Multipopulation aftereffects on the color-magnitude diagram and Cepheid variables of young stellar systems

Context: The evidence of a multipopulation scenario in Galactic globular clusters raises several questions about the formation and evolution of the two (or more) generations of stars. These populations show differences in their age and chemical composition. These differences are found in old- and intermediate- age stellar clusters in the Local Group. The observations of young stellar systems are expected to present footprints of multiple stellar populations. Aims: This theoretical work intends to be a specific step in exploring the space of the observational indicators of multipopulations, without covering all the combinations of parameters that may contribute to the formation of multiple generations of stars in a cluster or in galaxy. The goal is to shed light on the possible observational features expected by core He-burning stars that belong to two stellar populations with different original He content and ages. Methods: The tool adopted was the stellar population synthesis. We used new stellar and pulsation models to construct a homogeneous and consistent framework. Synthetic color-magnitude diagrams (CMDs) of young- and intermediate-age stellar systems (from 20 Myr up to 1 Gyr) were computed in several photometric bands to derive possible indicators of double populations both in the observed CMDs and in the pulsation properties of the Cepheids. Results: We predict that the morphology of the red/blue clump in VIK bands can be used to photometrically indicate the two stellar populations in a rich assembly of stars if there is a significant difference in their original He content. Moreover, the period distribution of the Cepheids appears to be widely affected by the coeval multiple generations of stars within stellar systems. We show that the Wesenheit relations may be affected by the helium content of the Cepheids.Comment: in press on A&

Photometric and spectroscopic study of the intermediate-age open cluster NGC 2355

In this paper we analyse the evolutionary status and properties of the old open cluster NGC 2355, located in the Galactic anticentre direction, as a part of the long term programme BOCCE. NGC 2355 was observed with LBC@LBT using the Bessel $B$, $V$, and $I_c$ filters. The cluster parameters have been obtained using the synthetic colour-magnitude diagram (CMD) method, as done in other papers of this series. Additional spectroscopic observations with FIES@NOT of three giant stars were used to determine the chemical properties of the cluster. Our analysis shows that NGC 2355 has metallicity slightly less than solar, with [Fe/H]$=-0.06$ dex, age between 0.8 and 1 Gyr, reddening $E(B-V)$ in the range 0.14 and 0.19 mag, and distance modulus $(m-M)_0$ of about 11 mag. We also investigated the abundances of O, Na, Al, $\alpha$, iron-peak, and neutron capture elements, showing that NGC 2355 falls within the abundance distribution of similar clusters (same age and metallicity). The Galactocentric distance of NGC~2355 places it at the border between two regimes of metallicity distribution; this makes it an important cluster for the study of the chemical properties and evolution of the disc.Comment: 20 pages, 11 figures, Accepted on MNRA

Avaliação de genótipos de cebolas em sistema de produção de base ecológica.

Resumos do VIII Congresso Brasileiro de Agroecologia - Porto Alegre/RS - 25 a 28/11/2013

Emergence of a confined state in a weakly bent wire

In this paper we use a simple straightforward technique to investigate the emergence of a bound state in a weakly bent wire. We show that the bend behaves like an infinitely shallow potential well, and in the limit of small bending angle and low energy the bend can be presented by a simple 1D delta function potential.Comment: 4 pages, 3 Postscript figures (uses Revtex); added references and rewritte

Ethanol potentiates hypoxic liver injury: role of hepatocyte Na+ overload

AbstractCentrilobular hypoxia has been suggested to contribute to hepatic damage caused by alcohol intoxication. However, the mechanisms involved are still poorly understood. We have investigated whether alterations of Na+ homeostasis might account for ethanol-mediated increase in hepatocyte sensitivity to hypoxia. Addition of ethanol (100 mmol/l) to isolated rat hepatocytes incubated under nitrogen atmosphere greatly stimulated cell death. An increase in intracellular Na+ levels preceded cell killing and Na+ levels in hepatocytes exposed to the combination of ethanol and hypoxia were almost twice those in hypoxic cells without ethanol. Na+ increase was also observed in hepatocytes incubated with ethanol in oxygenated buffer. Ethanol addition significantly lowered hepatocyte pH. Inhibiting ethanol and acetaldehyde oxidation with, respectively, 4-methylpyrazole and cyanamide prevented this effect. 4-methylpyrazole, cyanamide as well as hepatocyte incubation in a HCO3−-free buffer or in the presence of Na+/H+ exchanger blocker 5-(N,N-dimethyl)-amiloride also reduced Na+ influx in ethanol-treated hepatocytes. 4-methylpyrazole and cyanamide similarly prevented ethanol-stimulated Na+ accumulation and hepatocyte killing during hypoxia. Moreover, ethanol-induced Na+ influx caused cytotoxicity in hepatocytes pre-treated with Na+,K+-ATPase inhibitor ouabain. Also in this condition 4-methylpyrazole and 5-(N,N-dimethyl)-amiloride decreased cell killing. These results indicate that ethanol can promotes cytotoxicity in hypoxic hepatocytes by enhancing Na+ accumulation