Energy spread of ultracold electron bunches extracted from a laser cooled gas

Ultrashort and ultracold electron bunches created by near-threshold femtosecond photoionization of a laser-cooled gas hold great promise for single-shot ultrafast diffraction experiments. In previous publications the transverse beam quality and the bunch length have been determined. Here the longitudinal energy spread of the generated bunches is measured for the first time, using a specially developed Wien filter. The Wien filter has been calibrated by determining the average deflection of the electron bunch as a function of magnetic field. The measured relative energy spread $\frac{\sigma_{U}}{U} = 0.64 \pm 0.09\%$ agrees well with the theoretical model which states that it is governed by the width of the ionization laser and the acceleration length

Additional file 1: of Vascular-directed responses of microglia produced by methamphetamine exposure: indirect evidence that microglia are involved in vascular repair?

Figure S1A. DAB-Iba1 labeled microglia activation in the septum at 3 days after METH exposure. Figure S1B. DAB-Iba1 labeled microglia in the septum and hippocampus at 1day after METH exposure. Figure S2A. DAB-Iba1 labeled microglia in the hippocampus at 3 days after METH exposure. Figure S2B. DAB-Iba1 labeled microglia in the cudate/putamen at 1 day after METH exposure. Figure S3A. Coincidence of degenerating (FJc+) neurons and activated microglia in the thalamus at 3 days after METH. Figure S3B. FJc-labeling in the septum at 3 days after METH exposure. Figure S4. FJc-labeling in the medial hippocampus at 3 days after METH exposure. Figure S9. DAB-GFAP labeled astrocytes in the medial hippocampus at 3 days after METH exposure. (PDF 15680 kb

Additional file 20: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S15. Genes identified by both PePr and diffReps as having differential enrichment of H3K27ac in the frontal cortex of CORT + DFP exposed animals. (CSV 26 kb

Visual deficits in a mouse model of Batten disease are the result of optic nerve degeneration and loss of dorsal lateral geniculate thalamic neurons

Figure S3. Frontal cortex RNA-seq significantly enriched KEGG pathway annotations. KEGG pathways significantly enriched in genes that were differentially expressed in the frontal cortex of CORT + DFP exposed mice. (CSV 6 kb

Additional file 9: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S5. KEGG pathways enriched in the genes identified as uniquely differentially expressed in the hippocampus after CORT + DFP exposure, compared to all other groups. (CSV 103 kb

Additional file 11: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S7. Gene Ontology (GO) Biological Process (BP) annotations found to be enriched in genes significant in the frontal cortex (Additional file 6: Table S3) and enriched in genes significant in the hippocampus (Additional file 10: Table S6). (TIFF 1648 kb

Additional file 15: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S10. Gene Ontology (GO) Biological Process (BP) annotations found to be enriched in genes identified as uniquely differentially expressed in both the frontal cortex (Additional file 3: Table S1) and the hippocampus (Additional file 7: Table S4) after CORT + DFP exposure, compared to all other groups. (CSV 12 kb

Additional file 6: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S3. Gene Ontology (GO) Biological Process (BP) annotations enriched in the genes identified as uniquely differentially expressed in the frontal cortex after CORT + DFP exposure, compared to all other groups. (CSV 38 kb

Additional file 18: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S13. Genes identified by PePr as having differential enrichment of H3K27ac in the frontal cortex of CORT + DFP exposed animals. (CSV 519 bytes

Additional file 16: of Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

Table S11. Genes identified as containing differentially methylated regions in the frontal cortex of combined CORT + DFP exposed animals. (TIFF 3010 kb