An ultraviolet excess in the superluminous supernova Gaia16apd reveals a powerful central engine

Since the discovery of superluminous supernovae (SLSNe) in the last decade, it has been known that these events exhibit bluer spectral energy distributions than other supernova subtypes, with significant output in the ultraviolet. However, the event Gaia16apd seems to outshine even the other SLSNe at rest-frame wavelengths below $\sim 3000$ \AA. Yan et al (2016) have recently presented HST UV spectra and attributed the UV flux to low metallicity and hence reduced line blanketing. Here we present UV and optical light curves over a longer baseline in time, revealing a rapid decline at UV wavelengths despite a typical optical evolution. Combining the published UV spectra with our own optical data, we demonstrate that Gaia16apd has a much hotter continuum than virtually any SLSN at maximum light, but it cools rapidly thereafter and is indistinguishable from the others by $\sim 10$-15 days after peak. Comparing the equivalent widths of UV absorption lines with those of other events, we show that the excess UV continuum is a result of a more powerful central power source, rather than a lack of UV absorption relative to other SLSNe or an additional component from interaction with the surrounding medium. These findings strongly support the central-engine hypothesis for hydrogen-poor SLSNe. An explosion ejecting $M_{\rm ej} = 4 (0.2/\kappa)$ M$_\odot$, where $\kappa$ is the opacity in cm$^2$g$^{-1}$, and forming a magnetar with spin period $P=2$ ms, and $B=2\times10^{14}$ G (lower than other SLSNe with comparable rise-times) can consistently explain the light curve evolution and high temperature at peak. The host metallicity, $Z=0.18$ Z$_\odot$, is comparable to other SLSNe.Comment: Updated to match accepted version (ApJL

BVRI Light Curves for 22 Type Ia Supernovae

We present 1210 Johnson/Cousins B,V,R, and I photometric observations of 22 recent type Ia supernovae (SNe Ia): SN 1993ac, SN 1993ae, SN 1994M, SN 1994S, SN 1994T, SN 1994Q, SN 1994ae, SN 1995D, SN 1995E, SN 1995al, SN 1995ac, SN 1995ak, SN 1995bd, SN 1996C, SN 1996X, SN 1996Z, SN 1996ab, SN 1996ai, SN 1996bk, SN 1996bl, SN 1996bo, and SN 1996bv. Most of the photometry was obtained at the Fred Lawrence Whipple Observatory (FLWO) of the Harvard-Smithsonian Center for Astrophysics in a cooperative observing plan aimed at improving the data base for SN Ia. The redshifts of the sample range from $cz$=1200 to 37000 km s$^{-1}$ with a mean of $cz$=7000 km s$^{-1}$.Comment: Accepted to the Astronomical Journal, 41 pages, 8 figure

Time Dilation from Spectral Feature Age Measurements of Type Ia Supernovae

We have developed a quantitative, empirical method for estimating the age of Type Ia supernovae (SNe Ia) from a single spectral epoch. The technique examines the goodness of fit of spectral features as a function of the temporal evolution of a large database of SNe Ia spectral features. When a SN Ia spectrum with good signal-to-noise ratio over the rest frame range 3800 to 6800 A is available, the precision of a spectral feature age (SFA) is (1-sigma) ~ 1.4 days. SFA estimates are made for two spectral epochs of SN 1996bj (z=0.574) to measure the rate of aging at high redshift. In the 10.05 days which elapsed between spectral observations, SN 1996bj aged 3.35 $\pm$ 3.2 days, consistent with the 6.38 days of aging expected in an expanding Universe and inconsistent with no time dilation at the 96.4 % confidence level. The precision to which individual features constrain the supernova age has implications for the source of inhomogeneities among SNe Ia.Comment: 14 pages (LaTex), 7 postscript figures to Appear in the Astronomical Journa

The Luminosity of SN 1999by in NGC 2841 and the Nature of `Peculiar' Type Ia Supernovae

We present UBVRIJHK photometry and optical spectroscopy of the so-called 'peculiar' Type Ia supernova 1999by in NGC 2841. The observations began one week before visual maximum light which is well-defined by daily observations. The light curves and spectra are similar to those of the prototypical subluminous event SN 1991bg. We find that maximum light in B occurred on 1999 May 10.3 UT (JD 2,451,308.8 +/- 0.3) with B=13.66 +/- 0.02 mag and a color of B_max-V_max=0.51 +/- 0.03 mag. The late-time color implies minimal dust extinction from the host galaxy. Our photometry, when combined with the recent Cepheid distance to NGC 2841 (Macri et al. 2001), gives a peak absolute magnitude of M_B=-17.15 +/- 0.23 mag, making SN 1999by one of the least luminous Type Ia events ever observed. We estimate a decline rate parameter of dm15(B)=1.90 mag, versus 1.93 for SN 1991bg, where 1.10 is typical for so-called 'normal' events. We compare SN 1999by with other subluminous events and find that the B_max-V_max color correlates strongly with the decline rate and may be a more sensitive indicator of luminosity than the fading rate for these objects. We find a good correlation between luminosity and the depth of the spectral feature at 580 nm, which had been attributed solely to Si II. We show that in cooler photospheres the 580 nm feature is dominated by Ti II, which provides a simple physical explanation for the correlation. Using only subluminous Type Ia supernovae we derive a Hubble parameter of H_0=75 +12 -11 km/s Mpc, consistent with values found from brighter events.Comment: 36 preprint pages including 18 figures. Near-IR photometry of the SN has been added to the paper. Scheduled to appear in ApJ vol. 613 (September 2004). High-resolution version available from http://www.nd.edu/~pgarnavi/sn99by/sn99by.p

Loss of TOP3B leads to increased R-loop formation and genome instability

Topoisomerase III beta (TOP3B) is one of the least understood members of the topoisomerase family of proteins and remains enigmatic. Our recent data shed light on the function and relevance of TOP3B to disease. A homozygous deletion for the TOP3B gene was identified in a patient with bilateral renal cancer. Analyses in both patient and modelled human cells show the disruption of TOP3B causes genome instability with a rise in DNA damage and chromosome bridging (mis-segregation). The primary molecular defect underlying this pathology is a significant increase in R-loop formation. Our data show that TOP3B is necessary to prevent the accumulation of excessive R-loops and identify TOP3B as a putative cancer gene, and support recent data showing that R-loops are involved in cancer aetiology