5 research outputs found
Course of <i>Encepahlitozoon cuniculi</i> genotype II infection, including pattern of spore shedding and dissemination of infection to selected organs and tissues.
<p><b>a)</b> SCID mice, <b>b)</b> SCID mice treated with albendazole, <b>c)</b> BALB/c mice and BALB/c mice re-infected in chronic stage of infection, <b>d)</b> BALB/c mice treated with albendazole and BALB/c mice treated with albendazole with following re-infection, <b>e)</b> BALB/c mice immunosuppressed in chronic stage of infection, <b>f)</b> BALB/c mice immunosuppressed after albendazole treatment. <b>Light-gray field</b> β albendazole treatment; <b>dark-gray field</b> β dexamethasone immunosuppression; <b>black line</b> β course of <i>E. cuniculi</i> infection; <b>black dash line</b> - course of <i>E. cuniculi</i> re-infection; <b>cross</b> β <i>E. cuniculi</i> positive organ during primarily infection; <b>ring</b> β <i>E. cuniculi</i> positive organ during re-infection; <b>black square</b> β spores shedding during primarily infection; <b>black circle</b> β spores shedding during re-infection.</p
Design of experiments.
<p><b><sup>a</sup></b>inoculation with 200 ul sterilized deionised water; <b><sup>b</sup></b>infection with 10<sup>7 </sup><i>E. cuniculi</i> spores in 0.2 ml of sterilized deionised water; INF β infection; REINF β reinfection (black column); IMSUP β dexamethasone immunosuppression (highlighted in dark grey); TREAT β albendazole treatment (highlighted in light grey); <b>n1β</b> number of used animals; <b>n2</b>β number of dissected animals; <b>NS</b> β not shown; <b>x</b> β not observed due to mouse death; <b>DPI</b> β day post infection;</p
Neighbour-joining tree based on nucleotide sequences of the whole ITS region of Enterocytozoon bieneusi isolates, including our new sequences (underlined).
<p>Genotypes previously found in apes and humans are shaded. The host is listed for each sample. Values on branches are percent bootstrapping using 1 000 replicates. The bootstrap proportions greater than 50% are shown on each branch. Nucleotide sequences generated from this study are underlined and are deposited in the GenBank under Accession Nos. JQ837793-JQ837800.</p
<i>Enterocytozoon bieneusi</i>, <i>Encephalitozoon cuniculi</i>, <i>Cryptosporidium</i> spp. and <i>Giardia intestinalis</i> infection in wild western lowland gorillas (<i>Gorilla gorilla gorilla</i>) under different levels of human contact.
<p><b>D</b>β=β <i>E. bieneusi</i> genotype D; <b>gorilla 1</b> β=β <i>E. bieneusi</i> genotype gorilla 1; <b>gorilla 2</b> β=β <i>E. bieneusi</i> genotype gorilla 2; <b>gorilla 3</b> β=β <i>E. bieneusi</i> genotype gorilla 3; <b>EC I</b> β=β <i>E. cuniculi</i> genotype I; <b>EC II</b> β=β <i>E. cuniculi</i> genotype II; <b>A</b> β=β <i>Giardia intestinalis</i> assemblage A; <b>n<sup>1</sup></b> number of samples; <b>n<sup>2</sup></b> number of animals sampled.</p
Enterocytozoon bieneusi, Encephalitozoon cuniculi, Cryptosporidium spp. and Giardia intestinalis infection in humans, wild and domestic animals.
<p><b>n</b> β=β number of samples; <b>EC I</b> β=β <i>E. cuniculi</i> genotype I; <b>EC II</b> β=β <i>E. cuniculi</i> genotype II; <b>E</b> β=β <i>Giardia intestinalis</i> assemblage E; <b>A</b> β=β <i>Giardia intestinalis</i> assemblage A.</p