13 research outputs found
Correlação entre velocidade e agilidade em jovens atletas de futebol do sexo masculino
O futebol Ă© um dos esportes mais populares e praticados ao redor do mundo que durante o jogo exige movimentos de velocidade e mudança de direção, onde promove uma demanda ao atleta. Portanto, o objetivo do presente estudo foi Verificar se há correlação entre velocidade, agilidade e dĂ©ficit de mudança de direção (dĂ©ficit COD) em jovens atletas de futebol do sexo masculino. A amostra foi composta por 12 atletas de futebol, todos do sexo masculino com idades entre 14 a 16 anos, em um clube no municĂpio do Rio de Janeiro. Os atletas foram submetidos aos testes de agilidade (Zig-Zag COD), sprint (Vel20m) e a partir destes dois testes foi calculado o dĂ©ficit COD. Os resultados do teste de correlação de Pearson demonstraram correlação significativa forte (p>0,001; r= 0,81) entre Vel20m e dĂ©ficit COD. Ocorreu correlação negativa moderada (p=0,02; r= -0,68 entre dĂ©ficit COD e zig zag COD. Conclui-se que as variáveis velocidade e agilidade coletadas em teste de campo, ainda durante o perĂodo da Covid 19, nos meses de abril e maio de 2021, possuem correlação entre dĂ©ficit COD e sprint (vel20m)
Açaà (Euterpe oleracea Mart) seed extract protects against maternal vascular dysfunction, hypertension, and fetal growth restriction in experimental preeclampsia
Objective To investigate whether Euterpe oleracea Mart. (açaĂ) seed extract (ASE) prevents maternal cardiovascular changes and intrauterine growth restriction (IUGR) in experimental preeclampsia (PE). Methods ASE administration (200 mg/kg/day) during mid to late pregnancy in a rat model of L-NAME-induced PE. Results ASE impaired the maternal hypertension and microalbuminuria as well as the lower fetal and placental weight in experimental PE. ASE also prevented the maternal vascular dysfunction and lipoperoxidation in this model. Conclusion ASE protected against maternal cardiovascular changes and IUGR in the L-NAME-induced PE. The protective effect of ASE may be partly explained by its antioxidant property
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Mesenteric vascular reactivity.
<p>Effect of treatment with ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on vasodilator effects of ACh (A) and NG (B), and vasoconstrictor effects of NE (C) in mesenteric arterial bed from type 2 diabetic rats. Data are mean ± SEM, n = 10 for all groups. *Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Controls; <sup>+</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Sedentary D.</p
Expression of insulin cascade and pAMPK protein.
<p>Effect of treatment with ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on IR (A), AKT (B), pAKT (C), GLUT-4 (D) and pAMPK expressions in skeletal muscle from type 2 diabetic rats. Data are mean ± SEM, n = 4 for all groups. Using one-way ANOVA: *Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Controls; <sup>●</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Training C; <sup>+</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Sedentary D; <sup>$</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Training D; <sup>δ</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from ASE Sedentary D. Using unpaired Student's t-test: <sup>&</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Sedentary D.</p
Effects of ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on glucagon-like peptide-1 (GLP-1), leptin, and anti-inflammatory cytokine serum levels in type 2 diabetic animals.
<p>Effects of ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on glucagon-like peptide-1 (GLP-1), leptin, and anti-inflammatory cytokine serum levels in type 2 diabetic animals.</p
Insulin resistance and β-cell function.
<p>Effect of treatment with ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on serum insulin levels (A), HOMA-IR (B) and HOMA-B (C) indexes in type 2 diabetic rats. Data are mean ± SEM, n = 6 for all groups. *Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Controls; <sup>+</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Sedentary D. <sup>$</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Training D.</p
Effects of ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on HbA1c serum levels and lipid profile in type 2 diabetic animals.
<p>Effects of ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on HbA1c serum levels and lipid profile in type 2 diabetic animals.</p
Glycemic levels and body weight.
<p>Effect of treatment with ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on blood glucose levels (A) and body weight (B) in type 2 diabetic rats. Data are mean ± SEM, n = 10 for all groups. *Significantly different (p<0.05) from Controls; <sup>+</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Sedentary D; <sup>$</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Training D; <sup>δ</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from ASE Sedentary D.</p
Components of the insulin cascade and adiponectin expression.
<p>Effect of treatment with ASE (200mg/Kg/day) and exercise training (30 min/day; 5 days per week) on IR (A), AKT (B), pAKT (C), GLUT-4 (D) and adiponectin (E) expressions in adipose tissue from type 2 diabetic rats. Data are mean ± SEM, n = 4 for all groups. Using one-way ANOVA: *Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Controls; <sup>+</sup>Significantly different (p<0.05) from Sedentary D; <sup>$</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Training D; <sup>δ</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from ASE Sedentary D. Using unpaired Student's t-test: <sup>&</sup>Significantly different (<i>p ≤ 0</i>.<i>05</i>) from Sedentary D.</p