4 research outputs found

    Posturography and ocular torsion data from a typical subject.

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    <p>(A) Anterioposterior sway plots without (upper row blue trace) and with (lower row red trace) 5 mA GVS for SOT 5 (eyes closed and sway-referenced support surface). The subject fell during initial exposure to GVS, but anterioposterior sway decreased to baseline levels over the 12 weeks of GVS exposure, and this recovery was maintained 6 months after adaption. (B) Torsional (roll) eye position traces without (upper row blue trace) and with (lower row red trace) 5 mA GVS. In contrast to the posturography data, the vestibulo-ocular reflex response to GVS was unchanged by repeated GVS exposures.</p

    Comparison of no-GVS vs GVS Conditions (paired t-test, df = 9, two-tailed).

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    <p>Comparison of mean (SD) computerized dynamic posturography (CDP) composite equilibrium (EQ) score and vestibular index, and peak-to-peak ocular torsion, for each of the 14 test sessions over 36 weeks.</p><p>Comparison of no-GVS vs GVS Conditions (paired t-test, df = 9, two-tailed).</p

    Computerized dynamic posturography data from all 10 subjects (mean and 95%CI) without (blue trace) and with (red trace) 5 mA GVS.

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    <p>(A) The composite equilibrium score was significantly lower when first exposed to GVS, but recovered to baseline after 7 weeks (70 min total) exposure, and this recovery was retained for up to 6 months post adaptation. (B) The somatosensory index, and (C) the visual index, were unaffected by GVS exposure. (D) In contrast, the vestibular index (SOT5/SOT1 ratio), was significantly affected by GVS exposure, illustrating the almost exclusive impact of GVS on the vestibular component of postural control. The vestibular index recovered to baseline by week 8, following 70 min of cumulative GVS exposure, and this recovery was maintained 6 months post-adaptation.</p

    Torsional (roll) eye position data from all 10 subjects (mean and 95%CI) without (blue trace) and with (red trace) 5 mA GVS.

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    <p>There was a significant increase in ocular torsion during GVS relative to baseline, which was unaffected by repeated GVS exposure.</p
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