Equilibrium vortex formation in ultrarapidly rotating two-component Bose-Einstein condensates

Equilibrium vortex formation in rotating binary Bose gases with a rotating frequency higher than the harmonic trapping frequency is investigated theoretically. We consider the system being evaporatively cooled to form condensates and a combined numerical scheme is applied to ensure the binary system being in an authentic equilibrium state. To keep the system stable against the large centrifugal force of ultrafast rotation, a quartic trapping potential is added to the existing harmonic part. Using the Thomas-Fermi approximation, a critical rotating frequency \Omega_c is derived, which characterizes the structure with or without a central density hole. Vortex structures are studied in detail with rotation frequency both above and below ?\Omega_c and with respect to the miscible, symmetrically separated, and asymmetrically separated phases in their nonrotating ground-state counterparts.Comment: 7 pages, 7 figure

Spontaneous Crystallization of Skyrmions and Fractional Vortices in the Fast-rotating and Rapidly-quenched Spin-1 Bose-Einstein Condensates

We investigate the spontaneous generation of crystallized topological defects via the combining effects of fast rotation and rapid thermal quench on the spin-1 Bose-Einstein condensates. By solving the stochastic projected Gross-Pitaevskii equation, we show that, when the system reaches equilibrium, a hexagonal lattice of skyrmions, and a square lattice of half-quantized vortices can be formed in a ferromagnetic and antiferromagnetic spinor BEC, respetively, which can be imaged by using the polarization-dependent phase-contrast method

Generating Abstractive Summaries from Meeting Transcripts

Summaries of meetings are very important as they convey the essential content of discussions in a concise form. Generally, it is time consuming to read and understand the whole documents. Therefore, summaries play an important role as the readers are interested in only the important context of discussions. In this work, we address the task of meeting document summarization. Automatic summarization systems on meeting conversations developed so far have been primarily extractive, resulting in unacceptable summaries that are hard to read. The extracted utterances contain disfluencies that affect the quality of the extractive summaries. To make summaries much more readable, we propose an approach to generating abstractive summaries by fusing important content from several utterances. We first separate meeting transcripts into various topic segments, and then identify the important utterances in each segment using a supervised learning approach. The important utterances are then combined together to generate a one-sentence summary. In the text generation step, the dependency parses of the utterances in each segment are combined together to create a directed graph. The most informative and well-formed sub-graph obtained by integer linear programming (ILP) is selected to generate a one-sentence summary for each topic segment. The ILP formulation reduces disfluencies by leveraging grammatical relations that are more prominent in non-conversational style of text, and therefore generates summaries that is comparable to human-written abstractive summaries. Experimental results show that our method can generate more informative summaries than the baselines. In addition, readability assessments by human judges as well as log-likelihood estimates obtained from the dependency parser show that our generated summaries are significantly readable and well-formed.Comment: 10 pages, Proceedings of the 2015 ACM Symposium on Document Engineering, DocEng' 201

Piezoelectric control of the magnetic anisotropy via interface strain coupling in a composite multiferroic structure

We investigate theoretically the magnetic dynamics in a ferroelectric/ferromagnetic heterostructure coupled via strain-mediated magnetoelectric interaction. We predict an electric field-induced magnetic switching in the plane perpendicular to the magneto-crystalline easy axis, and trace this effect back to the piezoelectric control of the magnetoelastic coupling. We also investigate the magnetic remanence and the electric coercivity

THE SURFACE EMG ACTIVITY OF THE UPPER LIMB MUSCLES IN TABLE TENNIS FOREHAND DRIVES

The purpose of this study was to analyze the 3D kinematics variables and the upper limb muscle surface EMG activity of Taiwan elite table tennis players when they were performing forehand drives after receiving topspin and backspin services. Ten Vicon MX-13 2+cameras (Vicon, Oxford, UK, 250Hz) were used to record the 3D kinematics data and measured the EMG signals of seven upper limb muscles of the players. The results showed that the tactics of the table tennis players performed the forehand drive to receive backspin were both to increase the racket tilt angle in advance and to raise the path angle during the upswing phase. The players exerted greater muscular activity during receiving the backspin forehand drive than receiving topspin forehand drive in the wrist extensor, the biceps and the triceps

Melioidosis: an emerging infection in Taiwan?

From January 1982 to May 2000, 17 infections caused by Burkholderia pseudomallei were diagnosed in 15 patients in Taiwan; almost all the infections were diagnosed from 1994 to May 2000. Of the 15 patients, 9 (60%) had underlying diseases, and 10 (67%) had bacteremic pneumonia. Thirteen (76%) episodes of infection were considered indigenous. Four patients died of melioidosis. Seventeen B. pseudomallei isolates, recovered from eight patients from November 1996 to May 2000, were analyzed to determine their in vitro susceptibilities to 14 antimicrobial agents, cellular fatty acid and biochemical reaction profiles, and randomly amplified polymorphic DNA patterns. Eight strains (highly related isolates) were identified. All isolates were arabinose non-assimilators and were susceptible to amoxicillin-clavulanate, piperacillin-tazobactam, imipenem, and meropenem. No spread of the strain was documented

Nucleon Decay Matrix Elements from Lattice QCD

We present a model-independent calculation of hadron matrix elements for all dimension-six operators associated with baryon number violating processes using lattice QCD. The calculation is performed with the Wilson quark action in the quenched approximation at $\beta=6/g^2=6.0$ on a $28^2\times 48\times 80$ lattice. Our results cover all the matrix elements required to estimate the partial lifetimes of (proton,neutron)$\to$($\pi,K,\eta$) +(${\bar \nu},e^+,\mu^+$) decay modes. We point out the necessity of disentangling two form factors that contribute to the matrix element; previous calculations did not make the separation, which led to an underestimate of the physical matrix elements. With a correct separation, we find that the matrix elements have values 3-5 times larger than the smallest estimates employed in phenomenological analyses of the nucleon decays, which could give strong constraints on several GUT models. We also find that the values of the matrix elements are comparable with the tree-level predictions of chiral lagrangian.Comment: 53 pages, 18 eps figure

Schizosaccharomyces pombe Int6 and Ras Homologs Regulate Cell Division and Mitotic Fidelity via the Proteasome

AbstractYin6 is a yeast homolog of Int6, which is implicated in tumorigenesis. We show that Yin6 binds to and regulates proteasome activity. Overexpression of Yin6 strengthens proteasome function while inactivation weakens and causes the accumulation of polyubiquitinated proteins including securin/Cut2 and cyclin/Cdc13. Yin6 regulates the proteasome by preferentially interacting with Rpn5, a conserved proteasome subunit, and affecting its localization/assembly. We showed previously that Yin6 cooperates with Ras1 to mediate chromosome segregation; here, we demonstrate that Ras1 similarly regulates the proteasome via Rpn5. In yeast, human Int6 binds Rpn5 and regulates its localization. We propose that human Int6, either alone or cooperatively with Ras, influences proteasome activities via Rpn5. Inactivating Int6 can lead to accumulation of mitotic regulators affecting cell division and mitotic fidelity