2,921 research outputs found
Deep crustal heating by neutrinos from the surface of accreting neutron stars
We present a new mechanism for deep crustal heating in accreting neutron
stars. Charged pions () are produced in nuclear collisions on the
neutron star surface during active accretion and upon decay they provide a flux
of neutrinos into the neutron star crust. For massive and/or compact neutron
stars, neutrinos deposit of heat per
accreted nucleon into the inner crust. The strength of neutrino heating is
comparable to the previously known sources of deep crustal heating, such as
from pycnonuclear fusion reactions, and is relevant for studies of cooling
neutron stars. We model the thermal evolution of a transient neutron star in a
low-mass X-ray binary, and in the particular case of the neutron star
MXB~1659-29 we show that additional deep crustal heating requires a higher
thermal conductivity for the neutron star inner crust. A better knowledge of
pion production cross sections near threshold would improve the accuracy of our
predictions.Comment: 12 pages, 9 figures, 3 tables; [Added a new figure and edited the
text in response to Referee's remarks and suggestions
Antioxidant Supplementation Impairs Changes in Body Composition Induced by Strength Training in Young Women
International Journal of Exercise Science 12(2): 287-296, 2019. Strength training (ST) is known to promote muscle hypertrophy and body composition adaptations. However, only a few studies investigated the effects of ST combined with antioxidant supplementation (AS) on these adaptations. The aim of this study was to investigate chronic effects of ST combined with AS on fat mass (FM) and fat-free mass (FFM) of young women. In a double-blinded design, thirty-three subjects (22.9 ± 2.5 years, 57.7 ± 8.4 kg, 1.6 ± 0.6 m) were allocated into three groups: 1) vitamins (n=12), 2) placebo (n=11) and 3) control (n=10). Vitamins and placebo underwent a ST program for 10 weeks. Vitamins supplemented with vitamin C (1g/day) and E (400IU/day) during the training period. FM and FFM were assessed by DEXA. Multiple 3 x 2 (group x time) mixed- factor ANOVA with Tukey adjustment was performed to examine differences in the dependent variables. The significance level was set at P ≤ .05. Only placebo increased total FFM (34.9 ± 4.9 vs 36.3 ± 4.8 kg, P\u3c0.05) and decreased total FM (21.8 ± 7.8 vs 21.0 ± 8.3 kg, P\u3c0.05) after training for 10 weeks. Moreover, only placebo presented a significantly greater FFM percent change from pre to post-intervention compared to control (4.0 ± 3.4 vs -0.7 ± 3.1%, respectively, P \u3c 0.05). These results suggest that chronic AS can mitigate ST related improvements of body composition in young women
Regulation of succinate-fuelled mitochondrial respiration in liver and skeletal muscle of hibernating thirteen-lined ground squirrels.
Hibernating ground squirrels (Ictidomys tridecemlineatus) alternate between two distinct metabolic states throughout winter: torpor, during which metabolic rate (MR) and body temperature (Tb) are considerably suppressed, and interbout euthermia (IBE), during which MR and Tb briefly return to euthermic levels. Previous studies showed suppression of succinate-fuelled respiration during torpor in liver and skeletal muscle mitochondria; however, these studies used only a single, saturating succinate concentration. Therefore, they could not address whether mitochondrial metabolic suppression occurs under physiological substrate concentrations or whether differences in the kinetics of mitochondrial responses to changing substrate concentration might also contribute to mitochondrial metabolic regulation during torpor. The present study confirmed that succinate oxidation is reduced during torpor in liver and skeletal muscle at 37 and 10°C over a 100-fold range of succinate concentrations. At 37°C, this suppression resulted from inhibition of succinate dehydrogenase (SDH), which had a greater affinity for oxaloacetate (an SDH inhibitor) during torpor. At 10°C, SDH was not inhibited, suggesting that SDH inhibition initiates but does not maintain mitochondrial suppression during torpor. Moreover, in both liver and skeletal muscle, mitochondria from torpid animals maintained relatively higher respiration rates at low succinate concentrations, which reduces the extent of energy savings that can be achieved during torpor, but may also maintain mitochondrial oxidative capacity above some lower critical threshold, thereby preventing cellular and/or mitochondrial injury during torpor and facilitating rapid recruitment of oxidative capacity during arousal
Quark Coulomb Interactions and the Mass Difference of Mirror Nuclei
We study the Okamoto-Nolen-Schiffer (ONS) anomaly in the binding energy of
mirror nuclei at high density by adding a single neutron or proton to a quark
gluon plasma. In this high-density limit we find an anomaly equal to two-thirds
of the Coulomb exchange energy of a proton. This effect is dominated by quark
electromagnetic interactions---rather than by the up-down quark mass
difference. At normal density we calculate the Coulomb energy of neutron matter
using a string-flip quark model. We find a nonzero Coulomb energy because of
the neutron's charged constituents. This effect could make a significant
contribution to the ONS anomaly.Comment: 4 pages, 2 figs. sub. to Phys. Rev. Let
A variational approach to the QCD wave functional:Dynamical mass generation and confinement
We perform a variational calculation in the SU(N) Yang Mills theory in 3+1
dimensions. Our trial variational states are explicitly gauge invariant, and
reduce to simple Gaussian states in the zero coupling limit. Our main result is
that the energy is minimized for the value of the variational parameter away
form the perturbative value. The best variational state is therefore
characterized by a dynamically generated mass scale . This scale is related
to the perturbative scale by the following relation:
. Taking the one loop QCD -
function and we find (for N=3) the vacuum condensate
.Comment: 37 pages, (1 Figure available upon request), preprint LA-UR-94-2727,
PUPT-149
Roughness of Interfacial Crack Front: Correlated Percolation in the Damage Zone
We show that the roughness exponent zeta of an in-plane crack front slowly
propagating along a heterogeneous interface embeded in a elastic body, is in
full agreement with a correlated percolation problem in a linear gradient. We
obtain zeta=nu/(1+nu) where nu is the correlation length critical exponent. We
develop an elastic brittle model based on both the 3D Green function in an
elastic half-space and a discrete interface of brittle fibers and find
numerically that nu=1.5, We conjecture it to be 3/2. This yields zeta=3/5. We
also obtain by direct numerical simulations zeta=0.6 in excellent agreement
with our prediction. This modelling is for the first time in close agreement
with experimental observations.Comment: 4 pages RevTeX
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