6 research outputs found

    Low-sodium diet induces atherogenesis regardless of lowering blood pressure in hypertensive hyperlipidemic mice - Fig 5

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    <p>Histomorphometric analysis of immunofluorescence-stained AT1 receptor (<b>A</b> segments I and II; <b>B</b> segments III and IV), and vascular injury quantified by a histomorphometric analysis of immunofluorescence-stained CML (<b>C</b> segments I and II; <b>D</b> segments III and IV) and RAGE (<b>E</b> segments I and II; <b>F</b> segments III and IV); data are represented as the mean percentage of the total positively stained area of the aortic arch cross-sections; n = 4 mice per group. <sup>a</sup> <i>P</i> < 0.05, hypertensive mice fed a normal-sodium diet (H-NS) <i>vs</i> hypertensive mice fed a low-sodium diet (H-LS), Mann Whitney test. <sup>b</sup> <i>P</i> < 0.05, Kruskal Wallis with Dunn’s post hoc test applied for comparisons among LS groups.</p

    Gene expression (mRNA) of the AT1 receptor (<i>Agtr1</i>) and RAGE (<i>Ager</i>) in the mouse aortic arch.

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    <p>Data are expressed as relative mRNA units normalized to mouse β2M expression. Mann Whitney test was used for comparisons between hypertensive mice fed a normal-sodium (H-NS) diet and hypertensive mice fed a low-sodium (H-LS) diet. The Kruskal Wallis test with Dunn’s post hoc test was applied for comparisons among the LS groups; n ≥ 4 mice per group.</p
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