where activation of D1-signaling transiently facilitates mitochondrial function, that is, increased respiration and enhanced ATP levels, which ultimately enhances social competitive behavior. Therefore, our findings critically involve the mesolimbic system in the facilitating effects of diazepam on social competition and highlight mitochondrial function as a potential therapeutic target for anxiety-related social dysfunctions

Gentamicin Rapidly Inhibits Mitochondrial Metabolism in High-Frequency Cochlear Outer Hair Cells

Aminoglycosides (AG), including gentamicin (GM), are the most frequently used antibiotics in the world and are proposed to cause irreversible cochlear damage and hearing loss (HL) in 1/4 of the patients receiving these life-saving drugs. Akin to the results of AG ototoxicity studies, high-frequency, basal turn outer hair cells (OHCs) preferentially succumb to multiple HL pathologies while inner hair cells (IHCs) are much more resilient. To determine if endogenous differences in IHC and OHC mitochondrial metabolism dictate differential sensitivities to AG-induced HL, IHC- and OHC-specific changes in mitochondrial reduced nicotinamide adenine dinucleotide (NADH) fluorescence during acute (1 h) GM treatment were compared. GM-mediated decreases in NADH fluorescence and succinate dehydrogenase activity were observed shortly after GM application. High-frequency basal turn OHCs were found to be metabolically biased to rapidly respond to alterations in their microenvironment including GM and elevated glucose exposures. These metabolic biases may predispose high-frequency OHCs to preferentially produce cell-damaging reactive oxygen species during traumatic challenge. Noise-induced and age-related HL pathologies share key characteristics with AG ototoxicity, including preferential OHC loss and reactive oxygen species production. Data from this report highlight the need to address the role of mitochondrial metabolism in regulating AG ototoxicity and the need to illuminate how fundamental differences in IHC and OHC metabolism may dictate differences in HC fate during multiple HL pathologies