Measurement of the mass of the τ lepton

A data-driven energy scan in the immediate vicinity of the τ pair production threshold has been performed using the Beijing Spectrometer at the Beijing Electron-Positron Collider. Approximately 5 pb^(-1) of data, distributed over 12 scan points, have been collected. A previous mass value for the τ lepton, obtained using only the eμ final state, has been published. In this paper, the final BES result on the mass measurement is presented. The analysis is based on the combined data from the ee, eμ, eh, μμ, μh, and hh final states, where h denotes a charged π or K. A maximum likelihood fit to the τ pair production cross section data yields the value m_τ=1776.96_(-0.21)-0.17^(+0.18+0.25) MeV

Long-time Behavior of a Two-layer Model of Baroclinic Quasi-geostrophic Turbulence

We study a viscous two-layer quasi-geostrophic beta-plane model that is forced by imposition of a spatially uniform vertical shear in the eastward (zonal) component of the layer flows, or equivalently a spatially uniform north-south temperature gradient. We prove that the model is linearly unstable, but that non-linear solutions are bounded in time by a bound which is independent of the initial data and is determined only by the physical parameters of the model. We further prove, using arguments first presented in the study of the Kuramoto-Sivashinsky equation, the existence of an absorbing ball in appropriate function spaces, and in fact the existence of a compact finite-dimensional attractor, and provide upper bounds for the fractal and Hausdorff dimensions of the attractor. Finally, we show the existence of an inertial manifold for the dynamical system generated by the model's solution operator. Our results provide rigorous justification for observations made by Panetta based on long-time numerical integrations of the model equations

Measurement of the mass of the τ lepton

The mass of the τ lepton has been measured at the Beijing Electron-Positron Collider using the Beijing Spectrometer. A search near threshold for e^+e^-→τ^+τ^- was performed. Candidate events were identified by requiring that one τ decay via τ→eνν¯, and the other via τ→μνν¯. The mass value, obtained from a fit to the energy dependence of the τ^+τ^- cross section, is m_τ=1776.9_(-0.5)^(+0.4)±0.2 MeV

Management Impacts on Ammonia Volatilization from Swine Manure

Ammonia released from swine manure into the air is becoming an increasingly controversial topic in Iowa. This experiment was conducted to evaluate the potential of several management strategies to reduce ammonia volatilization from swine manure over time. In six benchtop trials using twenty-four 1-L manure storage vessels, treatments were applied to the vessels, and manure and air samples were analyzed for concentrations of ammonia and other forms of nitrogen. Segregated storage of urine and feces, keeping manure cool and still, addition of yucca extract, and acidification reduced ammonia volatilization

Direct measurement of B(D_s^+ → φX^+)

The absolute inclusive branching fraction of D_s^+→φX^+ has been measured from data collected by the BES detector at a center-of-mass energy of 4.03 GeV, corresponding to an integrated luminosity of 22.3 pb^(-1). At this energy, direct pair production e^+e^-→D_s^+D_s^- has been observed. We have selected D_s candidate events by reconstructing five hadronic decay modes D_s^+→φπ^+, K^(0*)K^+, K^0K^+, f0^(π+) and K^0K^-π^+π^+ and have searched for inclusive φ’s in the recoiling D_s^-. We observed three recoiling φ’s in the 166.4 ± 31.8 D_s candidate events, which leads to the absolute branching fraction B(D_s^+→φX^+)=(17.8(-7.2 -6.3)^(+15.1+0.6)) % and B(D_s-6.3+→φπ-6.3+)=(3.6_(-1.6 -1.3)(^_3.1+0.4) %. [S0556-2821(97)02423-5

Nitrogen Excretion and Ammonia Emissions from Pigs Fed Reduced Crude Protein Diets or Yucca Extract

Dietary strategies are an important part of management systems designed to reduce ammonia emitted from animal feeding operations. Two potential strategies (reducing dietary crude protein content and adding yucca extract to the diet) were evaluated in two swine feeding trials. Three groups of pigs were housed in environmental chambers in order to allow for direct measurement of ammonia emissions. Emission rates and excreta nitrogen and ammonium concentrations were analyzed. Feeding a diet with four amino acids resulted in large though nonsignificant reductions in ammonia emissions, and affected excreta nitrogen concentrations; increasing urinary ammonium-N while decreasing urinary and fecal total nitrogen and fecal ammonium-N. The only significant effect of dietary yucca was a decrease in urinary ammonium-N concentration. In both studies, ammonia emission rates increased over time as manure accumulated in the chamber pits

Apoptosome activation, an important molecular instigator in 6-mercaptopurine induced Leydig cell death.

Leydig cells are crucial to the production of testosterone in males. It is unknown if the cancer chemotherapeutic drug, 6-mercaptopurine (6 MP), produces Leydig cell failure among adult survivors of childhood acute lymphoblastic leukemia. Moreover, it is not known whether Leydig cell failure is due to either a loss of cells or an impairment in their function. Herein, we show, in a subset of childhood cancer survivors, that Leydig cell failure is related to the dose of 6 MP. This was extended, in a murine model, to demonstrate that 6 MP exposure induced caspase 3 activation, and the loss of Leydig cells was independent of Bak and Bax activation. The death of these non-proliferating cells was triggered by 6 MP metabolism, requiring formation of both cytosolic reactive oxygen species and thiopurine nucleotide triphosphates. The thiopurine nucleotide triphosphates (with physiological amounts of dATP) uniquely activated the apoptosome. An ABC transporter (Abcc4/Mrp4) reduced the amount of thiopurines, thereby providing protection for Leydig cells. The studies reported here demonstrate that the apoptosome is uniquely activated by thiopurine nucleotides and suggest that 6 MP induced Leydig cell death is likely a cause of Leydig cell failure in some survivors of childhood cancer