Indocencia

Treballs de l'alumnat del Grau de Comunicació Audiovisual, Facultat de Biblioteconomia i Documentació, Universitat de Barcelona, Projectes II - Grup8. Curs: 2014-2015, Tutora: Sílvia PairóDirector: Gerard Adell; Productor: Catalina Vives; Realització: Laura Ramiro; Càmera 1: Álvaro Gómez; Càmera 2: Rosa Martínez; Muntatge: Andrea Díaz; Entrevistadora: Marta Hernàndez; Postproducció: Andrea Díaz; Personatge: Professor Associat, Actor: Víctor Gómez; Entrevistats: Ana Medina, Sergio Villanueva, David Carpio, Antonio Madrid, Mónica Aranda, Mercè Amich i Jaume Sastre.[Video] Un documental sobre la mercantilizació de la Universitat de Barceloana a través de la figura del professor fals associat[Memòria] Indocencia és un documental que estudia i mostra la mercantilització de la Universitat de Barcelona a través de la figura del professor fals associat. Amb aquest objectiu, s'analitza la precarietat laboral que l'afecta així com la repercussió que suposa per la universitat l'augment del nombre de professors associats. Es mostra la història d'aquesta figura parlant dels possibles camins que porten a un docent a ser-ho, i els motius que aquest té. A més, es compta amb el testimoni de docents que es troben en aquesta situació i que poden parlar-nos en primera persona del que suposa treballar com a fals associat

Neurofeedback-Based Enhancement of Single-Trial Auditory Evoked Potentials: Treatment of Auditory Verbal Hallucinations in Schizophrenia.

Auditory verbal hallucinations depend on a broad neurobiological network ranging from the auditory system to language as well as memory-related processes. As part of this, the auditory N100 event-related potential (ERP) component is attenuated in patients with schizophrenia, with stronger attenuation occurring during auditory verbal hallucinations. Changes in the N100 component assumingly reflect disturbed responsiveness of the auditory system toward external stimuli in schizophrenia. With this premise, we investigated the therapeutic utility of neurofeedback training to modulate the auditory-evoked N100 component in patients with schizophrenia and associated auditory verbal hallucinations. Ten patients completed electroencephalography neurofeedback training for modulation of N100 (treatment condition) or another unrelated component, P200 (control condition). On a behavioral level, only the control group showed a tendency for symptom improvement in the Positive and Negative Syndrome Scale total score in a pre-/postcomparison ( t= 2.71, P = .054); however, no significant differences were found in specific hallucination related symptoms ( t= -0.53, P = .62). There was no significant overall effect of neurofeedback training on ERP components in our paradigm; however, we were able to identify different learning patterns, and found a correlation between learning and improvement in auditory verbal hallucination symptoms across training sessions ( r = 0.664, n = 9, P = .05). This effect results, with cautious interpretation due to the small sample size, primarily from the treatment group ( r = 0.97, n = 4, P = .03). In particular, a within-session learning parameter showed utility for predicting symptom improvement with neurofeedback training. In conclusion, patients with schizophrenia and associated auditory verbal hallucinations who exhibit a learning pattern more characterized by within-session aptitude may benefit from electroencephalography neurofeedback. Furthermore, independent of the training group, a significant spatial pre-post difference was found in the event-related component P200 ( P = .04)

Neurofeedback-Based Enhancement of Single Trial Auditory Evoked Potentials: Feasibility in Healthy Subjects.

Previous studies showed a global reduction of the event-related potential component N100 in patients with schizophrenia, a phenomenon that is even more pronounced during auditory verbal hallucinations. This reduction assumingly results from dysfunctional activation of the primary auditory cortex by inner speech, which reduces its responsiveness to external stimuli. With this study, we tested the feasibility of enhancing the responsiveness of the primary auditory cortex to external stimuli with an upregulation of the event-related potential component N100 in healthy control subjects. A total of 15 healthy subjects performed 8 double-sessions of EEG-neurofeedback training over 2 weeks. The results of the used linear mixed effect model showed a significant active learning effect within sessions ( t = 5.99, P < .001) against an unspecific habituation effect that lowered the N100 amplitude over time. Across sessions, a significant increase in the passive condition ( t = 2.42, P = .03), named as carry-over effect, was observed. Given that the carry-over effect is one of the ultimate aims of neurofeedback, it seems reasonable to apply this neurofeedback training protocol to influence the N100 amplitude in patients with schizophrenia. This intervention could provide an alternative treatment option for auditory verbal hallucinations in these patients