711 research outputs found

    Population of bound excited states in intermediate-energy fragmentation reactions

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    Fragmentation reactions with intermediate-energy heavy-ion beams exhibit a wide range of reaction mechanisms, ranging from direct reactions to statistical processes. We examine this transition by measuring the relative population of excited states in several sd-shell nuclei produced by fragmentation with the number of removed nucleons ranging from two to sixteen. The two-nucleon removal is consistent with a non-dissipative process whereas the removal of more than five nucleons appears to be mainly statistical.Comment: 5 pages, 6 figure

    Shell structure at N=28 near the dripline: spectroscopy of 42^{42}Si, 43^{43}P and 44^{44}S

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    Measurements of the N=28 isotones 42Si, 43P and 44S using one- and two-proton knockout reactions from the radioactive beam nuclei 44S and 46Ar are reported. The knockout reaction cross sections for populating 42Si and 43P and a 184 keV gamma-ray observed in 43P establish that the d_{3/2} and s_{1/2} proton orbits are nearly degenerate in these nuclei and that there is a substantial Z=14 subshell closure separating these two orbits from the d_{5/2} orbit. The increase in the inclusive two-proton knockout cross section from 42Si to 44S demonstrates the importance of the availability of valence protons for determining the cross section. New calculations of the two-proton knockout reactions that include diffractive effects are presented. In addition, it is proposed that a search for the d_{5/2} proton strength in 43P via a higher statistics one-proton knockout experiment could help determine the size of the Z=14 closure.Comment: Phys. Rev. C, in pres

    Spectroscopy of 54^{54}Ti and the systematic behavior of low energy octupole states in Ca and Ti isotopes

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    Excited states of the N=32N=32 nucleus 54^{54}Ti have been studied, via both inverse-kinematics proton scattering and one-neutron knockout from 55^{55}Ti by a liquid hydrogen target, using the GRETINA Îł\gamma-ray tracking array. Inelastic proton-scattering cross sections and deformation lengths have been determined. A low-lying octupole state has been tentatively identified in 54^{54}Ti for the first time. A comparison of (p,pâ€Č)(p,p') results on low-energy octupole states in the neutron-rich Ca and Ti isotopes with the results of Random Phase Approximation calculations demonstrates that the observed systematic behavior of these states is unexpected.Comment: 7 pages, 8 figure

    Spectroscopy of 54^{54}Ti and the systematic behavior of low energy octupole states in Ca and Ti isotopes

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    Excited states of the N=32N=32 nucleus 54^{54}Ti have been studied, via both inverse-kinematics proton scattering and one-neutron knockout from 55^{55}Ti by a liquid hydrogen target, using the GRETINA Îł\gamma-ray tracking array. Inelastic proton-scattering cross sections and deformation lengths have been determined. A low-lying octupole state has been tentatively identified in 54^{54}Ti for the first time. A comparison of (p,pâ€Č)(p,p') results on low-energy octupole states in the neutron-rich Ca and Ti isotopes with the results of Random Phase Approximation calculations demonstrates that the observed systematic behavior of these states is unexpected.Comment: 7 pages, 8 figure

    Extreme alpha-clustering in the 18O nucleus

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    The structure of the 18O nucleus at excitation energies above the alpha decay threshold was studied using 14C+alpha resonance elastic scattering. A number of states with large alpha reduced widths have been observed, indicating that the alpha-cluster degree of freedom plays an important role in this N not equal Z nucleus. However, the alpha-cluster structure of this nucleus is very different from the relatively simple pattern of strong alpha-cluster quasi-rotational bands in the neighboring 16O and 20Ne nuclei. A 0+ state with an alpha reduced width exceeding the single particle limit was identified at an excitation energy of 9.9+/-0.3 MeV. We discuss evidence that states of this kind are common in light nuclei and give possible explanations of this feature.Comment: 4 pages, 2 figures, 1 table. Resubmission with minor changes for clarity, including removal of one figur

    Two-neutron knockout from neutron-deficient 34^{34}Ar, 30^{30}S, and 26^{26}Si

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    Two-neutron knockout reactions from nuclei in the proximity of the proton dripline have been studied using intermediate-energy beams of neutron-deficient 34^{34}Ar, 30^{30}S, and 26^{26}Si. The inclusive cross sections, and also the partial cross sections for the population of individual bound final states of the 32^{32}Ar, 28^{28}S and 24^{24}Si knockout residues, have been determined using the combination of particle and Îł\gamma-ray spectroscopy. Similar to the two-proton knockout mechanism on the neutron-rich side of the nuclear chart, these two-neutron removal reactions from already neutron-deficient nuclei are also shown to be consistent with a direct reaction mechanism.Comment: Phys. Rev. C, rapid communication, in pres

    Aged-senescent cells contribute to impaired heart regeneration

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    Aging leads to increased cellular senescence and is associated with decreased potency of tissue-specific stem/progenitor cells. Here, we have done an extensive analysis of cardiac progenitor cells (CPCs) isolated from human subjects with cardiovascular disease, aged 32-86 years. In aged subjects (>70 years old), over half of CPCs are senescent (p16INK4A , SA-ÎČ-gal, DNA damage ÎłH2AX, telomere length, senescence-associated secretory phenotype [SASP]), unable to replicate, differentiate, regenerate or restore cardiac function following transplantation into the infarcted heart. SASP factors secreted by senescent CPCs renders otherwise healthy CPCs to senescence. Elimination of senescent CPCs using senolytics abrogates the SASP and its debilitative effect in vitro. Global elimination of senescent cells in aged mice (INK-ATTAC or wild-type mice treated with D + Q senolytics) in vivo activates resident CPCs and increased the number of small Ki67-, EdU-positive cardiomyocytes. Therapeutic approaches that eliminate senescent cells may alleviate cardiac deterioration with aging and restore the regenerative capacity of the heart
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