Emergence of Gapped Bulk and Metallic Side Walls in the Zeroth Landau level in Dirac and Weyl semimetals

Recent transport experiments have revealed the activation of longitudinal magnetoresistance of Weyl semimetals in the quantum limit, suggesting the breakdown of chiral anomaly in a strong magnetic field. Here we provide a general mechanism for gapping the zeroth chiral Landau levels applicable for both Dirac and Weyl semimetals. Our result shows that the zeroth Landau levels anticross when the magnetic axis is perpendicular to the Dirac/Weyl node separation and when the inverse magnetic length $l_B^{-1}$ is comparable to the node separation scale $\Delta k$. The induced bulk gap increases rapidly beyond a threshold field in Weyl semimetals, but has no threshold and is non-monotonic in Dirac systems due to the crossover between $l_B^{-1}>\Delta k$ and $l_B^{-1}<\Delta k$ regions. We also find that the Dirac and possibly Weyl systems host counterpropagating edge states between the zeroth Landau levels, leading to a state with metallic side walls and zero Hall conductance.Comment: 8 pages, 4 figure

Beyond the Individual in Controversial Science-Based Technology Attitude Formation and Regulation: The State Construction of Policy Alternatives in Asia

10.2139/ssrn.17175151-3

When chiral photons meet chiral fermions - Photoinduced anomalous Hall effects in Weyl semimetals

The Weyl semimetal is characterized by three-dimensional linear band touching points called Weyl nodes. These nodes come in pairs with opposite chiralities. We show that the coupling of circularly polarized photons with these chiral electrons generates a Hall conductivity without any applied magnetic field in the plane orthogonal to the light propagation. This phenomenon comes about because with all three Pauli matrices exhausted to form the three-dimensional linear dispersion, the Weyl nodes cannot be gapped. Rather, the net influence of chiral photons is to shift the positions of the Weyl nodes. Interestingly, the momentum shift is tightly correlated with the chirality of the node to produce a net anomalous Hall signal. Application of our proposal to the recently discovered TaAs family of Weyl semimetals leads to an order-of-magnitude estimate of the photoinduced Hall conductivity which is within the experimentally accessible range.Comment: 9 pages, 4 figure

Incidence of Pneumocystis pneumonia in immunocompromised patients without human immunodeficiency virus on intravenous pentamidine prophylaxis: A systematic review and meta-analysis

BACKGROUND: Trimethoprim-sulfamethoxazole (TMP-SMX) is a first-line METHOD: We performed a systematic review and meta-analysis to estimate breakthrough PCP incidence and adverse reactions in HIV-uninfected immunocompromised patients receiving IVP. MEDLINE, Embase, Web of Science, Cochrane Library, and ClinicalTrials.gov were searched from their inception until 15 December 2022. RESULTS: The pooled incidence of breakthrough PCP with IVP was 0.7% (95% CI, 0.3-1.4%, 16 studies, 3025 patients) and was similar when used as first-line prophylaxis (0.5%; 95% CI, 0.2-1.4%, 7 studies, 752 patients). The pooled incidence of adverse reactions was 11.3% (95% CI, 6.7-18.6%, 14 studies, 2068 patients). The pooled adverse event-related discontinuation was 3.7% (95% CI, 1.8-7.3%, 11 studies, 1802 patients), but was lower in patients receiving IVP monthly (2.0%; 95% CI 0.7-5.7%, 7 studies, 1182 patients). CONCLUSION: Monthly IVP is an appropriate second-line agent for PCP prophylaxis in certain non-HIV immunocompromised hosts, especially in patients with hematologic malignancies and hematopoietic stem cell transplant recipients. Using IVP for PCP prophylaxis as an alternative to oral TMP-SMX while patients are unable to tolerate enteral medication administration is feasible

Distributed Algorithms for Secure Multipath Routing in Attack-Resistant Networks

To proactively defend against intruders from readily jeopardizing single-path data sessions, we propose a distributed secure multipath solution to route data across multiple paths so that intruders require much more resources to mount successful attacks. Our work exhibits several important properties that include: (1) routing decisions are made locally by network nodes without the centralized information of the entire network topology, (2) routing decisions minimize throughput loss under a single-link attack with respect to different session models, and (3) routing decisions address multiple link attacks via lexicographic optimization. We devise two algorithms termed the Bound-Control algorithm and the Lex-Control algorithm, both of which provide provably optimal solutions. Experiments show that the Bound-Control algorithm is more effective to prevent the worst-case single-link attack when compared to the single-path approach, and that the Lex-Control algorithm further enhances the Bound-Control algorithm by countering severe single-link attacks and various types of multi-link attacks. Moreover, the Lex-Control algorithm offers prominent protection after only a few execution rounds, implying that we can sacrifice minimal routing protection for significantly improved algorithm performance. Finally, we examine the applicability of our proposed algorithms in a specialized defensive network architecture called the attack-resistant network and analyze how the algorithms address resiliency and security in different network settings

Human parathyroid hormone fragment stimulates the de novo synthesis of prostaglandin endoperoxide synthase in chick calvaria

The human parathyroid hormone N-terminal fragment [hPTH-(1–34)] increases the conversion of exogenous unsaturated fatty acids to prostaglandins (PGs) in calvarial homogenates. Enzyme activities were completely blocked by indomethacin (5 × 10−7 M), a PG synthase inhibitor, and actinomycin D (5 μM), an inhibitor of transcription, by binding to DNA. In addition, a potent inhibitor of protein synthesis, cycloheximide (10 μM), totally inhibited the stimulating effect of hPTH-(1–34) on prostaglandin endoperoxide synthase (PG synthase, EC 1.14.99.1). The stimulatory effect of hPTH-(1–34) on PG synthase was also reduced by the addition of stannous chloride. However, epidermal growth factor (EGF), platelet-derived activating factor (PDGF), and ionophore A23187 did not show the same stimulating effect as hPTH-(1–34) on PG synthase in calvaria. The results further demonstrated that PG synthase is a membrane-bound enzyme in chick calvaria. In this communication, evidence is presented that hPTH-(1–34) stimulates the de novo synthesis of PG synthase as demonstrated by the increased activity in calvarial homogenates and microsomes

Photocurrents in Weyl semimetals

The generation of photocurrent in an ideal two-dimensional Dirac spectrum is symmetry forbidden. In sharp contrast, we show that three-dimensional Weyl semimetals can generically support significant photocurrents due to the combination of inversion symmetry breaking and finite tilts of the Weyl spectra. Symmetry properties, chirality relations, and various dependencies of this photovoltaic effect on the system and the light source are explored in detail. Our results suggest that noncentrosymmetric Weyl materials can be advantageously applied to room temperature detections of mid- and far-infrared radiations.United States. Department of Energy (DE-FG02-03-ER46076

Soluble Host Defense Lectins in Innate Immunity to Influenza Virus

Host defenses against viral infections depend on a complex interplay of innate (nonspecific) and adaptive (specific) components. In the early stages of infection, innate mechanisms represent the main line of host defense, acting to limit the spread of virus in host tissues prior to the induction of the adaptive immune response. Serum and lung fluids contain a range of lectins capable of recognizing and destroying influenza A viruses (IAV). Herein, we review the mechanisms by which soluble endogenous lectins mediate anti-IAV activity, including their role in modulating IAV-induced inflammation and disease and their potential as prophylactic and/or therapeutic treatments during severe IAV-induced disease