Ambient Particulate Matter Air Pollution Exposure and Hypertension

The relationship between exposure to ambient particulate matter (PM) and hypertension in humans is controversial. The aim of the present meta-analysis was to quantify the predictability of ambient PM on the risk of incident hypertension in humans. The selection criteria included the studies that could provide quantitative estimates of the change in hypertension prevalence linked with exposure to either indicator of PM. The health outcome of “hypertension” was defined as systolic blood pressure (SBP) of 140 mmHg and greater and/or diastolic blood pressure (DBP) of 90 mmHg or greater and/or taking antihypertensive drugs or diagnosis by a physician or self-reported hypertension. A total of 647 studies were initially identified through online database searches, and finally five studies met the inclusion criteria. The combined results of reported relative risk from the five included studies revealed that ambient PM was positively associated with hypertension (OR = 1.03; 95% CI: 0.99–1.06), but this was not statistically significant. When stratified by the PM size, the results showed that the odds ratio for hypertension increased by 1.03 (95% CI: 0.93–1.14) per 10 μg/m3 increase in PM2.5, 1.04 (95% CI: 0.98–1.10) in PM10, and 0.99 (95% CI: 0.92–1.07) in PM2.5–10. In conclusion, to get more information about the associations between PM and hypertension, many studies need to be undertaken further to clarify these relationships

Unusual Compression Behavior of Columbite TiO2 via First-Principles Calculations

The physical mechanisms behind the reduction of the bulk modulus of a high-pressure cubic TiO2 phase are confirmed by first-principles calculations. An unusual and abrupt change occurs in the dependence of energy on pressure at 43 GPa, indicating a pressure-induced phase transition from columbite TiO2 to a newly-identified modified fluorite TiO2 with a Pca21 symmetry. Oxygen atom displacement in Pca21 TiO2 unexpectedly reduces the bulk modulus by 34% relative to fluorite TiO2. This discovering provides a direct evidence for understanding the compressive properties of such groups of homologous materialsComment: [email protected] or [email protected]

Air pollution emissions 2008–2018 from Australian coal mining: implications for public and occupational health

Occupational exposure limits for respirable coal dust are based on exposure during working hours, but coal miners may experience additional community-based exposures during nonworking hours. We analyzed Australia National Pollutant Inventory (NPI) data for the years 2008–2018 to estimate air pollutants (metals, nitrogen oxides, particulate matter ≤ 10 micrometers (PM10) and ≤ 2.5 micrometers (PM2.5)) originating from coal mines. PM10 levels from community-based air monitors in Queensland and New South Wales were also compared between mining and nonmining communities. Results indicated that tons of coal mined increased over the study period, and that levels of particulate matter, metals, and nitrogen oxides increased significantly over time as well. Coal mines accounted for 42.1% of national PM10 air emissions from NPI sites. PM2.5 from coal mines accounted for 19.5% of the national total, metals for 12.1%, and nitrogen oxides for 10.1%. Coal mining occurred in 57 different post codes; the 20 coal-mining post codes with the highest PM10 emissions were home to 160,037 people. Emissions of all studied pollutants were significantly higher from coal mining sites than from other types of NPI sites. Results from community-based air monitoring stations indicated significantly higher population PM10 exposure in coal mining communities than in nonmining communities. The health of the public at large is impacted by coal mining, but to the extent that miners also live near coal mining operations, their total exposure is underestimated by consideration of exposure only during working hours

Ab initio study of the formation of transparent carbon under pressure

A body-centered tetragonal carbon (bct-Carbon) allotrope has been predicted to be a transparent carbon polymorph obtained under pressure. The structural transition pathways from graphite to diamond, M-Carbon, and bct-Carbon are simulated and the lowest activation barrier is found for the graphite-bct transition. Furthermore, bct-Carbon has higher shear strength than diamond due to its perpendicular graphene-like structure. Our results provide a possible explanation for the formation of a transparent carbon allotrope via the cold compression of graphite. We also verify that this allotrope is hard enough to crack diamond.Comment: [email protected] or [email protected]

A New Algorithm for Boolean Operations on General Polygons

International audienceA new algorithm for Boolean operations on general planar polygons is presented. It is available for general planar polygons (manifold or non-manifold, with or without holes). Edges of the two general polygons are subdivided at the intersection points and touching points. Thus, the boundaryof the Boolean operation resultant polygon is made of some whole edges of the polygons after the subdivision process. We use the simplex theory to build the basic mathematical model of the new algorithm. The subordination problem between an edge and a polygon is reduced to a problem of determining whether a point is on some edges of some simplices or inside the simplices, and the associated simplicial chain of the resultant polygon is just an assembly of some simplices and their coefficients of the two polygons after the subdivision process. Examples show that the running time required bythe new algorithm is less than one-third of that bythe Rivero and Feito algorithm

3,5-Bis(4-meth­oxy­phen­yl)-4H-1,2,4-triazol-4-amine

The title compound, C16H16N4O2, crystallizes with two mol­ecules in the asymmetric unit, which are related by a non-crystallographic centre of inversion. The phenyl­ene rings are twisted out of the mean plane of the triazole ring by 19.3 (1) and 21.4 (1)° for one independent mol­ecule and by 16.3 (1) and 18.1 (1)° for the other mol­ecule. In the crystal, adjacent mol­ecules are linked by amine–triazole N—H⋯N hydrogen bonds, forming chains running along the a axis

Outdoor Air Pollution and Arterial Hypertension

Air pollution is a major environmental risk factor. There is accumulating evidence that air pollution could induce elevated blood pressure and potentiate hypertension. Acute elevations in the outdoor air pollution levels can trigger immediate or shortly delayed increases in arterial blood pressure. Moreover, few studies suggest that short-term increases in the levels of particulate and gaseous pollutants could lead to an acute onset of hypertension. Prolonged exposure to outdoor air pollution is associated with elevated blood pressure. Furthermore, some longitudinal studies have linked long-term exposure to air pollution with the incidence of hypertension. Various components of air pollution, such as inhalable particulate matter (PM2.5, PM10), nitrogen oxides, sulfur dioxide, and ozone, have shown associations with blood pressure in some studies. The hypothesized underlying mechanisms include inflammatory reactions and oxidative stress in lungs and in systemic circulation, imbalance of autonomous nervous system, and pathologic changes in vascular endothelium. In addition to “traditional” susceptible groups such as elderly individuals or patients with chronic diseases, children and pregnant women could be especially susceptible to the adverse effects of air pollution. The interplay of air pollution with the related environmental exposures, such as traffic noise and climate change, should be investigated further